His primary scientific interests are in Neuroscience, Dopamine, Brain stimulation reward, Dopaminergic and Internal medicine. His Neuroscience research integrates issues from Nonsynaptic plasticity and Long-term potentiation, Postsynaptic signal transduction. His Brain stimulation reward study combines topics in areas such as Adaptation, Ventral tegmental area and Vulnerability.
His Ventral tegmental area study incorporates themes from Chronic stress, Reward system and Tyrosine hydroxylase. His Dopamine receptor D3 study in the realm of Dopaminergic connects with subjects such as PPP1R1B. Many of his studies involve connections with topics such as Endocrinology and Internal medicine.
His scientific interests lie mostly in Neuroscience, Dopamine, Ventral tegmental area, Endocrinology and Internal medicine. His work in the fields of Neuroscience, such as Bursting, Neuron, Brain stimulation reward and Hippocampal formation, overlaps with other areas such as Prefrontal cortex. His Brain stimulation reward research incorporates themes from Chronic stress and Social defeat.
He combines subjects such as Stimulation and Pharmacology with his study of Dopamine. His study on Dopaminergic is often connected to Forebrain as part of broader study in Endocrinology. Within one scientific family, Donald C. Cooper focuses on topics pertaining to Catecholamine under Dopaminergic, and may sometimes address concerns connected to Lithium.
Donald C. Cooper spends much of his time researching Neuroscience, Pharmacology, Optogenetics, Neuron and Dopamine. Inhibitory postsynaptic potential is the focus of his Neuroscience research. His Pharmacology research incorporates elements of Autism and Terpene.
His research in Neuron intersects with topics in Escape response and Cell biology. His work on TRPC4 expands to the thematically related Dopamine. His Gene research incorporates themes from Dopaminergic, Endocrinology, Physiology and Cellular excitability.
Donald C. Cooper mainly focuses on Neuroscience, Optogenetics, Prefrontal cortex, Test strips and Biotechnology. His Neuroscience study incorporates themes from Extracellular and Coping. The study incorporates disciplines such as Neural activity and In vivo in addition to Optogenetics.
Prefrontal cortex is integrated with Extinction, Addiction, Inhibitory postsynaptic potential, Facilitation and Stressor in his study. Test strips combines with fields such as Early detection, Botrytis cinerea, Analysis tools and Mold in his work.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Molecular Adaptations Underlying Susceptibility and Resistance to Social Defeat in Brain Reward Regions
Vaishnav Krishnan;Ming Hu Han;Danielle L. Graham;Olivier Berton.
Cell (2007)
DARPP-32: Regulator of the Efficacy of Dopaminergic Neurotransmission
A. A. Fienberg;N. Hiroi;Paul G Mermelstein;W. J. Song.
Science (1998)
Regulation of dopaminergic transmission and cocaine reward by the Clock gene
Colleen A. McClung;Kyriaki Sidiropoulou;Kyriaki Sidiropoulou;Martha Vitaterna;Joseph S. Takahashi.
Proceedings of the National Academy of Sciences of the United States of America (2005)
Elimination of cocaine-induced hyperactivity and dopamine-mediated neurophysiological effects in dopamine D1 receptor mutant mice
Ming Xu;Xiu-Ti Hu;Donald C. Cooper;Rosario Moratalla.
Cell (1994)
Dopamine D3 Receptor Mutant Mice Exhibit Increased Behavioral Sensitivity to Concurrent Stimulation of D1 and D2 Receptors
Ming Xu;Timothy E Koeltzow;Giovanni Tirado Santiago;Rosario Moratalla.
Neuron (1997)
Cyclin-dependent kinase 5 governs learning and synaptic plasticity via control of NMDAR degradation.
Ammar H Hawasli;David R Benavides;Chan Nguyen;Janice W Kansy.
Nature Neuroscience (2007)
Mesolimbic Dopamine Neurons in the Brain Reward Circuit Mediate Susceptibility to Social Defeat and Antidepressant Action
Jun-Li Cao;Herbert E. Covington;Allyson K. Friedman;Matthew B. Wilkinson.
The Journal of Neuroscience (2010)
Notch1 is required for maintenance of the reservoir of adult hippocampal stem cells
Jessica L. Ables;Nathan A. DeCarolis;Madeleine A. Johnson;Phillip D. Rivera.
The Journal of Neuroscience (2010)
CREB regulation of nucleus accumbens excitability mediates social isolation-induced behavioral deficits
Deanna L Wallace;Ming-Hu Han;Danielle L Graham;Danielle L Graham;Thomas A Green;Thomas A Green.
Nature Neuroscience (2009)
Loss of autoreceptor functions in mice lacking the dopamine transporter
Sara R. Jones;Sara R. Jones;Raul R. Gainetdinov;Raul R. Gainetdinov;Xiu Ti Hu;Donald C. Cooper.
Nature Neuroscience (1999)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
Icahn School of Medicine at Mount Sinai
Rush University Medical Center
Icahn School of Medicine at Mount Sinai
Howard Hughes Medical Institute
University of Pittsburgh
MIT
Harvard University
The University of Texas Medical Branch at Galveston
Université Paris Cité
The University of Texas Southwestern Medical Center
Harvard University
Technical University of Denmark
Tianjin University
Swiss Federal Laboratories for Materials Science and Technology
US Forest Service
Tufts University
Vrije Universiteit Amsterdam
University of Pennsylvania
Yale University
Queen Mary University of London
Max Planck Society
National Center for Atmospheric Research
National Academies of Sciences, Engineering, and Medicine
New York Medical College
Stanford University
University of Copenhagen