D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 68 Citations 13,299 411 World Ranking 5043 National Ranking 38

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Apoptosis

His main research concerns Apoptosis, Cancer research, Cell biology, Protein kinase B and Molecular biology. His research in Apoptosis intersects with topics in Cancer cell and Cell growth. Gi-Young Kim has included themes like Cell culture, Downregulation and upregulation, Tumor suppressor gene, Cell cycle and Epigenetics in his Cancer research study.

His work in Cell biology addresses issues such as Biochemistry, which are connected to fields such as In vivo. Gi-Young Kim combines subjects such as Microglia, PI3K/AKT/mTOR pathway, Kinase, MAPK/ERK pathway and Pharmacology with his study of Protein kinase B. The concepts of his Molecular biology study are interwoven with issues in Tumor necrosis factor alpha, Isothiocyanate, DAPI, Poly ADP ribose polymerase and Cytotoxic T cell.

His most cited work include:

  • Anti-inflammatory effects of fucoidan through inhibition of NF-κB, MAPK and Akt activation in lipopolysaccharide-induced BV2 microglia cells (189 citations)
  • Anti-inflammatory effects of β-lapachone in lipopolysaccharide-stimulated BV2 microglia (151 citations)
  • Rosmarinic acid sensitizes cell death through suppression of TNF-α-induced NF-κB activation and ROS generation in human leukemia U937 cells (123 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Apoptosis, Cell biology, Molecular biology, Cancer research and Protein kinase B. Gi-Young Kim interconnects Cancer cell, Reactive oxygen species and Downregulation and upregulation in the investigation of issues within Apoptosis. His study explores the link between Cell biology and topics such as Oxidative stress that cross with problems in DNA damage and Oxidative phosphorylation.

His Molecular biology research is multidisciplinary, relying on both Tumor necrosis factor alpha, Viability assay, Cell culture, Nitric oxide and Western blot. His Cancer research research focuses on subjects like Cell cycle, which are linked to Cell growth. His Protein kinase B study incorporates themes from PI3K/AKT/mTOR pathway and Protein kinase A.

He most often published in these fields:

  • Apoptosis (48.40%)
  • Cell biology (39.89%)
  • Molecular biology (28.99%)

What were the highlights of his more recent work (between 2017-2021)?

  • Apoptosis (48.40%)
  • Cell biology (39.89%)
  • Reactive oxygen species (15.69%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Apoptosis, Cell biology, Reactive oxygen species, Oxidative stress and Signal transduction. His studies in Apoptosis integrate themes in fields like Cancer cell and Cancer research. His Cancer research study combines topics in areas such as Cancer and Cyclin-dependent kinase, Cyclin A.

His research integrates issues of Sulforaphane, Camptothecin, Glutathione, Viability assay and Molecular biology in his study of Reactive oxygen species. His Signal transduction research integrates issues from Tumor necrosis factor alpha, Receptor, Osteoclast and RANKL. His biological study spans a wide range of topics, including Inflammation, Lipopolysaccharide and Nitric oxide.

Between 2017 and 2021, his most popular works were:

  • Spermidine Protects against Oxidative Stress in Inflammation Models Using Macrophages and Zebrafish. (34 citations)
  • Isorhamnetin alleviates lipopolysaccharide-induced inflammatory responses in BV2 microglia by inactivating NF-κB, blocking the TLR4 pathway and reducing ROS generation (25 citations)
  • Protective Effect of Phloroglucinol on Oxidative Stress-Induced DNA Damage and Apoptosis through Activation of the Nrf2/HO-1 Signaling Pathway in HaCaT Human Keratinocytes. (22 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Apoptosis

His primary areas of investigation include Cell biology, Reactive oxygen species, Apoptosis, Oxidative stress and Signal transduction. His Cell biology research incorporates elements of Fin regeneration, Zebrafish, Osteoblast, Bone morphogenetic protein 2 and Alkaline phosphatase. The various areas that Gi-Young Kim examines in his Reactive oxygen species study include Glutathione, Kinase, Phosphorylation, MAPK/ERK pathway and Transforming growth factor.

His study in Apoptosis is interdisciplinary in nature, drawing from both Cancer cell and Cancer research. Gi-Young Kim works mostly in the field of Cancer research, limiting it down to topics relating to Genistein and, in certain cases, Protein kinase B, as a part of the same area of interest. His Oxidative stress research is multidisciplinary, incorporating elements of HaCaT, Oxidative phosphorylation, PI3K/AKT/mTOR pathway and DNA damage.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Anti-inflammatory effects of fucoidan through inhibition of NF-κB, MAPK and Akt activation in lipopolysaccharide-induced BV2 microglia cells

Hye Young Park;Min Ho Han;Cheol Park;Cheng-Yun Jin.
Food and Chemical Toxicology (2011)

280 Citations

Anti-inflammatory effects of β-lapachone in lipopolysaccharide-stimulated BV2 microglia

Dong-Oh Moon;Yung Hyun Choi;Nam-Duk Kim;Yeong-Min Park.
International Immunopharmacology (2007)

226 Citations

Up-regulation of Nrf2-mediated heme oxygenase-1 expression by eckol, a phlorotannin compound, through activation of Erk and PI3K/Akt.

Ki Cheon Kim;Kyoung Ah Kang;Rui Zhang;Mei Jing Piao.
The International Journal of Biochemistry & Cell Biology (2010)

203 Citations

Rosmarinic acid sensitizes cell death through suppression of TNF-α-induced NF-κB activation and ROS generation in human leukemia U937 cells

Dong-Oh Moon;Mun-Ock Kim;Jae-Dong Lee;Yung Hyun Choi.
Cancer Letters (2010)

185 Citations

Inhibitory effects of eicosapentaenoic acid on lipopolysaccharide-induced activation in BV2 microglia

Dong-Oh Moon;Ki-Cheon Kim;Cheng-Yun Jin;Min-Ho Han.
International Immunopharmacology (2007)

169 Citations

Butein induces G2/M phase arrest and apoptosis in human hepatoma cancer cells through ROS generation

Dong-Oh Moon;Mun-Ock Kim;Yung Hyun Choi;Jin Won Hyun.
Cancer Letters (2010)

161 Citations

β-Sitosterol Induces Anti-proliferation and Apoptosis in Human Leukemic U937 Cells through Activation of Caspase-3 and Induction of Bax/Bcl-2 Ratio

Cheol Park;Dong-Oh Moon;Chung-Ho Rhu;Byung Tae Choi.
Biological & Pharmaceutical Bulletin (2007)

141 Citations

Anti-inflammatory effects of cordycepin via suppression of inflammatory mediators in BV2 microglial cells.

Jin-Woo Jeong;Cheng-Yun Jin;Gi-Young Kim;Jae-Dong Lee.
International Immunopharmacology (2010)

138 Citations

Curcumin attenuates inflammatory response in IL-1β-induced human synovial fibroblasts and collagen-induced arthritis in mouse model

Dong-Oh Moon;Mun-Ok Kim;Yung Hyun Choi;Yung-Min Park.
International Immunopharmacology (2010)

138 Citations

Epigenetic changes induced by oxidative stress in colorectal cancer cells: methylation of tumor suppressor RUNX3

Kyoung Ah Kang;Rui Zhang;Gi Young Kim;Suk Chul Bae.
Tumor Biology (2012)

133 Citations

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