His scientific interests lie mostly in Virology, Virus, Interferon, Influenza A virus and Molecular biology. His research in Virology tackles topics such as Virulence which are related to areas like Immune system. The Virus study combines topics in areas such as Recombinant virus and Gene.
His Interferon study combines topics from a wide range of disciplines, such as Signal transduction, DEAD Box Protein 58, MDA5 and Interferon regulatory factors. His studies in Influenza A virus integrate themes in fields like VP40, Nucleoprotein and Viral transformation. His Molecular biology study integrates concerns from other disciplines, such as Vero cell, MAPK14, Newcastle disease, RNA silencing and Phosphorylation.
The scientist’s investigation covers issues in Virology, Virus, Ebola virus, Interferon and Viral replication. In his work, Immunity is strongly intertwined with Innate immune system, which is a subfield of Virology. His study on Influenza A virus, Hemagglutinin and Orthomyxoviridae is often connected to H5N1 genetic structure as part of broader study in Virus.
His work carried out in the field of Ebola virus brings together such families of science as Polymerase, Filoviridae, Nucleoprotein and Cell biology. In his research on the topic of Interferon, RNA silencing is strongly related with RNA-binding protein. His Viral replication research incorporates themes from RNA and Viral protein.
His main research concerns Virology, Ebola virus, Virus, Interferon and Marburg virus. His study of VP40 is a part of Virology. His study in Ebola virus is interdisciplinary in nature, drawing from both RNA, Filoviridae, Genome, Nucleoprotein and Viral replication.
Many of his research projects under Virus are closely connected to Trizol with Trizol, tying the diverse disciplines of science together. The concepts of his Interferon study are interwoven with issues in Ubiquitin, Receptor, Antagonism, N-terminus and MDA5. The study incorporates disciplines such as Antibody and Virulence in addition to Marburg virus.
Christopher F. Basler mainly focuses on Virology, Virus, Ebola virus, Evolutionary biology and Order Mononegavirales. His work on Genome expands to the thematically related Virology. Christopher F. Basler studies Virus, focusing on Viral replication in particular.
The various areas that Christopher F. Basler examines in his Viral replication study include Vero cell, Nucleoprotein and Endocytosis. His Ebola virus research is multidisciplinary, incorporating elements of RIG-I, Interferon and Small molecule. Christopher F. Basler combines subjects such as Taxonomy, Phylogenetics and Mononegavirales with his study of Evolutionary biology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Characterization of the reconstructed 1918 Spanish influenza pandemic virus
Terrence M. Tumpey;Christopher F. Basler;Patricia V. Aguilar;Hui Zeng.
Activation of Interferon Regulatory Factor 3 Is Inhibited by the Influenza A Virus NS1 Protein
Julie Talon;Curt M. Horvath;Rosalind Polley;Christopher F. Basler.
Journal of Virology (2000)
Pathogenicity of Influenza Viruses with Genes from the 1918 Pandemic Virus: Functional Roles of Alveolar Macrophages and Neutrophils in Limiting Virus Replication and Mortality in Mice
Terrence M. Tumpey;Adolfo García-Sastre;Jeffery K. Taubenberger;Peter Palese.
Journal of Virology (2005)
Structure of the Uncleaved Human H1 Hemagglutinin from the Extinct 1918 Influenza Virus
James Stevens;Adam L. Corper;Christopher F. Basler;Jeffery K. Taubenberger.
Genomic analysis of increased host immune and cell death responses induced by 1918 influenza virus
John C. Kash;Terrence M. Tumpey;Sean C. Proll;Victoria Carter.
The Ebola virus VP35 protein functions as a type I IFN antagonist.
Christopher F. Basler;Xiuyan Wang;Elke Mühlberger;Victor Volchkov.
Proceedings of the National Academy of Sciences of the United States of America (2000)
The Ebola Virus VP35 Protein Inhibits Activation of Interferon Regulatory Factor 3
Christopher F. Basler;Andrea Mikulasova;Luis Martinez-Sobrido;Jason Paragas.
Journal of Virology (2003)
Neutralizing antibodies derived from the B cells of 1918 influenza pandemic survivors
Xiaocong Yu;Tshidi Tsibane;Patricia A. McGraw;Frances S. House.
Cellular transcriptional profiling in influenza A virus-infected lung epithelial cells: The role of the nonstructural NS1 protein in the evasion of the host innate defense and its potential contribution to pandemic influenza
Gary K. Geiss;Mirella Salvatore;Terrence M. Tumpey;Victoria S. Carter.
Proceedings of the National Academy of Sciences of the United States of America (2002)
Ebola Virus VP35 Protein Binds Double-Stranded RNA and Inhibits Alpha/Beta Interferon Production Induced by RIG-I Signaling
Washington B. Cárdenas;Yueh Ming Loo;Michael Gale;Amy L. Hartman.
Journal of Virology (2006)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: