D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 58 Citations 47,211 104 World Ranking 8625 National Ranking 3876

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Apoptosis
  • Cancer

His primary areas of study are Cell biology, Apoptosis, Bcl-2-associated X protein, Programmed cell death and Bcl-2 Homologous Antagonist-Killer Protein. He works on Cell biology which deals in particular with Mitochondrion. The concepts of his Apoptosis study are interwoven with issues in Signal transduction and Gene expression.

His work carried out in the field of Bcl-2-associated X protein brings together such families of science as Molecular biology and Cytochrome c. His biological study spans a wide range of topics, including Autophagy, Autophagosome, Homeostasis, Necrosis and Physiology. His Bcl-2 Homologous Antagonist-Killer Protein research is multidisciplinary, incorporating perspectives in Unfolded protein response and Bcl-2 family, BH3 Mimetic ABT-737.

His most cited work include:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (3385 citations)
  • Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Cell biology, Programmed cell death, Apoptosis, Autophagy and Cancer research. His research on Cell biology frequently connects to adjacent areas such as Bcl-2-associated X protein. As a member of one scientific family, Wei-Xing Zong mostly works in the field of Bcl-2-associated X protein, focusing on Bcl-2 Homologous Antagonist-Killer Protein and, on occasion, BH3 Mimetic ABT-737 and Bcl-2 family.

His study in Apoptosis is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha and Molecular biology. Wei-Xing Zong has included themes like Endocytosis, Computational biology and Kinase in his Autophagy study. He has researched Computational biology in several fields, including Chaperone-mediated autophagy and Autolysosome.

He most often published in these fields:

  • Cell biology (66.67%)
  • Programmed cell death (36.19%)
  • Apoptosis (35.24%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cell biology (66.67%)
  • Autophagy (20.95%)
  • Cancer research (20.95%)

In recent papers he was focusing on the following fields of study:

Wei-Xing Zong mostly deals with Cell biology, Autophagy, Cancer research, Glutamine and Biochemistry. His study ties his expertise on Cell together with the subject of Cell biology. His studies examine the connections between Autophagy and genetics, as well as such issues in Computational biology, with regards to Autolysosome, Chaperone-mediated autophagy and Scoring system.

The concepts of his Autolysosome study are interwoven with issues in Multicellular organism and Programmed cell death. His Chaperone-mediated autophagy research includes elements of BECN1, MAP1LC3B, Sequestosome 1, Autophagosome and Physiology. His studies deal with areas such as Carcinogenesis, Immune system, Immunotherapy and HMGB1 as well as Cancer research.

Between 2015 and 2021, his most popular works were:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Mitochondria and Cancer (346 citations)
  • TRIM21 Ubiquitylates SQSTM1/p62 and Suppresses Protein Sequestration to Regulate Redox Homeostasis (80 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)

8302 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

7788 Citations

Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death

Michael C. Wei;Wei-Xing Zong;Emily H.-Y. Cheng;Tullia Lindsten.
Science (2001)

4552 Citations

The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues.

Tullia Lindsten;Andrea J. Ross;Ayala King;Wei Xing Zong.
Molecular Cell (2000)

1730 Citations

Necrotic death as a cell fate

Wei-Xing Zong;Craig B. Thompson.
Genes & Development (2006)

1156 Citations

BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak

Wei-Xing Zong;Tullia Lindsten;Andrea J. Ross;Grant R. MacGregor.
Genes & Development (2001)

1071 Citations

The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-κB that blocks TNFα-induced apoptosis

Wei-Xing Zong;Leonard C. Edelstein;Cailin Chen;Judy Bash.
Genes & Development (1999)

884 Citations

Alkylating DNA damage stimulates a regulated form of necrotic cell death

Wei-Xing Zong;Dara Ditsworth;Daniel E. Bauer;Zhao-Qi Wang.
Genes & Development (2004)

784 Citations

Bax and Bak can localize to the endoplasmic reticulum to initiate apoptosis

Wei-Xing Zong;Chi Li;Georgia Hatzivassiliou;Tullia Lindsten.
Journal of Cell Biology (2003)

725 Citations

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