Yoshinori Fukui focuses on Cell biology, Dock2, Lymphocyte, Motility and B cell. His Cell biology research incorporates themes from Acquired immune system, Immunology and Cytoskeleton. His studies deal with areas such as ELMO1, Phosphoinositide 3-kinase, Chemotaxis, Actin and Rac GTP-Binding Proteins as well as Dock2.
His work carried out in the field of Phosphoinositide 3-kinase brings together such families of science as Lymphocyte homing receptor, Synapse, Integrin and Immunological Synapses. His studies in Lymphocyte integrate themes in fields like Chemokine, T cell and Actin cytoskeleton. His Motility research focuses on RAC1 and how it connects with Chimera, Secretion and Microbiology.
His primary areas of investigation include Cell biology, Dock2, Immunology, Guanine nucleotide exchange factor and Molecular biology. Yoshinori Fukui has researched Cell biology in several fields, including Chemokine, Immune system and Lymphocyte. His study looks at the intersection of Chemokine and topics like Integrin with Actin remodeling and Lamellipodium.
The study incorporates disciplines such as Lymphocyte homing receptor and B cell in addition to Lymphocyte. His Dock2 research includes elements of ELMO1, Chemotaxis, Actin cytoskeleton, Motility and Actin. His Molecular biology research incorporates elements of T cell, T-cell receptor, CD8, Cytotoxic T cell and Major histocompatibility complex.
Cell biology, Dock2, Guanine nucleotide exchange factor, Signal transduction and Inflammation are his primary areas of study. His specific area of interest is Cell biology, where Yoshinori Fukui studies Microtubule. His biological study spans a wide range of topics, including Cd4 t cell, Chemotaxis, Actin cytoskeleton, Small GTPase and Lymph.
Within one scientific family, Yoshinori Fukui focuses on topics pertaining to Rac GTP-Binding Proteins under Guanine nucleotide exchange factor, and may sometimes address concerns connected to HEK 293 cells, Receptor tyrosine kinase and AXL receptor tyrosine kinase. His study looks at the relationship between Signal transduction and topics such as Immunoglobulin E, which overlap with Degranulation. While the research belongs to areas of Inflammation, Yoshinori Fukui spends his time largely on the problem of Receptor, intersecting his research to questions surrounding Metabolism and Mitochondrion.
Yoshinori Fukui spends much of his time researching Cell biology, Signal transduction, Guanine nucleotide exchange factor, Immunology and Dock5. His Cell biology research includes themes of Dendritic cell, Dock2, Cell growth and Cellular differentiation. The concepts of his Dock2 study are interwoven with issues in Actin cytoskeleton and Small GTPase.
Yoshinori Fukui works mostly in the field of Signal transduction, limiting it down to concerns involving Immunoglobulin E and, occasionally, EPAS1 and Degranulation. His Immunology research integrates issues from Dermatology and Receptor. His research integrates issues of Cancer cell, Glutamine and Microtubule in his study of Dock5.
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Haematopoietic cell-specific CDM family protein DOCK2 is essential for lymphocyte migration
Yoshinori Fukui;Osamu Hashimoto;Osamu Hashimoto;Terukazu Sanui;Takamasa Oono.
Neutrophils scan for activated platelets to initiate inflammation
Vinatha Sreeramkumar;José M. Adrover;Ivan Ballesteros;Maria Isabel Cuartero.
Sequential regulation of DOCK2 dynamics by two phospholipids during neutrophil chemotaxis.
Akihiko Nishikimi;Hideo Fukuhara;Wenjuan Su;Tsunaki Hongu.
Differential requirements for DOCK2 and phosphoinositide-3-kinase γ during T and B lymphocyte homing
César Nombela-Arrieta;Rosa Ana Lacalle;Marı́a C. Montoya;Yuya Kunisaki.
DOCK8 is a Cdc42 activator critical for interstitial dendritic cell migration during immune responses
Yosuke Harada;Yoshihiko Tanaka;Masao Terasawa;Markus Pieczyk.
DOCK2 is a Rac activator that regulates motility and polarity during neutrophil chemotaxis
Yuya Kunisaki;Akihiko Nishikimi;Yoshihiko Tanaka;Ryosuke Takii.
Journal of Cell Biology (2006)
Emerging role of interleukin-31 and interleukin-31 receptor in pruritus in atopic dermatitis.
M. Furue;K. Yamamura;M. Kido-Nakahara;T. Nakahara.
Shigella IpgB1 promotes bacterial entry through the ELMO-Dock180 machinery.
Yutaka Handa;Masato Suzuki;Kenji Ohya;Hiroki Iwai.
Nature Cell Biology (2007)
A central role for DOCK2 during interstitial lymphocyte motility and sphingosine-1-phosphate-mediated egress.
César Nombela-Arrieta;Thorsten R. Mempel;Silvia F. Soriano;Irina Mazo.
Journal of Experimental Medicine (2007)
A functional genomics predictive network model identifies regulators of inflammatory bowel disease.
Lauren A. Peters;Jacqueline Perrigoue;Arthur Mortha;Arthur Mortha;Alina Iuga.
Nature Genetics (2017)
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