Kay L. Double focuses on Parkinson's disease, Substantia nigra, Pathology, Oxidative stress and Human brain. His study looks at the relationship between Parkinson's disease and fields such as Neuroscience, as well as how they intersect with chemical problems. Kay L. Double studies Substantia nigra, namely Neuromelanin.
His Neuromelanin research is multidisciplinary, incorporating elements of Melanin and Intracellular. The Atrophy research he does as part of his general Pathology study is frequently linked to other disciplines of science, such as White matter, therefore creating a link between diverse domains of science. His Oxidative stress research integrates issues from Ferritin and Alpha-synuclein.
The scientist’s investigation covers issues in Substantia nigra, Parkinson's disease, Neuroscience, Internal medicine and Neuromelanin. His work carried out in the field of Substantia nigra brings together such families of science as Oxidative stress and Neurodegeneration. Kay L. Double has included themes like Neurology and Human brain in his Parkinson's disease study.
The various areas that Kay L. Double examines in his Neuroscience study include Degeneration, Dementia and Proteasome. His work deals with themes such as Muscle tone and Endocrinology, which intersect with Internal medicine. The study incorporates disciplines such as Biophysics and Melanin, Biochemistry, Ferritin, Intracellular in addition to Neuromelanin.
His primary scientific interests are in Parkinson's disease, Substantia nigra, Neuroscience, Dopaminergic and Internal medicine. His studies deal with areas such as Oxidative stress and Physical therapy as well as Parkinson's disease. His Substantia nigra research includes themes of Transcranial Doppler, Abnormality and Ageing.
He has researched Neuroscience in several fields, including Lewy body and Neurodegeneration. His biological study spans a wide range of topics, including Basal forebrain, Nucleus basalis, Cholinergic, Cholinergic neuron and Dementia. His Internal medicine research is multidisciplinary, relying on both Neurogenesis and Endocrinology.
Kay L. Double mainly investigates Parkinson's disease, Oxidative stress, Alpha-synuclein, Neuroscience and Substantia nigra. The subject of his Parkinson's disease research is within the realm of Pathology. His research in Pathology intersects with topics in Superoxide dismutase and Human brain.
Kay L. Double works mostly in the field of Alpha-synuclein, limiting it down to topics relating to Neurodegeneration and, in certain cases, Ageing and Central nervous system disease. Neuroscience is represented through his Dopaminergic and Basal forebrain research. His Substantia nigra research is under the purview of Dopamine.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Brain iron pathways and their relevance to Parkinson's disease.
D Berg;M. Gerlach;M. B. H. Youdim;K. L. Double.
Journal of Neurochemistry (2002)
Neuromelanin in human dopamine neurons: comparison with peripheral melanins and relevance to Parkinson's disease.
H Fedorow;F Tribl;G Halliday;M Gerlach.
Progress in Neurobiology (2005)
Language-associated Cortical Regions Are Proportionally Larger in the Female Brain
Jenny Harasty;Kay L. Double;Glenda M. Halliday;Jillian J. Kril.
JAMA Neurology (1997)
The relevance of iron in the pathogenesis of Parkinson's disease.
Mario E. Götz;Kay Double;Manfred Gerlach;Moussa B. H. Youdim.
Annals of the New York Academy of Sciences (2004)
Regional Specificity of Brain Atrophy in Huntington's Disease
G.M. Halliday;D.A. McRitchie;V. Macdonald;K.L. Double.
Experimental Neurology (1998)
Topography of brain atrophy during normal aging and alzheimer's disease
K.L. Double;G.M. Halliday;G.M. Halliday;J.J. Krill;J.J. Krill;J.A. Harasty.
Neurobiology of Aging (1996)
Hippocampal Lewy pathology and cholinergic dysfunction are associated with dementia in Parkinson's disease.
Hélène Hall;Stefanie Reyes;Natalie Landeck;Chris Bye.
Brain (2014)
Oxidative stress in the aging substantia nigra and the etiology of Parkinson's disease
Benjamin G. Trist;Dominic J. Hare;Dominic J. Hare;Kay L. Double.
Aging Cell (2019)
Iron-binding characteristics of neuromelanin of the human substantia nigra.
Kay L. Double;Manfred Gerlach;Volker Schünemann;Alfred X. Trautwein.
Biochemical Pharmacology (2003)
α-Synuclein redistributes to neuromelanin lipid in the substantia nigra early in Parkinson's disease
Glenda M. Halliday;Anita Ophof;Melissa Broe;Poul H. Jensen.
Brain (2005)
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