D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 48 Citations 9,016 126 World Ranking 3585 National Ranking 316

Overview

What is he best known for?

The fields of study he is best known for:

  • Neuroscience
  • Hippocampus
  • Internal medicine

His primary areas of investigation include Neuroscience, Hippocampus, Hippocampal formation, Long-term potentiation and Synaptic plasticity. His Hippocampus research is multidisciplinary, incorporating perspectives in Perirhinal cortex and Cognitive science. His work deals with themes such as Stimulus, Lesion and Contextual Associations, which intersect with Hippocampal formation.

Mark Andrew Good has included themes like AMPA receptor and Neurotransmission in his Synaptic plasticity study. His study in Neurotransmission is interdisciplinary in nature, drawing from both Glutamate receptor and NMDA receptor. His Dentate gyrus study integrates concerns from other disciplines, such as Nonsynaptic plasticity, Synaptic fatigue and Amyloid precursor protein.

His most cited work include:

  • Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic mice (848 citations)
  • Distinct components of spatial learning revealed by prior training and NMDA receptor blockade. (513 citations)
  • Distinct components of spatial learning revealed by prior training and NMDA receptor blockade. (513 citations)

What are the main themes of his work throughout his whole career to date?

Mark Andrew Good focuses on Neuroscience, Hippocampal formation, Hippocampus, Stimulus and Spatial memory. His research integrates issues of Long-term potentiation and Lesion in his study of Neuroscience. His study on Long-term potentiation also encompasses disciplines like

  • Synaptic plasticity together with Neurotransmission,
  • Glutamate receptor that intertwine with fields like Amygdala.

The concepts of his Hippocampal formation study are interwoven with issues in NMDA receptor and Discrimination learning. His study explores the link between Hippocampus and topics such as Amyloid precursor protein that cross with problems in Genetically modified mouse and Cell biology. Mark Andrew Good usually deals with Spatial memory and limits it to topics linked to Long-term memory and Memory consolidation.

He most often published in these fields:

  • Neuroscience (70.59%)
  • Hippocampal formation (37.50%)
  • Hippocampus (30.15%)

What were the highlights of his more recent work (between 2013-2021)?

  • Neuroscience (70.59%)
  • Hippocampal formation (37.50%)
  • Disease (6.62%)

In recent papers he was focusing on the following fields of study:

His main research concerns Neuroscience, Hippocampal formation, Disease, Hippocampus and Down syndrome. His study in the fields of Cognition, Electrophysiology and Prefrontal cortex under the domain of Neuroscience overlaps with other disciplines such as Chromosome 21 and Immunotherapy. As part of the same scientific family, Mark Andrew Good usually focuses on Hippocampal formation, concentrating on Dementia and intersecting with Haploinsufficiency and Medical education.

His Disease research is multidisciplinary, relying on both Bioinformatics, Clinical psychology, Age related, Depression and Risk factor. Mark Andrew Good has researched Hippocampus in several fields, including Object, Amyloid beta, Set and Ageing. Mark Andrew Good has researched Down syndrome in several fields, including Infants toddlers, Pathological and Pediatrics.

Between 2013 and 2021, his most popular works were:

  • Dietary DHA supplementation causes selective changes in phospholipids from different brain regions in both wild type mice and the Tg2576 mouse model of Alzheimer's disease (42 citations)
  • ACE2 activation protects against cognitive decline and reduces amyloid pathology in the Tg2576 mouse model of Alzheimer’s disease (27 citations)
  • Long-term multi-species Lactobacillus and Bifidobacterium dietary supplement enhances memory and changes regional brain metabolites in middle-aged rats. (27 citations)

In his most recent research, the most cited papers focused on:

  • Neuroscience
  • Internal medicine
  • Cognition

His scientific interests lie mostly in Neuroscience, Chromosome 21, Recognition memory, Prefrontal cortex and Hippocampal formation. The Neuroscience study combines topics in areas such as Conditioning process and Cognitive psychology. His studies in Recognition memory integrate themes in fields like Developmental psychology, Hippocampus and Physiology.

He interconnects DYRK1A, Electrophysiology and Spatial memory in the investigation of issues within Prefrontal cortex. His Hippocampal formation study is associated with Endocrinology. His Endocrinology research is multidisciplinary, incorporating perspectives in NMDA receptor and Internal medicine.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic mice

Paul F. Chapman;Gail L. White;Matthew W. Jones;Deirdre Cooper-Blacketer.
Nature Neuroscience (1999)

1123 Citations

Distinct components of spatial learning revealed by prior training and NMDA receptor blockade.

D. M. Bannerman;M. A. Good;M. A. Good;S. P. Butcher;M. Ramsay.
Nature (1995)

779 Citations

Double dissociation of function within the hippocampus: a comparison of dorsal, ventral, and complete hippocampal cytotoxic lesions.

D. M. Bannerman;B. K. Yee;Mark Andrew Good;M. J. Heupel.
Behavioral Neuroscience (1999)

462 Citations

Selective hippocampal lesions abolish the contextual specificity of latent inhibition and conditioning.

Robert Colin Honey;Mark Andrew Good.
Behavioral Neuroscience (1993)

460 Citations

Conditioning and contextual retrieval in hippocampal rats.

Mark Andrew Good;Robert Colin Honey.
Behavioral Neuroscience (1991)

367 Citations

Hippocampal lesions disrupt navigation based on cognitive maps but not heading vectors

John M. Pearce;A. D. L. Roberts;Mark Andrew Good.
Nature (1998)

363 Citations

Dissociable Effects of Selective Lesions to Hippocampal Subsystems on Exploratory Behavior, Contextual Learning, and Spatial Learning

Mark Andrew Good;Robert Colin Honey.
Behavioral Neuroscience (1997)

273 Citations

Hippocampal Lesions Disrupt an Associative Mismatch Process

Robert Colin Honey;Andrew A. Watt;Mark Andrew Good.
The Journal of Neuroscience (1998)

228 Citations

Cyclical changes in endogenous levels of oestrogen modulate the induction of LTD and LTP in the hippocampal CA1 region.

Mark Andrew Good;Mark Day;Janice L. Muir.
European Journal of Neuroscience (1999)

202 Citations

Identifying cortical inputs to the rat hippocampus that subserve allocentric spatial processes: A simple problem with a complex answer

John Patrick Aggleton;Seralynne Denise Vann;Catherine J. P. Oswald;Mark Andrew Good.
Hippocampus (2000)

193 Citations

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