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Biology and Biochemistry

D-Index
53
Citations
7329
World Ranking
16387
National Ranking
6773

Overview

Dzung H. Dinh is affiliated with the University of Illinois at Chicago in the United States. Their research focuses primarily within the fields of Biochemistry, Genetics, and Molecular Biology, with significant contributions in Molecular Biology, Cancer Research, and Oncology. Other subfields of study include Gender Studies and Emergency Medicine.

Their work extensively covers various scientific topics, including:

  • Protease and Inhibitor Mechanisms
  • Cancer-related molecular mechanisms research
  • Signaling Pathways in Disease
  • Peptidase Inhibition and Analysis
  • S100 Proteins and Annexins
  • Mitochondrial Function and Pathology
  • Cancer, Hypoxia, and Metabolism

Dinh has published articles in multiple venues, with frequent publications in:

  • Oncogene
  • Journal of Biological Chemistry
  • International Journal of Oncology
  • North American Spine Society Journal (NASSJ)
  • Cureus

The scientific papers authored or coauthored by Dinh include:

  • "[Retracted] Metabolic remodeling precedes mitochondrial outer membrane permeabilization in human glioma xenograft cells," 2021, International Journal of Oncology
  • "Educational impact of early COVID-19 operating room restrictions on neurosurgery resident training in the United States: A multicenter study," 2022, North American Spine Society Journal (NASSJ)
  • "We Tabulated and Organized American Board of Neurological Surgeons Primary Exam Keywords (2015-2023) so You Don't Have to," 2023, Cureus
  • "Retraction: Specific interference of urokinase-type plasminogen activator receptor and matrix metalloproteinase-9 gene expression induced by double-stranded RNA results in decreased invasion, tumor growth, and angiogenesis in gliomas," 2020, Journal of Biological Chemistry
  • "Retraction Note to: uPAR and cathepsin B downregulation induces apoptosis by targeting calcineurin A to BAD via Bcl-2 in glioma," 2021, Journal of Neuro-Oncology

Dinh's frequent collaborators include William C. Olivero, Sajani S. Lakka, Christopher S. Gondi, Meena Gujrati, and Jasti S. Rao.

Best Publications

  • Inhibition of cathepsin B and MMP-9 gene expression in glioblastoma cell line via RNA interference reduces tumor cell invasion, tumor growth and angiogenesis.

    Sajani S Lakka;Christopher S Gondi;Niranjan Yanamandra;William C Olivero

  • Mesenchymal stem cells in the treatment of spinal cord injuries: A review

    Venkata Ramesh Dasari;Krishna Kumar Veeravalli;Dzung H Dinh

  • RNAi-mediated inhibition of cathepsin B and uPAR leads to decreased cell invasion, angiogenesis and tumor growth in gliomas

    Christopher S Gondi;Sajani S Lakka;Dzung H Dinh;William C Olivero;William C Olivero

  • Specific interference of urokinase-type plasminogen activator receptor and matrix metalloproteinase-9 gene expression induced by double-stranded RNA results in decreased invasion, tumor growth, and angiogenesis in gliomas

    Sajani S. Lakka;Christopher S. Gondi;Dzung H. Dinh;William C. Olivero

  • Upregulation of PTEN in Glioma Cells by Cord Blood Mesenchymal Stem Cells Inhibits Migration via Downregulation of the PI3K/Akt Pathway

    Venkata Ramesh Dasari;Kiranpreet Kaur;Kiran Kumar Velpula;Meena Gujrati

  • Adenovirus-mediated transfer of siRNA against MMP-2 mRNA results in impaired invasion and tumor-induced angiogenesis, induces apoptosis in vitro and inhibits tumor growth in vivo in glioblastoma

    Odysseas Kargiotis;Chandramu Chetty;Christopher S. Gondi;Andrew J. Tsung

  • Axonal remyelination by cord blood stem cells after spinal cord injury.

