His primary areas of study are Cell biology, Apoptosis, Bcl-2-associated X protein, Programmed cell death and Bcl-2 Homologous Antagonist-Killer Protein. He works on Cell biology which deals in particular with Mitochondrion. The concepts of his Apoptosis study are interwoven with issues in Signal transduction and Gene expression.
His work carried out in the field of Bcl-2-associated X protein brings together such families of science as Molecular biology and Cytochrome c. His biological study spans a wide range of topics, including Autophagy, Autophagosome, Homeostasis, Necrosis and Physiology. His Bcl-2 Homologous Antagonist-Killer Protein research is multidisciplinary, incorporating perspectives in Unfolded protein response and Bcl-2 family, BH3 Mimetic ABT-737.
His main research concerns Cell biology, Programmed cell death, Apoptosis, Autophagy and Cancer research. His research on Cell biology frequently connects to adjacent areas such as Bcl-2-associated X protein. As a member of one scientific family, Wei-Xing Zong mostly works in the field of Bcl-2-associated X protein, focusing on Bcl-2 Homologous Antagonist-Killer Protein and, on occasion, BH3 Mimetic ABT-737 and Bcl-2 family.
His study in Apoptosis is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha and Molecular biology. Wei-Xing Zong has included themes like Endocytosis, Computational biology and Kinase in his Autophagy study. He has researched Computational biology in several fields, including Chaperone-mediated autophagy and Autolysosome.
Wei-Xing Zong mostly deals with Cell biology, Autophagy, Cancer research, Glutamine and Biochemistry. His study ties his expertise on Cell together with the subject of Cell biology. His studies examine the connections between Autophagy and genetics, as well as such issues in Computational biology, with regards to Autolysosome, Chaperone-mediated autophagy and Scoring system.
The concepts of his Autolysosome study are interwoven with issues in Multicellular organism and Programmed cell death. His Chaperone-mediated autophagy research includes elements of BECN1, MAP1LC3B, Sequestosome 1, Autophagosome and Physiology. His studies deal with areas such as Carcinogenesis, Immune system, Immunotherapy and HMGB1 as well as Cancer research.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death
Michael C. Wei;Wei-Xing Zong;Emily H.-Y. Cheng;Tullia Lindsten.
The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues.
Tullia Lindsten;Andrea J. Ross;Ayala King;Wei Xing Zong.
Molecular Cell (2000)
Necrotic death as a cell fate
Wei-Xing Zong;Craig B. Thompson.
Genes & Development (2006)
BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak
Wei-Xing Zong;Tullia Lindsten;Andrea J. Ross;Grant R. MacGregor.
Genes & Development (2001)
The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-κB that blocks TNFα-induced apoptosis
Wei-Xing Zong;Leonard C. Edelstein;Cailin Chen;Judy Bash.
Genes & Development (1999)
Alkylating DNA damage stimulates a regulated form of necrotic cell death
Wei-Xing Zong;Dara Ditsworth;Daniel E. Bauer;Zhao-Qi Wang.
Genes & Development (2004)
Bax and Bak can localize to the endoplasmic reticulum to initiate apoptosis
Wei-Xing Zong;Chi Li;Georgia Hatzivassiliou;Tullia Lindsten.
Journal of Cell Biology (2003)
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