His primary areas of investigation include Neuroscience, Hippocampus, Hippocampal formation, Long-term potentiation and Synaptic plasticity. His Hippocampus research is multidisciplinary, incorporating perspectives in Perirhinal cortex and Cognitive science. His work deals with themes such as Stimulus, Lesion and Contextual Associations, which intersect with Hippocampal formation.
Mark Andrew Good has included themes like AMPA receptor and Neurotransmission in his Synaptic plasticity study. His study in Neurotransmission is interdisciplinary in nature, drawing from both Glutamate receptor and NMDA receptor. His Dentate gyrus study integrates concerns from other disciplines, such as Nonsynaptic plasticity, Synaptic fatigue and Amyloid precursor protein.
Mark Andrew Good focuses on Neuroscience, Hippocampal formation, Hippocampus, Stimulus and Spatial memory. His research integrates issues of Long-term potentiation and Lesion in his study of Neuroscience. His study on Long-term potentiation also encompasses disciplines like
The concepts of his Hippocampal formation study are interwoven with issues in NMDA receptor and Discrimination learning. His study explores the link between Hippocampus and topics such as Amyloid precursor protein that cross with problems in Genetically modified mouse and Cell biology. Mark Andrew Good usually deals with Spatial memory and limits it to topics linked to Long-term memory and Memory consolidation.
His main research concerns Neuroscience, Hippocampal formation, Disease, Hippocampus and Down syndrome. His study in the fields of Cognition, Electrophysiology and Prefrontal cortex under the domain of Neuroscience overlaps with other disciplines such as Chromosome 21 and Immunotherapy. As part of the same scientific family, Mark Andrew Good usually focuses on Hippocampal formation, concentrating on Dementia and intersecting with Haploinsufficiency and Medical education.
His Disease research is multidisciplinary, relying on both Bioinformatics, Clinical psychology, Age related, Depression and Risk factor. Mark Andrew Good has researched Hippocampus in several fields, including Object, Amyloid beta, Set and Ageing. Mark Andrew Good has researched Down syndrome in several fields, including Infants toddlers, Pathological and Pediatrics.
His scientific interests lie mostly in Neuroscience, Chromosome 21, Recognition memory, Prefrontal cortex and Hippocampal formation. The Neuroscience study combines topics in areas such as Conditioning process and Cognitive psychology. His studies in Recognition memory integrate themes in fields like Developmental psychology, Hippocampus and Physiology.
He interconnects DYRK1A, Electrophysiology and Spatial memory in the investigation of issues within Prefrontal cortex. His Hippocampal formation study is associated with Endocrinology. His Endocrinology research is multidisciplinary, incorporating perspectives in NMDA receptor and Internal medicine.
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Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic mice
Paul F. Chapman;Gail L. White;Matthew W. Jones;Deirdre Cooper-Blacketer.
Nature Neuroscience (1999)
Distinct components of spatial learning revealed by prior training and NMDA receptor blockade.
D. M. Bannerman;M. A. Good;M. A. Good;S. P. Butcher;M. Ramsay.
Double dissociation of function within the hippocampus: a comparison of dorsal, ventral, and complete hippocampal cytotoxic lesions.
D. M. Bannerman;B. K. Yee;Mark Andrew Good;M. J. Heupel.
Behavioral Neuroscience (1999)
Selective hippocampal lesions abolish the contextual specificity of latent inhibition and conditioning.
Robert Colin Honey;Mark Andrew Good.
Behavioral Neuroscience (1993)
Conditioning and contextual retrieval in hippocampal rats.
Mark Andrew Good;Robert Colin Honey.
Behavioral Neuroscience (1991)
Hippocampal lesions disrupt navigation based on cognitive maps but not heading vectors
John M. Pearce;A. D. L. Roberts;Mark Andrew Good.
Dissociable Effects of Selective Lesions to Hippocampal Subsystems on Exploratory Behavior, Contextual Learning, and Spatial Learning
Mark Andrew Good;Robert Colin Honey.
Behavioral Neuroscience (1997)
Hippocampal Lesions Disrupt an Associative Mismatch Process
Robert Colin Honey;Andrew A. Watt;Mark Andrew Good.
The Journal of Neuroscience (1998)
Cyclical changes in endogenous levels of oestrogen modulate the induction of LTD and LTP in the hippocampal CA1 region.
Mark Andrew Good;Mark Day;Janice L. Muir.
European Journal of Neuroscience (1999)
Identifying cortical inputs to the rat hippocampus that subserve allocentric spatial processes: A simple problem with a complex answer
John Patrick Aggleton;Seralynne Denise Vann;Catherine J. P. Oswald;Mark Andrew Good.
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