    Venkata Ramesh Dasari;Daniel G. Spomar;Christopher S. Gondi;Christopher A. Sloffer

  • MMP-9 Induces CD44 Cleavage and CD44 mediated Cell Migration in Glioblastoma Xenograft Cells

    Chandramu Chetty;Sravan K. Vanamala;Christopher S. Gondi;Dzung H. Dinh

  • Downregulation of uPA, uPAR and MMP-9 using small, interfering, hairpin RNA (siRNA) inhibits glioma cell invasion, angiogenesis and tumor growth

    Christopher S. Gondi;Sajani S. Lakka;Dzung H. Dinh;William Charles Olivero

  • MMP-2 siRNA Inhibits Radiation-Enhanced Invasiveness in Glioma Cells

    Aruna Venkata Badiga;Chandramu Chetty;Divya Kesanakurti;Deepthi Are

  • Downregulation of MMP-9 in ERK-mutated stable transfectants inhibits glioma invasion in vitro

    Sajani S. Lakka;Sushma L. Jasti;Christopher Gondi;Douglas D Boyd

  • Role of MMP-2 in the Regulation of IL-6/Stat3 Survival Signaling via Interaction With α5β1 Integrin in glioma

    Divya Kesanakurti;Chandramu Chetty;Dzung H. Dinh;Meena Gujrati

  • Downregulation of uPA inhibits migration and PI3k/Akt signaling in glioblastoma cells

    Nirmala Chandrasekar;Sanjeeva Mohanam;Meena Gujrati;William C Olivero

  • Expression of antisense uPAR and antisense uPA from a bicistronic adenoviral construct inhibits glioma cell invasion, tumor growth, and angiogenesis.

    Christopher S Gondi;Sajani S Lakka;Niranjan Yanamandra;Khawar Siddique

  • Adenovirus-mediated expression of antisense MMP-9 in glioma cells inhibits tumor growth and invasion

    Sajani S Lakka;Mannari Rajan;Christopher Gondi;Niranjan Yanamandra

  • Downregulation of uPAR and Cathepsin B Induces Apoptosis via Regulation of Bcl-2 and Bax and Inhibition of the PI3K/Akt Pathway in Gliomas

    Ramarao Malla;Sreelatha Gopinath;Kiranmai Alapati;Christopher S. Gondi

  • Blockade of cathepsin B expression in human glioblastoma cells is associated with suppression of angiogenesis.

    Niranjan Yanamandra;Krishna V Gumidyala;Kevin G Waldron;Meena Gujrati

  • Down-regulation of uPAR and uPA activates caspase-mediated apoptosis and inhibits the PI3K/AKT pathway

    Christopher S Gondi;Neelima Kandhukuri;Dzung H Dinh;Meena Gujrati

  • Intraperitoneal injection of a hairpin RNA-expressing plasmid targeting urokinase-type plasminogen activator (uPA) receptor and uPA retards angiogenesis and inhibits intracranial tumor growth in nude mice.

    Christopher S. Gondi;Sajani S. Lakka;Dzung H. Dinh;William C. Olivero;William C. Olivero

  • Down-regulation of cathepsin B expression impairs the invasive and tumorigenic potential of human glioblastoma cells.

    Sanjeeva Mohanam;Sushma L. Jasti;Sudha R. Kondraganti;Nirmala Chandrasekar

Frequent Co-Authors

Jasti S. Rao
Jasti S. Rao University of Illinois at Chicago
Francis Ali-Osman
Francis Ali-Osman Duke University
Gregory N. Fuller
Gregory N. Fuller The University of Texas MD Anderson Cancer Center
Donald C. Foster
Donald C. Foster Just Biotherapeutics (United States)
Garth L. Nicolson
Garth L. Nicolson University of Helsinki
Walter Kisiel
Walter Kisiel University of New Mexico
Srinivasa M. Srinivasula
Srinivasa M. Srinivasula Indian Institute of Science Education and Research, Thiruvananthapuram
Ching-Hsuan Tung
Ching-Hsuan Tung Cornell University
Douglas D. Boyd
Douglas D. Boyd The University of Texas MD Anderson Cancer Center
Ralph Weissleder
Ralph Weissleder Harvard University

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