His primary scientific interests are in Biochemistry, Sphingolipid, Fumonisin, Ceramide synthase and Sphingosine. His Biochemistry research incorporates elements of Lycopersicon and Cell biology. His Sphingolipid research is multidisciplinary, incorporating elements of Neural tube defect, Biosynthesis, Enzyme, Fumonisin B1 and Callus.
His Fumonisin research is within the category of Fusarium. The various areas that Ronald T. Riley examines in his Ceramide synthase study include Cell culture, Signal transduction, Cell growth and Hepatocyte. His Sphingosine research incorporates themes from Biomarker and Chromatography.
Ronald T. Riley mainly focuses on Fumonisin, Biochemistry, Fumonisin B1, Ceramide synthase and Sphingolipid. Many of his studies involve connections with topics such as Microbiology and Fumonisin. Ronald T. Riley combines subjects such as Toxicity, Endocrinology, Kidney, Internal medicine and Pharmacology with his study of Fumonisin B1.
His study focuses on the intersection of Toxicity and fields such as Carcinogen with connections in the field of Carcinogenesis. His work carried out in the field of Ceramide synthase brings together such families of science as Dose–response relationship, Hepatocyte, Serine C-palmitoyltransferase, Molecular biology and Myriocin. His biological study spans a wide range of topics, including Neural tube, Ceramide, Biosynthesis and Metabolism.
His scientific interests lie mostly in Fumonisin, Fumonisin B1, Mycotoxin, Biochemistry and Food science. His work deals with themes such as Ochratoxin A and Ceramide synthase, which intersect with Fumonisin. The concepts of his Fumonisin B1 study are interwoven with issues in Ochratoxins, Aspergillus tubingensis, Aspergillus and Toxicity.
His study in Mycotoxin is interdisciplinary in nature, drawing from both Biomarker, Fusarium and Aflatoxin. In the subject of general Biochemistry, his work in Sphingolipid is often linked to Red Blood Cell Folate, thereby combining diverse domains of study. His Sphingolipid study which covers Receptor that intersects with Ceramide.
Ronald T. Riley mainly investigates Fumonisin, Fumonisin B1, Urinary system, Urine and Food science. His research integrates issues of Ceramide synthase, Excretion and Aflatoxin in his study of Fumonisin. Ronald T. Riley interconnects Neural tube, Embryo and Sphingolipid in the investigation of issues within Ceramide synthase.
The Aflatoxin study combines topics in areas such as Cohort and Co exposure. His studies deal with areas such as Wasting, Weaning and Environmental health as well as Fumonisin B1. His Mycotoxin research integrates issues from Aspergillus flavus, Microbiology, Secondary metabolism and Fusarium.
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IARC Monographs on the evaluation of carcinogenic risks to humans: Some traditional herbal medicines, some mycotoxins, naphthalene and styrene
Ahti Anttila;Ramesh V. Bhat;James A. Bond;Susan J. Borghoff.
IARC Monographs on the Evaluation of Carcinogenic Risks to Humans (2002)
Sphingolipids - The enigmatic lipid class: Biochemistry, physiology and pathophysiology
Merrill Ah;Schmelz Em;Dillehay Dl;Spiegel S.
Toxicology and Applied Pharmacology (1997)
Fumonisins Disrupt Sphingolipid Metabolism, Folate Transport, and Neural Tube Development in Embryo Culture and In Vivo: A Potential Risk Factor for Human Neural Tube Defects among Populations Consuming Fumonisin-Contaminated Maize
Walter F O Marasas;Ronald T. Riley;Katherine A. Hendricks;Victoria L. Stevens.
Journal of Nutrition (2004)
Sphingolipid metabolism: roles in signal transduction and disruption by fumonisins.
A H Merrill;M C Sullards;E Wang;K A Voss.
Environmental Health Perspectives (2001)
Alteration of Tissue and Serum Sphinganine to Sphingosine Ratio: An Early Biomarker of Exposure to Fumonisin-Containing Feeds in Pigs
Ronald T. Riley;Nyeon Hyoung An;Jency L. Showker;Hwan Soo Yoo.
Toxicology and Applied Pharmacology (1993)
Fumonisin- and AAL-Toxin-Induced Disruption of Sphingolipid Metabolism with Accumulation of Free Sphingoid Bases
H. K. Abbas;T. Tanaka;S. O. Duke;J. K. Porter.
Plant Physiology (1994)
Increases in serum sphingosine and sphinganine and decreases in complex sphingolipids in ponies given feed containing fumonisins, mycotoxins produced by Fusarium moniliforme.
Elaine Wang;P. Frank Ross;Terrance M. Wilson;Ronald T. Riley.
Journal of Nutrition (1992)
Sphingolipid perturbations as mechanisms for fumonisin carcinogenesis.
Ronald T. Riley;Evaristus Enongene;Kenneth A. Voss;William P. Norred.
Environmental Health Perspectives (2001)
Fumonisin inhibition of de novo sphingolipid biosynthesis and cytotoxicity are correlated in LLC-PK1 cells.
Hwan-Soo Yoo;William P. Norred;Elaine Wang;Alfred H. Merrill.
Toxicology and Applied Pharmacology (1992)
Cyclopiazonic acid inhibition of the Ca2+-transport ATPase in rat skeletal muscle sarcoplasmic reticulum vesicles.
Douglas E. Goeger;Ronald T. Riley;Joe W. Dorner;Richard J. Cole.
Biochemical Pharmacology (1988)
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