Arachidonic acid, Cyclooxygenase and ATP synthase are the main areas of his Enzyme studies. He undertakes interdisciplinary study in the fields of Biochemistry and Immunology through his works. Thomas E. Eling conducts interdisciplinary study in the fields of Immunology and Biochemistry through his research. Thomas E. Eling combines Gene and Prostaglandin in his research. He integrates Prostaglandin and Gene in his research. In his research, Thomas E. Eling performs multidisciplinary study on Cancer research and Carcinogenesis. He integrates Carcinogenesis with Cancer research in his research. Many of his studies on Pharmacology apply to Sulindac as well. As part of his studies on Sulindac, Thomas E. Eling often connects relevant subjects like Nonsteroidal.
In his papers, Thomas E. Eling integrates diverse fields, such as Biochemistry and Immunology. He integrates Immunology with Biochemistry in his research. While working in this field, he studies both Enzyme and Peroxidase. Thomas E. Eling undertakes multidisciplinary studies into Gene and Cell biology in his work. Thomas E. Eling integrates many fields, such as Cell biology and Molecular biology, in his works. Thomas E. Eling performs integrative study on Molecular biology and Gene expression in his works. He connects Gene expression with Gene in his research. He merges many fields, such as Internal medicine and Endocrinology, in his writings. His work often combines Endocrinology and Internal medicine studies.
A significant part of his Cancer research incorporates Carcinogenesis, Colorectal cancer and Prostate cancer studies. His Cancer research extends to Carcinogenesis, which is thematically connected. Thomas E. Eling undertakes interdisciplinary study in the fields of Colorectal cancer and Cancer research through his research. He connects Cancer research with Prostate cancer in his study. His Internal medicine study frequently draws connections to other fields, such as Receptor. Many of his studies on Receptor apply to Internal medicine as well. His Biochemistry study often links to related topics such as Biomarker. His work on Leptin expands to the thematically related Endocrinology. Thomas E. Eling integrates Leptin and Adipose tissue in his research.
His study on Internal medicine is mostly dedicated to connecting different topics, such as Transforming growth factor. While working on this project, Thomas E. Eling studies both Transforming growth factor and Cancer. He performs integrative study on Cancer and Cancer research in his works. By researching both Cancer research and Prostate cancer, Thomas E. Eling produces research that crosses academic boundaries. While working in this field, Thomas E. Eling studies both Prostate cancer and Colorectal cancer. His multidisciplinary approach integrates Colorectal cancer and Pancreatic cancer in his work. His research ties Internal medicine and Pancreatic cancer together. His study ties his expertise on Apoptosis together with the subject of Biochemistry. In most of his Apoptosis studies, his work intersects topics such as Biochemistry.
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The horseradish peroxidase-catalyzed oxidation of 3,5,3',5'-tetramethylbenzidine. Free radical and charge-transfer complex intermediates.
P D Josephy;T Eling;R P Mason.
Journal of Biological Chemistry (1982)
High levels of GDF15 in thalassemia suppress expression of the iron regulatory protein hepcidin.
Toshihiko Tanno;Natarajan V Bhanu;Patricia A Oneal;Sung-Ho Goh.
Nature Medicine (2007)
Prostaglandin H synthase and xenobiotic oxidation.
T E Eling;D C Thompson;G L Foureman;J F Curtis.
Annual Review of Pharmacology and Toxicology (1990)
Role of reactive oxygen species in LPS-induced production of prostaglandin E2 in microglia.
Tongguang Wang;Liya Qin;Bin Liu;Yuxin Liu.
Journal of Neurochemistry (2004)
Resveratrol enhances the expression of non-steroidal anti-inflammatory drug-activated gene (NAG-1) by increasing the expression of p53
Seung Joon Baek;Leigh C. Wilson;Thomas E. Eling.
Carcinogenesis (2002)
Cyclooxygenase Inhibitors Induce the Expression of the Tumor Suppressor Gene EGR-1, Which Results in the Up-Regulation of NAG-1, an Antitumorigenic Protein
Seung Joon Baek;Jong-Sik Kim;Scott M Moore;Seong-Ho Lee.
Molecular Pharmacology (2005)
Epicatechin gallate-induced expression of NAG-1 is associated with growth inhibition and apoptosis in colon cancer cells.
Seung Joon Baek;Jong-Sik Kim;Felix R. Jackson;Thomas E. Eling.
Carcinogenesis (2004)
Modulation of the epidermal growth factor mitogenic response by metabolites of linoleic and arachidonic acid in Syrian hamster embryo fibroblasts. Differential effects in tumor suppressor gene (+) and (-) phenotypes.
W.C. Glasgow;C.A. Afshari;J.C. Barrett;T.E. Eling.
Journal of Biological Chemistry (1992)
Nonsteroidal Anti-Inflammatory Drug-Activated Gene-1 Over Expression in Transgenic Mice Suppresses Intestinal Neoplasia
Seung Joon Baek;Ryuji Okazaki;Seong-Ho Lee;Jeanelle Martinez.
Gastroenterology (2006)
Mitogenic signaling by epidermal growth factor (EGF), but not platelet-derived growth factor, requires arachidonic acid metabolism in BALB/c 3T3 cells : modulation of EGF-dependent c-myc expression by prostaglandins
J A Handler;R M Danilowicz;T E Eling.
Journal of Biological Chemistry (1990)
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