D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 42 Citations 4,818 93 World Ranking 4435 National Ranking 224

Overview

What is he best known for?

The fields of study he is best known for:

  • Neuron
  • Neurotransmitter
  • Neuroscience

His main research concerns Neuroscience, Synapsin, Neurotransmission, Cell biology and Electrophysiology. In general Neuroscience, his work in Hippocampus, Neurotransmitter and Neocortex is often linked to High resolution and Gene Knockdown Techniques linking many areas of study. His study in Synapsin is interdisciplinary in nature, drawing from both Synapsin I and Inhibitory postsynaptic potential.

Pietro Baldelli studied Neurotransmission and Long-term potentiation that intersect with Neurotrophin, GABAA receptor, Depolarization and Actin. Pietro Baldelli interconnects Synaptic plasticity and In vivo in the investigation of issues within Cell biology. Pietro Baldelli combines subjects such as Neuroplasticity and Nerve net with his study of Electrophysiology.

His most cited work include:

  • The synapsins: Key actors of synapse function and plasticity (383 citations)
  • Acute stress increases depolarization-evoked glutamate release in the rat prefrontal/frontal cortex: the dampening action of antidepressants. (216 citations)
  • SYN1 loss-of-function mutations in autism and partial epilepsy cause impaired synaptic function (154 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Neuroscience, Cell biology, Synapsin, Neurotransmission and Synaptic plasticity. His research ties gamma-Aminobutyric acid and Neuroscience together. His studies in Cell biology integrate themes in fields like Exocytosis, Neurotrophin and In vivo.

His Synapsin research incorporates elements of Long-term potentiation, Synapsin I and Epileptogenesis, Epilepsy. His Neurotransmission study combines topics from a wide range of disciplines, such as Neuroplasticity, Hippocampus, Knockout mouse and Anatomy. His research in Synaptic plasticity tackles topics such as Sodium channel which are related to areas like Homeostasis, Premovement neuronal activity and Biological neural network.

He most often published in these fields:

  • Neuroscience (68.70%)
  • Cell biology (33.91%)
  • Synapsin (41.74%)

What were the highlights of his more recent work (between 2017-2021)?

  • Neuroscience (68.70%)
  • Synaptic vesicle (20.87%)
  • Cell biology (33.91%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Neuroscience, Synaptic vesicle, Cell biology, Neurotransmission and Excitatory postsynaptic potential. His work on Neuroscience is being expanded to include thematically relevant topics such as Pharmacological interventions. His work on Synapsin I and Synapsin as part of his general Synaptic vesicle study is frequently connected to Loss function and Neurodevelopmental disorder, thereby bridging the divide between different branches of science.

The study incorporates disciplines such as Synaptic plasticity, Glutamate receptor, Glutamatergic and Neurotransmitter in addition to Neurotransmission. In his study, GABAergic is inextricably linked to Axon initial segment, which falls within the broad field of Excitatory postsynaptic potential. His Inhibitory postsynaptic potential research includes themes of Electrophysiology and Neuron.

Between 2017 and 2021, his most popular works were:

  • PRRT2 controls neuronal excitability by negatively modulating Na+ channel 1.2/1.6 activity (45 citations)
  • Microbiota-gut brain axis involvement in neuropsychiatric disorders (35 citations)
  • Neurite-Enriched MicroRNA-218 Stimulates Translation of the GluA2 Subunit and Increases Excitatory Synaptic Strength (18 citations)

In his most recent research, the most cited papers focused on:

  • Neuron
  • Neurotransmitter
  • Biochemistry

Pietro Baldelli mostly deals with Premovement neuronal activity, Cell biology, Excitatory postsynaptic potential, Neuroscience and Neurotransmission. His research in Premovement neuronal activity intersects with topics in Hyperpolarization, Photostimulation, Synaptic vesicle and Biophysics. His Cell biology research integrates issues from AMPA receptor, Glutamatergic, Neurite and Neuron.

His Excitatory postsynaptic potential research incorporates themes from Hippocampal formation, Hippocampus and Homeostasis. His work on Excitatory synapse as part of general Neuroscience study is frequently linked to Synaptic scaling, bridging the gap between disciplines. His research on Neurotransmission frequently connects to adjacent areas such as Synaptic plasticity.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The synapsins: Key actors of synapse function and plasticity

F. Cesca;P. Baldelli;F. Valtorta;F. Benfenati.
Progress in Neurobiology (2010)

578 Citations

Acute stress increases depolarization-evoked glutamate release in the rat prefrontal/frontal cortex: the dampening action of antidepressants.

Laura Musazzi;Marco Milanese;Pasqualina Farisello;Simona Zappettini.
PLOS ONE (2010)

291 Citations

SYN1 loss-of-function mutations in autism and partial epilepsy cause impaired synaptic function

Anna Fassio;Lysanne Patry;Sonia Congia;Franco Onofri.
Human Molecular Genetics (2011)

215 Citations

Lack of Synapsin I Reduces the Readily Releasable Pool of Synaptic Vesicles at Central Inhibitory Synapses

Pietro Baldelli;Anna Fassio;Flavia Valtorta;Fabio Benfenati.
The Journal of Neuroscience (2007)

170 Citations

Protein kinase A-mediated synapsin I phosphorylation is a central modulator of Ca2+-dependent synaptic activity.

Andrea Menegon;Dario Bonanomi;Chiara Albertinazzi;Francesco Lotti.
The Journal of Neuroscience (2006)

158 Citations

Effective gating charges per channel in voltage-dependent K+ and Ca2+ channels.

Francesca Noceti;Pietro Baldelli;Xiangyang Wei;Ning Qin.
The Journal of General Physiology (1996)

133 Citations

Opposite Changes in Glutamatergic and GABAergic Transmission Underlie the Diffuse Hyperexcitability of Synapsin I–Deficient Cortical Networks

Michela Chiappalone;Michela Chiappalone;Silvia Casagrande;Mariateresa Tedesco;Flavia Valtorta.
Cerebral Cortex (2009)

124 Citations

Brain-Derived Neurotrophic Factor Enhances GABA Release Probability and Nonuniform Distribution of N- and P/Q-Type Channels on Release Sites of Hippocampal Inhibitory Synapses

Pietro Baldelli;Jesus-Miguel Hernandez-Guijo;Valentina Carabelli;Emilio Carbone.
The Journal of Neuroscience (2005)

113 Citations

Emergent functional properties of neuronal networks with controlled topology.

Emanuele Marconi;Thierry Nieus;Alessandro Maccione;Pierluigi Valente.
PLOS ONE (2012)

110 Citations

PRRT2 Is a Key Component of the Ca2+-Dependent Neurotransmitter Release Machinery

Pierluigi Valente;Enrico Castroflorio;Pia Rossi;Manuela Fadda.
Cell Reports (2016)

109 Citations

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