His primary scientific interests are in Olfactory epithelium, Olfaction, Epithelium, Pathology and Neurogenesis. He interconnects Olfactory ensheathing glia, Olfactory mucosa and Cell biology in the investigation of issues within Olfactory epithelium. His Olfaction research is multidisciplinary, incorporating perspectives in Basal, Audiology, Communication, Degeneration and Odor.
He combines subjects such as Internal medicine, Quantitative analysis, Endocrinology and Sniffing with his study of Odor. His research investigates the connection between Epithelium and topics such as Lesion that intersect with problems in Respiratory system. Steven L. Youngentob studied Pathology and Olfactory nerve that intersect with Anatomy, Glial cell proliferation, Reinnervation and Olfactory marker protein.
Steven L. Youngentob mostly deals with Olfaction, Odor, Olfactory epithelium, Neuroscience and Olfactory system. The concepts of his Olfaction study are interwoven with issues in Sensory threshold, Psychophysics, Neural coding and Communication. His work deals with themes such as Endocrinology, Internal medicine, Nicotine, Physiology and Sniffing, which intersect with Odor.
His Olfactory epithelium research includes themes of Epithelium, Lesion, Pathology and Olfactory bulb, Olfactory ensheathing glia. The Olfactory ensheathing glia study combines topics in areas such as Immunology and Cell biology. His work on Sensory system as part of general Neuroscience study is frequently linked to Statistical analysis, bridging the gap between disciplines.
Odor, Physiology, Olfaction, Ethanol and Taste are his primary areas of study. His Odor study incorporates themes from Internal medicine and Nicotine. Steven L. Youngentob connects Olfaction with Behavioral response in his research.
In the field of Ethanol, his study on Ethanol abuse overlaps with subjects such as Naltrexone. Steven L. Youngentob has researched Taste in several fields, including Alcohol, Nicotine replacement therapy, Anesthesia and Irritation. His research integrates issues of Fetus and Endocrinology in his study of Offspring.
His primary areas of study are Fetus, Endocrinology, Internal medicine, Ethanol abuse and Gestation. The study of Fetus is intertwined with the study of Olfactory mucosa in a number of ways. His Olfactory mucosa research incorporates elements of Stimulus, Odor, Olfactory bulb, Addiction and Nicotine.
Steven L. Youngentob frequently studies issues relating to Offspring and Nicotine. The study incorporates disciplines such as Gestational exposure, Animal studies and Pregnancy in addition to Ethanol abuse.
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Adult olfactory epithelium contains multipotent progenitors that give rise to neurons and non‐neural cells
Josee M.T. Huard;Steven L. Youngentob;Bradley J. Goldstein;Marla B. Luskin.
The Journal of Comparative Neurology (1998)
Reconstitution of the rat olfactory epithelium after methyl bromide‐induced lesion
James E. Schwob;Steven L. Youngentob;Renee C. Mezza.
The Journal of Comparative Neurology (1995)
A quantitative analysis of sniffing strategies in rats performing odor detection tasks.
Steven L. Youngentob;Maxwell M. Mozell;Paul R. Sheehe;David E. Hornung.
Physiology & Behavior (1987)
The aging olfactory epithelium: Neurogenesis, response to damage, and odorant-induced activity
Alice T. Loo;Steven L. Youngentob;Paul F. Kent;James E. Schwob.
International Journal of Developmental Neuroscience (1996)
Odorant Receptor Expression Patterns Are Restored in Lesion-Recovered Rat Olfactory Epithelium
Carrie L. Iwema;Hengsheng Fang;Daniel B. Kurtz;Steven L. Youngentob.
The Journal of Neuroscience (2004)
Globose basal cells are required for reconstitution of olfactory epithelium after methyl bromide lesion
Woochan Jang;Steven L. Youngentob;James E. Schwob.
The Journal of Comparative Neurology (2003)
Reinnervation of the rat olfactory bulb after methyl bromide-induced lesion: timing and extent of reinnervation.
James E. Schwob;Steven L. Youngentob;George Ring;Carrie L. Iwema.
The Journal of Comparative Neurology (1999)
Retroviral lineage studies of the rat olfactory epithelium.
James E. Schwob;Josee M.T. Huard;Marla B. Luskin;Steven L. Youngentob.
Chemical Senses (1994)
OMP gene deletion causes an elevation in behavioral threshold sensitivity.
Steven L. Youngentob;Frank L. Margolis.
Expression patterns of basic helix‐loop‐helix transcription factors define subsets of olfactory progenitor cells
Glen L. Manglapus;Glen L. Manglapus;Steven L. Youngentob;James E. Schwob.
The Journal of Comparative Neurology (2004)
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