His scientific interests lie mostly in Immunology, Immune system, Virology, Influenza A virus and T cell. His Immunology study focuses mostly on Virus, Immunity, Innate immune system, Inflammation and Proinflammatory cytokine. The concepts of his Immune system study are interwoven with issues in Virus latency and Epidemiology.
Paul G. Thomas is interested in Vaccination, which is a branch of Virology. His Influenza A virus research incorporates elements of Acquired immune system, Influenza A virus subtype H5N1 and Respiratory infection. His T cell study combines topics in areas such as Epitope, Cytotoxic T cell, CD8 and Paired Analysis.
Immunology, Virology, Immune system, T cell and Virus are his primary areas of study. His Immunology research is multidisciplinary, incorporating perspectives in Disease and Lung. His work deals with themes such as Cell, Cancer research and Cytokine, which intersect with Immune system.
He has researched T cell in several fields, including Cytotoxic T cell and CD8, Avidity, Antigen. His research integrates issues of Acquired immune system and Pandemic in his study of Virus. His T-cell receptor study incorporates themes from Epitope, Receptor, Major histocompatibility complex and Cell biology.
His main research concerns Immunology, Virus, Immune system, CD8 and T-cell receptor. His research brings together the fields of Disease and Immunology. He works on Virus which deals in particular with Influenza A virus.
The subject of his Influenza A virus research is within the realm of Virology. He is studying T cell, which is a component of Immune system. His T-cell receptor research incorporates themes from Epitope, Human leukocyte antigen, Major histocompatibility complex, Antigen and Cytotoxic T cell.
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The Intracellular Sensor NLRP3 Mediates Key Innate and Healing Responses to Influenza A Virus via the Regulation of Caspase-1
Paul G. Thomas;Pradyot Dash;Jerry R. Aldridge;Ali H. Ellebedy.
Cell-mediated Protection in Influenza Infection
Paul G. Thomas;Rachael Keating;Diane J. Hulse-Post;Peter C. Doherty.
Emerging Infectious Diseases (2006)
TNF/iNOS-producing dendritic cells are the necessary evil of lethal influenza virus infection
Jerry R Aldridge;Carson E Moseley;David A Boltz;Nicholas J Negovetich.
Proceedings of the National Academy of Sciences of the United States of America (2009)
Influenza and the challenge for immunology.
Peter C Doherty;Peter C Doherty;Stephen J Turner;Richard G Webby;Paul G Thomas.
Nature Immunology (2006)
Maturation of Dendritic Cell 2 Phenotype by a Helminth Glycan Uses a Toll-Like Receptor 4-Dependent Mechanism
Paul G. Thomas;Michele R. Carter;Olga Atochina;Akram A. Da’Dara.
Journal of Immunology (2003)
Quantifiable predictive features define epitope-specific T cell receptor repertoires
Pradyot K Dash;Andrew J. Fiore-Gartland;Tomer Hertz;Tomer Hertz;George C. Wang.
De Novo Epigenetic Programs Inhibit PD-1 Blockade-Mediated T Cell Rejuvenation
Hazem E. Ghoneim;Yiping Fan;Ardiana Moustaki;Hossam A. Abdelsamed.
Receptor interacting protein kinase 2-mediated mitophagy regulates inflammasome activation during virus infection
Christopher R. Lupfer;Paul G Thomas;Paras K. Anand;Peter Vogel.
Nature Immunology (2013)
Depletion of Alveolar Macrophages during Influenza Infection Facilitates Bacterial Superinfections
Hazem E. Ghoneim;Paul G. Thomas;Jonathan A. McCullers;Jonathan A. McCullers.
Journal of Immunology (2013)
Cytomegalovirus infection enhances the immune response to influenza
David Furman;Vladimir Jojic;Shalini Sharma;Shai S. Shen-Orr.
Science Translational Medicine (2015)
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