His main research concerns Cell biology, Kinase, Phosphorylation, Protein kinase B and Akt/PKB signaling pathway. PI3K/AKT/mTOR pathway, Serine/threonine-specific protein kinase, Extracellular, Phosphatase and Transforming growth factor are among the areas of Cell biology where he concentrates his study. His research integrates issues of Colony-stimulating factor and Tyrosine kinase, Signal transduction in his study of Kinase.
His research on Phosphorylation focuses in particular on Mitogen-activated protein kinase kinase. His work deals with themes such as Cancer research and MAPK/ERK pathway, which intersect with Protein kinase B. His work carried out in the field of Akt/PKB signaling pathway brings together such families of science as c-Raf and MAP kinase kinase kinase.
His primary areas of investigation include Cell biology, Biochemistry, Molecular biology, Kinase and Phosphorylation. His Cell biology study which covers Apoptosis that intersects with Cytokine. The Biochemistry study which covers Platelet-activating factor that intersects with Platelet.
Within one scientific family, he focuses on topics pertaining to Interleukin 3 under Molecular biology, and may sometimes address concerns connected to Interleukin 5. His study looks at the relationship between Kinase and topics such as Growth factor, which overlap with Immunology. His Protein kinase B study also includes fields such as
Vincent Duronio mainly focuses on Cell biology, Tendon, Transforming growth factor, Haematopoiesis and Angiopoietin. His Ceramide research extends to the thematically linked field of Cell biology. His Ceramide research includes elements of Signal transduction, Lipid signaling and Kinase.
His Tendon research is multidisciplinary, incorporating elements of Angiogenesis and Neovascularization. His research investigates the link between Transforming growth factor and topics such as Cancer research that cross with problems in Collagen i. His PI3K/AKT/mTOR pathway research integrates issues from Extracellular matrix and Phosphorylation.
Vincent Duronio focuses on Cell biology, Tendon, Macrophage colony-stimulating factor, Lipid signaling and Ceramide. His Phosphorylation and Extracellular matrix study in the realm of Cell biology interacts with subjects such as Mechanotherapy. His Tendon research is multidisciplinary, incorporating perspectives in Protein kinase B and PI3K/AKT/mTOR pathway.
Macrophage colony-stimulating factor is connected with Signal transduction, Sphingomyelin phosphodiesterase and Acid sphingomyelinase in his study.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Rapid formation of diacylglycerol from phosphatidylcholine: a pathway for generation of a second messenger.
Jeffrey M. Besterman;Vincent Duronio;Pedro Cuatrecasas.
Proceedings of the National Academy of Sciences of the United States of America (1986)
The life of a cell: apoptosis regulation by the PI3K/PKB pathway.
Biochemical Journal (2008)
Regulation of Bad Phosphorylation and Association with Bcl-xL by the MAPK/Erk Kinase
Michael P. Scheid;Kathryn M. Schubert;Vincent Duronio.
Journal of Biological Chemistry (1999)
Dissociation of cytokine-induced phosphorylation of Bad and activation of PKB/akt: Involvement of MEK upstream of Bad phosphorylation
Michael P. Scheid;Vincent Duronio.
Proceedings of the National Academy of Sciences of the United States of America (1998)
p21ras activation via hemopoietin receptors and c-kit requires tyrosine kinase activity but not tyrosine phosphorylation of p21ras GTPase-activating protein.
Vincent Duronio;Melanie J. Welham;Samuel Abraham;Peter Dryden.
Proceedings of the National Academy of Sciences of the United States of America (1992)
Ceramide Inhibits Protein Kinase B/Akt by Promoting Dephosphorylation of Serine 473 *
Kathryn M. Schubert;Michael P. Scheid;Vincent Duronio.
Journal of Biological Chemistry (2000)
Downstream Signalling Events Regulated by Phosphatidylinositol 3-Kinase Activity
Vincent Duronio;Michael P Scheid;Susan Ettinger.
Cellular Signalling (1998)
Phosphatidylinositol (3,4,5)P3 Is Essential but Not Sufficient for Protein Kinase B (PKB) Activation; Phosphatidylinositol (3,4)P2 Is Required for PKB Phosphorylation at Ser-473: STUDIES USING CELLS FROM SH2-CONTAINING INOSITOL-5-PHOSPHATASE KNOCKOUT MICE *
Michael P. Scheid;Michael Huber;Jacqueline E. Damen;Michael Hughes.
Journal of Biological Chemistry (2002)
Activation of Akt/protein kinase B in epithelial cells by the Salmonella typhimurium effector sigD.
Olivia Steele-Mortimer;Leigh A. Knodler;Sandra L. Marcus;Michael P. Scheid.
Journal of Biological Chemistry (2000)
Small-molecule agonists of SHIP1 inhibit the phosphoinositide 3-kinase pathway in hematopoietic cells.
Christopher J. Ong;Andrew Ming-Lum;Matt Nodwell;Ali Ghanipour.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: