Cell biology, Phosphoinositide 3-kinase, G protein, Biochemistry and Protein subunit are his primary areas of study. His work on G protein-coupled receptor, PI3K/AKT/mTOR pathway and Signal transduction as part of general Cell biology research is frequently linked to Set, thereby connecting diverse disciplines of science. The various areas that Bernd Nürnberg examines in his Phosphoinositide 3-kinase study include Phosphatidylinositol, Intracellular and Voltage-dependent calcium channel.
In his research, Molecular biology is intimately related to L-type calcium channel, which falls under the overarching field of Intracellular. His G protein study incorporates themes from In vitro, GTPase and Stimulation. His research in Protein subunit intersects with topics in Enzyme activator, Receptor tyrosine kinase, Wortmannin and Pleckstrin homology domain.
Bernd Nürnberg spends much of his time researching Cell biology, Biochemistry, G protein, Molecular biology and Heterotrimeric G protein. His Cell biology research incorporates elements of Protein subunit and Voltage-dependent calcium channel. His work deals with themes such as Enzyme activator, Mutant, Class Ib Phosphatidylinositol 3-Kinase and Function, which intersect with Protein subunit.
His study with G protein involves better knowledge in Receptor. The Molecular biology study which covers Phosphorylation that intersects with Kinase. Bernd Nürnberg has included themes like HEK 293 cells, Pleckstrin homology domain, Phosphatidylinositol and Intracellular in his Phosphoinositide 3-kinase study.
Bernd Nürnberg focuses on Cell biology, Platelet, G protein, Thapsigargin and Cancer research. His studies in Cell biology integrate themes in fields like IRF4 and Pattern recognition receptor. His work on Platelet activation as part of general Platelet study is frequently linked to Ovarian cancer, bridging the gap between disciplines.
His work on Gi alpha subunit, Heterotrimeric G protein and Pertussis toxin as part of general G protein research is often related to Blood–brain barrier, thus linking different fields of science. His Thapsigargin research includes elements of STIM1, SGK1 and Downregulation and upregulation. His Cancer research study integrates concerns from other disciplines, such as Prostaglandin E2, Airway, Myeloid-derived Suppressor Cell and Agonist.
His primary scientific interests are in Cell biology, G protein, Disease, Protein subunit and Effector. His Cell biology research is mostly focused on the topic Receptor tyrosine kinase. His study in G protein is interdisciplinary in nature, drawing from both Cell signaling, Immune system, Function, Enzyme and PI3K/AKT/mTOR pathway.
His Disease research incorporates themes from Diabetes mellitus, Genetics and Metabolic disorder. His Protein subunit research integrates issues from GTPase, G protein-coupled receptor, Kinase activity and Phosphoinositide 3-kinase. Bernd Nürnberg has researched Effector in several fields, including Intracellular parasite, Secretion, Microbiology, Heterotrimeric G protein and Gi alpha subunit.
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Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes
Daniel J. Klionsky;Hagai Abeliovich;Patrizia Agostinis;Devendra K. Agrawal.
Roles of Gβγ in membrane recruitment and activation of p110γ/p101 phosphoinositide 3-kinase γ
Carsten Brock;Carsten Brock;Michael Schaefer;H. Peter Reusch;Cornelia Czupalla;Cornelia Czupalla.
Journal of Cell Biology (2003)
Roles of Non-catalytic Subunits in Gβγ-induced Activation of Class I Phosphoinositide 3-Kinase Isoforms β and γ
Udo Maier;Aleksei Babich;Bernd Nürnberg.
Journal of Biological Chemistry (1999)
Gβγ Stimulates Phosphoinositide 3-Kinase-γ by Direct Interaction with Two Domains of the Catalytic p110 Subunit
Daniela Leopoldt;Theodor Hanck;Torsten Exner;Udo Maier.
Journal of Biological Chemistry (1998)
PI3K promotes voltage-dependent calcium channel trafficking to the plasma membrane.
Patricia Viard;Adrian J Butcher;Guillaume Halet;Anthony Davies.
Nature Neuroscience (2004)
Bivalent role of the phosphatidylinositol-3-kinase (PI3K) during influenza virus infection and host cell defence.
Christina Ehrhardt;Henju Marjuki;Thorsten Wolff;Bernd Nürnberg.
Cellular Microbiology (2006)
Receptors and G proteins as primary components of transmembrane signal transduction. Part 1. G-protein-coupled receptors: structure and function.
Gudermann T;Nürnberg B;Schultz G.
Journal of Molecular Medicine (1995)
Leptin Induces Endothelial Cell Migration Through Akt, Which Is Inhibited by PPARγ-Ligands
Stephan Goetze;Anne Bungenstock;Cornelia Czupalla;Friedrich Eilers.
Receptors and G proteins as primary components of transmembrane signal transduction
B. Nürnberg;T. Gudermann;G. Schultz.
Journal of Molecular Medicine (1995)
Gbetagamma dimers stimulate vascular L-type Ca2+ channels via phosphoinositide 3-kinase.
Patricia Viard;Torsten Exner;Udo Maier;Jean Mironneau.
The FASEB Journal (1999)
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