Fellow of The Academy of Medical Sciences, United Kingdom
Anthony H. Dickenson mostly deals with Nociception, Neuroscience, Neuropathic pain, Pharmacology and Spinal cord. His Nociception research is multidisciplinary, incorporating elements of Anesthesia and Endocrinology, Central nervous system, Inhibitory postsynaptic potential. His work carried out in the field of Neuroscience brings together such families of science as NMDA receptor, Hyperalgesia and Nociceptor.
The concepts of his Neuropathic pain study are interwoven with issues in Peripheral neuropathy, Gabapentin, Physical therapy, Chronic pain and Allodynia. The various areas that he examines in his Pharmacology study include Capsaicin, TRPV1, Opioid and Opiate. Anthony H. Dickenson interconnects Excitatory postsynaptic potential and Premovement neuronal activity in the investigation of issues within Spinal cord.
His primary areas of investigation include Neuroscience, Nociception, Neuropathic pain, Pharmacology and Spinal cord. His study in Neuroscience is interdisciplinary in nature, drawing from both NMDA receptor and Hyperalgesia, Allodynia. His Nociception research is multidisciplinary, relying on both Anesthesia and Endocrinology, Stimulation, Central nervous system.
His Neuropathic pain study integrates concerns from other disciplines, such as Peripheral neuropathy, Analgesic, Nerve injury, Gabapentin and Chronic pain. The Pharmacology study combines topics in areas such as Agonist, -Naloxone, Antagonist, Opioid and Nociceptor. His research integrates issues of Anatomy, Receptor, Excitatory postsynaptic potential, Premovement neuronal activity and Brainstem in his study of Spinal cord.
Anthony H. Dickenson mainly investigates Neuropathic pain, Neuroscience, Pharmacology, Nociception and Chronic pain. His Neuropathic pain study is concerned with the larger field of Anesthesia. As a part of the same scientific study, Anthony H. Dickenson usually deals with the Anesthesia, concentrating on Electrophysiology and frequently concerns with Voltage-dependent calcium channel.
His Pharmacology study combines topics from a wide range of disciplines, such as Calcium channel, Monoamine neurotransmitter, Antagonist and Opioid. The study incorporates disciplines such as Stimulation and Central nervous system in addition to Nociception. His work in Spinal cord addresses subjects such as Peripheral nerve injury, which are connected to disciplines such as Allodynia.
His main research concerns Neuropathic pain, Neuroscience, Pharmacology, Nociception and Hyperalgesia. Anthony H. Dickenson has included themes like Gabapentin, Analgesic, Chronic pain and Calcium channel in his Neuropathic pain study. His Neuroscience research includes themes of Monoamine neurotransmitter and Peripheral nerve injury.
His Pharmacology study incorporates themes from Agonist and Stimulation. His Nociception research incorporates elements of Bone pain, Physical therapy, Cancer pain and Bioinformatics. His Hyperalgesia research includes elements of Threshold of pain and Opioid.
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Evidence for a role of the NMDA receptor in the frequency dependent potentiation of deep rat dorsal horn nociceptive neurones following c fibre stimulation.
A.H. Dickenson;Ann F. Sullivan.
The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways
Armen N. Akopian;Veronika Souslova;Steven England;Steven England;Kenji Okuse.
Nature Neuroscience (1999)
Evidence for spinal N-methyl-d-aspartate receptor involvement in prolonged chemical nociception in the rat
Jane E. Haley;Ann F. Sullivan;Anthony H. Dickenson.
Brain Research (1990)
Subcutaneous formalin-induced activity of dorsal horn neurones in the rat: differential response to an intrathecal opiate administered pre or post formalin.
Anthony H. Dickenson;Ann F. Sullivan.
Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain
Mohammed A. Nassar;L. Caroline Stirling;Greta Forlani;Mark D. Baker.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Bad news from the brain: descending 5-HT pathways that control spinal pain processing.
Rie Suzuki;Lars J. Rygh;Anthony H. Dickenson.
Trends in Pharmacological Sciences (2004)
Spinal cord mechanisms of pain
R. D’Mello;A.H. Dickenson.
BJA: British Journal of Anaesthesia (2008)
Broad-Spectrum, Non-Opioid Analgesic Activity by Selective Modulation of Neuronal Nicotinic Acetylcholine Receptors
A. W. Bannon;M. W. Decker;M. W. Holladay;P. Curzon.
The pharmacology of excitatory and inhibitory amino acid-mediated events in the transmission and modulation of pain in the spinal cord.
A.H. Dickenson;V. Chapman;G.M. Green.
General Pharmacology-the Vascular System (1997)
Warm-coding deficits and aberrant inflammatory pain in mice lacking P2X3 receptors.
Veronika Souslova;Paolo Cesare;Yanning Ding;Armen N. Akopian.
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