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Biology and Biochemistry

D-Index
61
Citations
11148
World Ranking
11533
National Ranking
14

Overview

Petr Ježek is affiliated with the Institute of Physiology in the Czech Republic. Their research primarily focuses on areas within Biochemistry, Genetics and Molecular Biology as well as Medicine. They have contributed extensively to several related subfields, including Molecular Biology, Physiology, Surgery, Materials Chemistry, and Endocrinology, Diabetes and Metabolism.

The main topics of Petr Ježek's scientific work encompass Mitochondrial Function and Pathology, Adipose Tissue and Metabolism, Pancreatic Function and Diabetes, ATP Synthase and ATPases Research, Diet, Metabolism, and Disease, Cancer, Hypoxia, and Metabolism, as well as Metabolism, Diabetes, and Cancer.

Frequent co-authors in their publications include Martin Jabůrek, Hana Engstová, Andrea Dlasková, Pavla Průchová, and Blanka Holendová.

Petr Ježek has published in many venues, with notable recurring publication platforms such as Free Radical Biology and Medicine, Biochimica et Biophysica Acta (BBA) - Bioenergetics, Antioxidants and Redox Signaling, Scientific Reports, and Antioxidants.

Recent papers authored or co-authored by Petr Ježek include:

  • Mitochondrial Cristae Morphology Reflecting Metabolism, Superoxide Formation, Redox Homeostasis, and Pathology (2023, Antioxidants and Redox Signaling)
  • Redox Signaling from Mitochondria: Signal Propagation and Its Targets (2020, Biomolecules)

Other relevant papers within their recent academic contribution include:

  • Glucose-Stimulated Insulin Secretion Fundamentally Requires H2O2 Signaling by NADPH Oxidase 4 (2020, Diabetes)
  • Mitochondrial Dynamics and Cristae Shape Changes During Metabolic Reprogramming (2023, Antioxidants and Redox Signaling)
  • Mitochondrial Superoxide Production Decreases on Glucose-Stimulated Insulin Secretion in Pancreatic β Cells Due to Decreasing Mitochondrial Matrix NADH/NAD+ Ratio (2020, Antioxidants and Redox Signaling)

Best Publications

  • On the Mechanism of Fatty Acid-induced Proton Transport by Mitochondrial Uncoupling Protein

    Keith D. Garlid;David E. Orosz;Martin Modrianský;Stefano Vassanelli

  • Transport Function and Regulation of Mitochondrial Uncoupling Proteins 2 and 3

    Martin Jabůrek;Miroslav Vařecha;Ruth E. Gimeno;Marlene Dembski

  • Waves of gene regulation suppress and then restore oxidative phosphorylation in cancer cells.

    Katarína Smolková;Lydie Plecitá-Hlavatá;Nadége Bellance;Giovanni Benard

  • Fatty acid cycling mechanism and mitochondrial uncoupling proteins

    P. Jezek;H. Engstova;M. Zackova;A. E. Vercesi

  • Metabolic and Proliferative State of Vascular Adventitial Fibroblasts in Pulmonary Hypertension Is Regulated Through a MicroRNA-124/PTBP1 (Polypyrimidine Tract Binding Protein 1)/Pyruvate Kinase Muscle Axis

    Hui Zhang;Daren Wang;Min Li;Lydie Plecitá-Hlavatá

  • The mechanism of proton transport mediated by mitochondrial uncoupling proteins

    Keith D. Garlid;Martin Jabůrek;Petr Ježek

  • Possible physiological roles of mitochondrial uncoupling proteins—UCPn

    Petr Ježek

  • Sample drift correction in 3D fluorescence photoactivation localization microscopy

    Michael J Mlodzianoski;John M Schreiner;Steven P Callahan;Katarina Smolková

  • How do uncoupling proteins uncouple

    Keith D. Garlid;Martin Jabůrek;Petr Ježek;Miroslav Vařecha

  • Transport of anions and protons by the mitochondrial uncoupling protein and its regulation by nucleotides and fatty acids. A new look at old hypotheses

    P Jezek;D E Orosz;M Modriansky;K D Garlid

  • Metabolic Reprogramming Regulates the Proliferative and Inflammatory Phenotype of Adventitial Fibroblasts in Pulmonary Hypertension Through the Transcriptional Corepressor C-Terminal Binding Protein-1.

    Min Li;Suzette Riddle;Hui Zhang;Angelo D’Alessandro

  • New substrates and competitive inhibitors of the Cl- translocating pathway of the uncoupling protein of brown adipose tissue mitochondria.

    P Jezek;K D Garlid

  • Mitochondrial uncoupling proteins and phylogenesis--UCP4 as the ancestral uncoupling protein.

    Petr Hanák;Petr Ježek

  • Acinetobacter parvus sp. nov., a small-colony-forming species isolated from human clinical specimens.

    Alexandr Nemec;Lenie Dijkshoorn;Ilse Cleenwerck;Thierry De Baere

  • Mitochondrial bioenergetic adaptations of breast cancer cells to aglycemia and hypoxia.

    Katarína Smolková;Nadège Bellance;Francesca Scandurra;Elisabeth Génot

  • Mammalian mitochondrial uncoupling proteins

    P Jezek;K D Garlid

  • Mitochondrial Uncoupling Proteins: Subtle Regulators of Cellular Redox Signaling.

    Petr Ježek;Blanka Holendová;Keith D. Garlid;Martin Jabůrek

  • The Role of Mitochondrial NADPH-Dependent Isocitrate Dehydrogenase in Cancer Cells.

    Katarína Smolková;Petr Ježek

  • Distinctions and similarities of cell bioenergetics and the role of mitochondria in hypoxia, cancer, and embryonic development

    Petr Jezek;Lydie Plecitá-Hlavatá;Katarína Smolková;Rodrigue Rossignol

  • Selective Disruption of Respiratory Supercomplexes as a New Strategy to Suppress Her2high Breast Cancer

    Katerina Rohlenova;Karishma Sachaphibulkij;Jan Stursa;Ayenachew Bezawork-Geleta

Frequent Co-Authors

Keith D. Garlid
Keith D. Garlid Portland State University
Daniel Horák
Daniel Horák Czech Academy of Sciences
Kurt R. Stenmark
Kurt R. Stenmark University of Colorado Anschutz Medical Campus
Miroslav Šlouf
Miroslav Šlouf Czech Academy of Sciences
Angelo D'Alessandro
Angelo D'Alessandro University of Colorado Denver
Vladimír Křen
Vladimír Křen Czech Academy of Sciences
Paulo Arruda
Paulo Arruda State University of Campinas
Mariusz R. Wieckowski
Mariusz R. Wieckowski Instytut Biologii Doświadczalnej im. Marcelego Nenckiego
Louis A. Tartaglia
Louis A. Tartaglia Toledo Clinic Cancer Center
Juan F. Medrano
Juan F. Medrano University of California, Davis

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