His primary scientific interests are in Cell biology, Apoptosis, Molecular biology, Myocyte and Internal medicine. His Transfection research extends to Cell biology, which is thematically connected. The various areas that Christopher C. Glembotski examines in his Apoptosis study include Tumor necrosis factor alpha and Signal transduction.
Christopher C. Glembotski has included themes like Mitogen-activated protein kinase, Receptor and Protein kinase A, p38 mitogen-activated protein kinases in his Molecular biology study. His Myocyte research includes themes of Heat shock protein and Phosphorylation. His Internal medicine study incorporates themes from Endocrinology, Transcription factor and Transgene.
His primary areas of study are Cell biology, Internal medicine, Endocrinology, Endoplasmic reticulum and Myocyte. His Cell biology study integrates concerns from other disciplines, such as Heat shock protein and Transcription factor. His work on Heart failure, Paracrine signalling, Autocrine signalling and Peptide hormone as part of general Internal medicine study is frequently connected to Phenylephrine, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
His research integrates issues of Receptor, Protein kinase C and Calcium in his study of Endocrinology. His work on Unfolded protein response and ATF6 as part of general Endoplasmic reticulum research is frequently linked to Membrane protein, bridging the gap between disciplines. Christopher C. Glembotski interconnects Apoptosis, Cell, Cardiovascular physiology and Protein biosynthesis in the investigation of issues within Myocyte.
Christopher C. Glembotski spends much of his time researching Cell biology, Unfolded protein response, Endoplasmic reticulum, ATF6 and Myocyte. His Cell biology study combines topics in areas such as Transcription factor and Gene expression. His work carried out in the field of Unfolded protein response brings together such families of science as Secretion, Internal medicine, Heart disease and Knockout mouse.
His Internal medicine research focuses on In vivo and how it relates to Tumor necrosis factor alpha, BMS-345541, Ca2+/calmodulin-dependent protein kinase and NFKB1. His studies deal with areas such as Stroke, Neuroscience, Gene knockdown and Effector as well as ATF6. His Myocyte research is multidisciplinary, relying on both Fibroblast, Transforming growth factor, Myocardial infarction and Cardiovascular physiology.
His main research concerns Unfolded protein response, Endoplasmic reticulum, Internal medicine, ATF6 and Proteostasis. His research investigates the connection between Unfolded protein response and topics such as Myocardial infarction that intersect with issues in Circulatory system. His study with Endoplasmic reticulum involves better knowledge in Cell biology.
The various areas that Christopher C. Glembotski examines in his Internal medicine study include Endocrinology and Cardiology. His study in ATF6 is interdisciplinary in nature, drawing from both Stroke, Neuroscience and Activating transcription factor. The Proteostasis study combines topics in areas such as Function, Cytosol, Ischemia, Myocyte and Mitochondrion.
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Tumor necrosis factor alpha-induced apoptosis in cardiac myocytes. Involvement of the sphingolipid signaling cascade in cardiac cell death.
K. A. Krown;M. T. Page;C. Nguyen;D. Zechner.
Journal of Clinical Investigation (1996)
Identification in pituitary tissue of a peptide alpha-amidation activity that acts on glycine-extended peptides and requires molecular oxygen, copper, and ascorbic acid
Betty A. Eipper;Richard E. Mains;Christopher C. Glembotski.
Proceedings of the National Academy of Sciences of the United States of America (1983)
Cardiotrophin-1 Activates a Distinct Form of Cardiac Muscle Cell Hypertrophy ASSEMBLY OF SARCOMERIC UNITS IN SERIES VIA gp130/LEUKEMIA INHIBITORY FACTOR RECEPTOR-DEPENDENT PATHWAYS
Kai C. Wollert;Tetsuya Taga;Mikiyoshi Saito;Masashi Narazaki.
Journal of Biological Chemistry (1996)
p38 MAPK and NF-κB Collaborate to Induce Interleukin-6 Gene Expression and Release EVIDENCE FOR A CYTOPROTECTIVE AUTOCRINE SIGNALING PATHWAY IN A CARDIAC MYOCYTE MODEL SYSTEM
Rian Craig;Andrea M Larkin;Amy M Mingo;Donna J Thuerauf.
Journal of Biological Chemistry (2000)
A role for the p38 mitogen-activated protein kinase pathway in myocardial cell growth, sarcomeric organization, and cardiac-specific gene expression.
Dietmar Zechner;Donna J. Thuerauf;Deanna S. Hanford;Patrick M. McDonough.
Journal of Cell Biology (1997)
Activation of the Unfolded Protein Response in Infarcted Mouse Heart and Hypoxic Cultured Cardiac Myocytes
Donna J. Thuerauf;Marie Marcinko;Natalie Gude;Marta Rubio.
Circulation Research (2006)
Endoplasmic Reticulum Stress Gene Induction and Protection From Ischemia/Reperfusion Injury in the Hearts of Transgenic Mice With a Tamoxifen-Regulated Form of ATF6
Joshua J. Martindale;Rayne Fernandez;Donna Thuerauf;Ross Whittaker.
Circulation Research (2006)
MKK6 Activates Myocardial Cell NF-κB and Inhibits Apoptosis in a p38 Mitogen-activated Protein Kinase-dependent Manner *
Dietmar Zechner;Rian Craig;Deanna S. Hanford;Patrick M. McDonough.
Journal of Biological Chemistry (1998)
LPS-induced autophagy is mediated by oxidative signaling in cardiomyocytes and is associated with cytoprotection.
Hua Yuan;Cynthia N. Perry;Chengqun Huang;Eri Iwai-Kanai.
American Journal of Physiology-heart and Circulatory Physiology (2009)
Pim-1 regulates cardiomyocyte survival downstream of Akt
John A Muraski;Marcello Rota;Yu Misao;Jenna Fransioli.
Nature Medicine (2007)
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