David M. Bannerman mostly deals with Neuroscience, Hippocampal formation, Hippocampus, Long-term potentiation and Synaptic plasticity. His work deals with themes such as NMDA receptor and Lesion, which intersect with Neuroscience. His work in the fields of Hippocampal formation, such as Dentate gyrus, overlaps with other areas such as Calbindin.
The various areas that David M. Bannerman examines in his Hippocampus study include Dorsum and T-maze. The Long-term potentiation study combines topics in areas such as Glutamate receptor and Memory consolidation. His studies deal with areas such as No synthase, Systemic injection and Nitric oxide as well as Synaptic plasticity.
David M. Bannerman mostly deals with Neuroscience, Hippocampal formation, Hippocampus, Spatial memory and Long-term potentiation. His studies in Neuroscience integrate themes in fields like Synaptic plasticity, Lesion and Glutamate receptor, AMPA receptor, NMDA receptor. His study in Glutamate receptor is interdisciplinary in nature, drawing from both Neurotransmission and Amygdala.
His work focuses on many connections between Hippocampal formation and other disciplines, such as Working memory, that overlap with his field of interest in Systemic inflammation. His studies link T-maze with Hippocampus. In his study, which falls under the umbrella issue of Long-term potentiation, Long-term memory is strongly linked to Memory consolidation.
His main research concerns Neuroscience, Wakefulness, Cognition, NMDA receptor and Hippocampal formation. His studies in Neuroscience integrate themes in fields like Long-term potentiation and Neurodegeneration. In his study, Glutamate receptor is strongly linked to Neurotransmitter, which falls under the umbrella field of Long-term potentiation.
He has researched Cognition in several fields, including Forebrain, Environmental enrichment and Expressed emotion. His NMDA receptor research integrates issues from Associative learning, Hippocampus, Postsynaptic potential and Nervous system. His Hippocampal formation study incorporates themes from Cognitive map, Direct experience and Spatial memory.
His scientific interests lie mostly in Neuroscience, Hippocampal formation, Spatial memory, Neurodegeneration and Disease. His work is connected to Amygdala, Parvalbumin, Associative learning, Disinhibition and Excitatory postsynaptic potential, as a part of Neuroscience. The concepts of his Hippocampal formation study are interwoven with issues in Long-term potentiation, Basal, Neurology and Transgene.
His Spatial memory research is multidisciplinary, incorporating perspectives in Risk gene, GRIA1, Habituation and Salience. Neurodegeneration is often connected to Disease mechanisms in his work.
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Regional dissociations within the hippocampus--memory and anxiety.
D.M Bannerman;J.N.P Rawlins;S.B McHugh;R.M.J Deacon.
Neuroscience & Biobehavioral Reviews (2004)
Action sets and decisions in the medial frontal cortex
M.F.S. Rushworth;M.F.S. Rushworth;M.E. Walton;S.W. Kennerley;D.M. Bannerman.
Trends in Cognitive Sciences (2004)
Distinct components of spatial learning revealed by prior training and NMDA receptor blockade.
D. M. Bannerman;M. A. Good;M. A. Good;S. P. Butcher;M. Ramsay.
Separate neural pathways process different decision costs
Peter H Rudebeck;Mark E Walton;Angharad N Smyth;David M Bannerman.
Nature Neuroscience (2006)
Functional Specialization within Medial Frontal Cortex of the Anterior Cingulate for Evaluating Effort-Related Decisions
Mark E. Walton;David M. Bannerman;Karin Alterescu;Matthew F. S. Rushworth.
The Journal of Neuroscience (2003)
Ventral hippocampal lesions affect anxiety but not spatial learning.
DM Bannerman;Matthew S Grubb;RM Deacon;BK Yee.
Behavioural Brain Research (2003)
Hippocampal synaptic plasticity, spatial memory and anxiety
David M. Bannerman;Rolf Sprengel;David J. Sanderson;Stephen B. McHugh.
Nature Reviews Neuroscience (2014)
Recruitment of parvalbumin-positive interneurons determines hippocampal function and associated behavior.
Elke C. Fuchs;Aleksandar R. Zivkovic;Mark O. Cunningham;Steven Middleton.
Catechol-o-methyltransferase inhibition improves set-shifting performance and elevates stimulated dopamine release in the rat prefrontal cortex.
E M Tunbridge;D M Bannerman;T Sharp;P J Harrison.
The Journal of Neuroscience (2004)
Double dissociation of function within the hippocampus: a comparison of dorsal, ventral, and complete hippocampal cytotoxic lesions.
D. M. Bannerman;B. K. Yee;Mark Andrew Good;M. J. Heupel.
Behavioral Neuroscience (1999)
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