His primary scientific interests are in Neuroscience, Endocrinology, Internal medicine, Synaptic plasticity and Electrophysiology. His work on Hippocampal formation as part of general Neuroscience research is often related to Ca2 channels, thus linking different fields of science. His work carried out in the field of Endocrinology brings together such families of science as Pharmacology and MEDLINE.
Christian Alzheimer has researched Synaptic plasticity in several fields, including Inhibitory postsynaptic potential, Neurotransmission, Long-term potentiation, Signal transduction and Neurogenesis. The concepts of his Neurotransmission study are interwoven with issues in Muscarinic acetylcholine receptor M4, Muscarinic acetylcholine receptor M2, Muscarinic acetylcholine receptor, Muscarinic agonist and Bicuculline. His Electrophysiology research incorporates themes from Peripheral, Electric stimulation and Excitatory postsynaptic potential.
His main research concerns Neuroscience, Cell biology, Biophysics, Internal medicine and Endocrinology. His Neuroscience research is multidisciplinary, incorporating elements of Postsynaptic potential, Neurotransmission and G protein-coupled inwardly-rectifying potassium channel. He has included themes like Muscarinic acetylcholine receptor, Muscarinic acetylcholine receptor M2, Glutamatergic, Inhibitory postsynaptic potential and Long-term potentiation in his Neurotransmission study.
His Cell biology research includes themes of Hippocampal formation, Downregulation and upregulation, Neuroprotection and Potassium channel. His biological study spans a wide range of topics, including Receptor and Adenosine. His Electrophysiology study integrates concerns from other disciplines, such as Synaptic plasticity, HaCaT and Cromakalim.
Christian Alzheimer spends much of his time researching Cell biology, Inhibitory postsynaptic potential, Neuroscience, Neurotransmission and Antipsychotic. His research investigates the connection between Cell biology and topics such as Potassium channel that intersect with problems in Proteolysis and Apoptosis. Christian Alzheimer works mostly in the field of Neuroscience, limiting it down to topics relating to Postsynaptic potential and, in certain cases, Nociception.
His research in Neurotransmission intersects with topics in Glutamate decarboxylase, Scaffold protein and Pyridoxal. His Dopamine receptor D2 research incorporates elements of Haloperidol, Long-term potentiation and Dopamine transporter. His Excitatory postsynaptic potential research is multidisciplinary, relying on both Muscarinic acetylcholine receptor M2, Muscarine, Hippocampal formation, Bursting and Axon.
His scientific interests lie mostly in Amyloid precursor protein, Antipsychotic, Neuroscience, Dopamine transporter and Fluorescence microscope. His Amyloid precursor protein research includes elements of Hippocampal mossy fiber, Excitatory postsynaptic potential, Hippocampal formation, Ion channel and Cell biology. Christian Alzheimer interconnects Postsynaptic potential and Long-term depression in the investigation of issues within Neuroscience.
His Dopamine transporter research incorporates themes from Long-term potentiation, Dopamine receptor D2 and Neuron. Christian Alzheimer integrates many fields, such as Fluorescence microscope, Amyloid precursor protein secretase, Small molecule, Biophysics and Live cell imaging, in his works. As part of his studies on Dopaminergic, Christian Alzheimer often connects relevant subjects like Pharmacology.
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Phenotypic and Biochemical Analyses of BACE1- and BACE2-deficient Mice
Diana Dominguez;Jos Tournoy;Dieter Hartmann;Tobias Huth.
Journal of Biological Chemistry (2005)
Acid sphingomyelinase-ceramide system mediates effects of antidepressant drugs
Erich Gulbins;Erich Gulbins;Monica Palmada;Martin Reichel;Anja Lüth.
Nature Medicine (2013)
Tau-Induced Defects in Synaptic Plasticity, Learning, and Memory Are Reversible in Transgenic Mice after Switching Off the Toxic Tau Mutant
Astrid Sydow;Anneke Van der Jeugd;Fang Zheng;Tariq Ahmed.
The Journal of Neuroscience (2011)
Roles of activin in tissue repair, fibrosis, and inflammatory disease
Sabine Werner;Christian Alzheimer.
Cytokine & Growth Factor Reviews (2006)
Dendritic Na+ channels amplify EPSPs in hippocampal CA1 pyramidal cells.
R. Lipowsky;T. Gillessen;C. Alzheimer.
Journal of Neurophysiology (1996)
M2 muscarinic acetylcholine receptor knock-out mice show deficits in behavioral flexibility, working memory, and hippocampal plasticity.
Thomas Seeger;Irina Fedorova;Fang Zheng;Tsuyoshi Miyakawa.
The Journal of Neuroscience (2004)
Induction of activin A is essential for the neuroprotective action of basic fibroblast growth factor in vivo
Y. P. Tretter;Moritz Hertel;B. Munz;B. Munz;G. ten Bruggencate.
Nature Medicine (2000)
Anticancer drug oxaliplatin induces acute cooling-aggravated neuropathy via sodium channel subtype NaV1.6-resurgent and persistent current
Ruth Sittl;Angelika Lampert;Tobias Huth;E. Theresa Schuy.
Proceedings of the National Academy of Sciences of the United States of America (2012)
Fibroblast growth factors and neuroprotection.
Christian Alzheimer;Sabine Werner.
Advances in Experimental Medicine and Biology (2003)
Amplification of EPSPs by Low Ni2+- and Amiloride-Sensitive Ca2+ Channels in Apical Dendrites of Rat CA1 Pyramidal Neurons
Thomas Gillessen;Christian Alzheimer.
Journal of Neurophysiology (1997)
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