His main research concerns Genetics, Glucocerebrosidase, Gene, Molecular biology and Immunology. His research on Genetics often connects related areas such as Old Order Amish. His Glucocerebrosidase study combines topics in areas such as Phenotype, Allele, Null allele, Genotype and Gaucher's disease.
He works mostly in the field of Null allele, limiting it down to topics relating to Fetus and, in certain cases, Pathology. His work deals with themes such as Protein primary structure, Biochemistry, Proteolysis, Coding region and Complementary DNA, which intersect with Molecular biology. His research investigates the connection between Immunology and topics such as Glucocerebroside that intersect with problems in Lysosomal storage disease.
Edward I. Ginns focuses on Genetics, Glucocerebrosidase, Molecular biology, Biochemistry and Disease. His work on Genetics deals in particular with Gene, Gene mapping, Exon, Locus and Allele. His study looks at the relationship between Gene mapping and fields such as Genetic linkage, as well as how they intersect with chemical problems.
His research in Glucocerebrosidase focuses on subjects like Phenotype, which are connected to Hydrops fetalis. In his work, Nucleic acid sequence is strongly intertwined with Complementary DNA, which is a subfield of Molecular biology. In his research, Lysosomal storage disease and Glucocerebroside is intimately related to Immunology, which falls under the overarching field of Disease.
The scientist’s investigation covers issues in Internal medicine, Disease, Endocrinology, Genetics and Glucocerebrosidase. His work on Adenosine as part of his general Internal medicine study is frequently connected to Etacrynic acid, thereby bridging the divide between different branches of science. Edward I. Ginns has researched Disease in several fields, including Genetic enhancement and Virology.
The concepts of his Endocrinology study are interwoven with issues in Na+/K+-ATPase, Triphosphatase, Prefrontal cortex and Old Order Amish. Edward I. Ginns integrates Genetics and Ellis van creveld in his studies. His Glucocerebrosidase study is concerned with the larger field of Pathology.
Edward I. Ginns mainly focuses on Internal medicine, Endocrinology, Prefrontal cortex, Genetics and Glucocerebrosidase. The study incorporates disciplines such as Psychosis and Parvalbumin in addition to Internal medicine. His Endocrinology research includes elements of Regulation of gene expression and Neuroscience.
He works mostly in the field of Prefrontal cortex, limiting it down to topics relating to Cerebral cortex and, in certain cases, Schizophrenia, Frontal lobe, Hippocampus and Estrogen, as a part of the same area of interest. Edward I. Ginns performs integrative study on Genetics and CDH2. His biological study deals with issues like Neuroinflammation, which deal with fields such as Synaptic plasticity.
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Re-evaluation of the linkage relationship between chromosome 11p loci and the gene for bipolar affective disorder in the old order amish
John R. Kelsoe;Edward I. Ginns;Janice A. Egeland;Daniela S. Gerhard.
Vasopressin V1b receptor knockout reduces aggressive behavior in male mice.
S. R. Wersinger;Edward I. Ginns;A-M O'Carroll;S. J. Lolait.
Molecular Psychiatry (2002)
Mice that lack thrombospondin 2 display connective tissue abnormalities that are associated with disordered collagen fibrillogenesis, an increased vascular density, and a bleeding diathesis.
Themis R. Kyriakides;Yu Hong Zhu;Lynne T. Smith;Steven D. Bain.
Journal of Cell Biology (1998)
A mutation in the human glucocerebrosidase gene in neuronopathic Gaucher's disease.
Shoji Tsuji;Prabhakara V. Choudary;Brian M. Martin;Barbara K. Stubblefield.
The New England Journal of Medicine (1987)
Consequences of beta-glucocerebrosidase deficiency in epidermis. Ultrastructure and permeability barrier alterations in Gaucher disease.
W M Holleran;E I Ginns;G K Menon;J U Grundmann.
Journal of Clinical Investigation (1994)
Deficiency in mouse oxytocin prevents milk ejection, but not fertility or parturition.
W. Scott Young;Emily Shepard;Janet Amico;Lothar Hennighausen.
Journal of Neuroendocrinology (1996)
Animal model of Gaucher's disease from targeted disruption of the mouse glucocerebrosidase gene.
V. L. J. Tybulewicz;V. L. J. Tybulewicz;M. L. Tremblay;M. L. Tremblay;LaMarca;R. Willemsen.
Molecular Determinants of Dysregulated GABAergic Gene Expression in the Prefrontal Cortex of Subjects with Schizophrenia
Nikolaos Mellios;Hsien-Sung Huang;Stephen P. Baker;Marzena Galdzicka.
Biological Psychiatry (2009)
Lack of an association between a dopamine-4 receptor polymorphism and attention-deficit/hyperactivity disorder: genetic and brain morphometric analyses.
F X Castellanos;E Lau;N Tayebi;P Lee.
Molecular Psychiatry (1998)
Genetic heterogeneity in type 1 Gaucher disease: multiple genotypes in Ashkenazic and non-Ashkenazic individuals.
Shoji Tsuji;Brian M. Martin;John A. Barranger;Barbara K. Stubblefield.
Proceedings of the National Academy of Sciences of the United States of America (1988)
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