His primary areas of investigation include Gap junction, Connexin, Endocrinology, Internal medicine and Cell biology. Otto Traub studies Lucifer yellow, a branch of Gap junction. His research in Connexin intersects with topics in Immunology, Carcinoma, Pathology, Exocrine gland and Polyclonal antibodies.
His Endocrinology research integrates issues from Wild type, Gene, Second messenger system and Cardiac cycle. His work on Preputial gland, Endocrine gland and Pancreas as part of general Internal medicine research is frequently linked to Sinus rhythm and Seminal vesicle, bridging the gap between disciplines. Otto Traub has researched Cell biology in several fields, including Embryonic stem cell, Apoptosis, Astrocyte, Neuropil and Neuroscience.
Otto Traub mainly investigates Connexin, Gap junction, Cell biology, Molecular biology and Internal medicine. His Connexin research includes themes of Gene expression, Northern blot, Immunology, Neuroscience and Embryo. His Gap junction study integrates concerns from other disciplines, such as Cell junction and Anatomy.
The concepts of his Cell biology study are interwoven with issues in Cell culture, Transfection and Genetics. His Molecular biology research incorporates themes from Immunofluorescence, Downregulation and upregulation, Messenger RNA and Phosphorylation. Many of his research projects under Internal medicine are closely connected to Decidua with Decidua, tying the diverse disciplines of science together.
Otto Traub spends much of his time researching Cell biology, Connexin, Gap junction, Molecular biology and Transfection. Otto Traub combines subjects such as Choriocarcinoma and Anatomy with his study of Cell biology. His Connexin research is multidisciplinary, relying on both Cerebellum, Neuroglia, Cerebral cortex, Blastocyst and Epidermis.
The various areas that he examines in his Gap junction study include Biophysics, Cell junction and Gene isoform. His work carried out in the field of Molecular biology brings together such families of science as Downregulation and upregulation and Mutant. His Transfection study combines topics in areas such as Cell growth and Phosphorylation.
The scientist’s investigation covers issues in Connexin, Molecular biology, Downregulation and upregulation, Cell junction and Gap junction. His Connexin research is multidisciplinary, incorporating perspectives in Hippocampal formation, Central nervous system, Neuroscience, Neuron and NeuN. His Molecular biology study combines topics from a wide range of disciplines, such as Cell culture, Immunoprecipitation, Cell growth, Cell division and Transcription.
His Cell junction research is multidisciplinary, incorporating elements of Endothelial stem cell, Lucifer yellow, Aorta, Anatomy and Cell biology. His research in Cell biology intersects with topics in Genetics and Endothelium. Otto Traub performs multidisciplinary study in Gap junction and Trophoblast in his work.
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Specific permeability and selective formation of gap junction channels in connexin-transfected HeLa cells.
C Elfgang;R Eckert;H Lichtenberg-Fraté;A Butterweck.
Journal of Cell Biology (1995)
DEFECTIVE PROPAGATION OF SIGNALS GENERATED BY SYMPATHETIC NERVE STIMULATION IN THE LIVER OF CONNEXIN32-DEFICIENT MICE
Eric Nelles;Christoph Butzler;Dirk Jung;Achim Temme.
Proceedings of the National Academy of Sciences of the United States of America (1996)
Reduced cardiac conduction velocity and predisposition to arrhythmias in connexin40-deficient mice
Susanne Kirchhoff;Eric Nelles;Andreas Hagendorff;Olaf Krüger.
Current Biology (1998)
Comparative characterization of the 21-kD and 26-kD gap junction proteins in murine liver and cultured hepatocytes.
Otto Traub;Jutta Look;Rolf Dermietzel;Franz Brümmer.
Journal of Cell Biology (1989)
Defective vascular development in connexin 45-deficient mice
Olaf Krüger;Achim Plum;Jung-Sun Kim;Elke Winterhager.
Expression of the gap-junction connexins 26 and 30 in the rat cochlea
J. Lautermann;W.-J. F. Ten Cate;P. Altenhoff;R. Grummer.
Cell and Tissue Research (1998)
Transplacental uptake of glucose is decreased in embryonic lethal connexin26-deficient mice.
Heinz-Dieter Gabriel;Dirk Jung;Christoph Bützler;Achim Temme.
Journal of Cell Biology (1998)
Unique and shared functions of different connexins in mice
A. Plum;G. Hallas;T. Magin;F. Dombrowski.
Current Biology (2000)
Expression of Cx26, Cx32 and Cx43 gap junction proteins in normal and neoplastic human tissues.
Klaus K. Wilgenbus;Charles J. Kirkpatrick;Ruth Knuechel;Klaus Willecke.
International Journal of Cancer (1992)
Gap junction protein connexin40 is preferentially expressed in vascular endothelium and conductive bundles of rat myocardium and is increased under hypertensive conditions.
B. Bastide;Ludwig Neyses;D. Ganten;M. Paul.
Circulation Research (1993)
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