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D-Index & Metrics

Biology and Biochemistry

D-Index
55
Citations
11533
World Ranking
15004
National Ranking
462

Overview

Roberto Testi is affiliated with the University of Rome Tor Vergata in Italy. Their research spans multiple fields with a strong focus on medicine, biochemistry, genetics, and molecular biology, alongside neuroscience. The scientist's work covers a range of subfields including molecular biology, cellular and molecular neuroscience, neurology, oncology, and infectious diseases.

The main topics investigated by Roberto Testi include genetic neurodegenerative diseases, mitochondrial function and pathology, COVID-19 and healthcare impacts, Parkinson's disease mechanisms and treatments, autopsy techniques and outcomes, and body image and dysmorphia studies.

Recent papers authored or co-authored by Roberto Testi illustrate this diverse research interest. These include:

  • Management of the corpse with suspect, probable or confirmed COVID-19 respiratory infection (2020) in Pathologica
  • Preliminary Post-Mortem COVID-19 Evidence of Endothelial Injury and Factor VIII Hyperexpression (2020) in Diagnostics
  • The Impact of COVID-19 Pandemic and Lockdown on Alcohol Consumption: A Perspective From Hair Analysis (2021) in Frontiers in Psychiatry
  • Drug Repositioning in Friedreich Ataxia (2022) in Frontiers in Neuroscience
  • Safety and Efficacy Of Interferon γ in Friedreich's Ataxia (2020) in Movement Disorders

Roberto Testi frequently collaborates with several researchers, including Alessandra Rufini, Ivano Condò, Florence Malisan, Andrea Martinuzzi, and Andrea Quatrana. This network of co-authors indicates a collaborative research approach across related biomedical disciplines.

Publications by Roberto Testi commonly appear in venues such as Frontiers in Neuroscience, Human Molecular Genetics, Pathologica, Diagnostics, and Scientific Reports. The presence in these diverse journals highlights a multidisciplinary engagement with both clinical and molecular aspects of medical research.

Best Publications

  • Potential Involvement of Fas and Its Ligand in the Pathogenesis of Hashimoto's Thyroiditis

    Carla Giordano;Giorgio Stassi;Ruggero De Maria;Matilde Todaro

  • Apoptotic signaling through CD95 (Fas/Apo-1) activates an acidic sphingomyelinase.

    M G Cifone;R De Maria;P Roncaioli;M R Rippo

  • Identity of Leu-19 (CD56) leukocyte differentiation antigen and neural cell adhesion molecule.

    L L Lanier;R Testi;J Bindl;J H Phillips

  • The CD69 receptor: a multipurpose cell-surface trigger for hematopoietic cells

    Roberto Testi;Roberto Testi;Daniele D'Ambrosio;Daniele D'Ambrosio;Ruggero De Maria;Ruggero De Maria;Angela Santoni;Angela Santoni

  • Requirement for GD3 Ganglioside in CD95- and Ceramide-Induced Apoptosis

    Ruggero De Maria;Luisa Lenti;Luisa Lenti;Florence Malisan;Florence Malisan;Federica d'Agostino;Federica d'Agostino

  • Multiple pathways originate at the Fas/APO-1 (CD95) receptor: sequential involvement of phosphatidylcholine-specific phospholipase C and acidic sphingomyelinase in the propagation of the apoptotic signal.

    M. G. Cifone;P. Roncaioli;R. De Maria;G. Camarda

  • T cell activation via Leu-23 (CD69).

    R Testi;J H Phillips;L L Lanier

  • Nitric Oxide Primes Pancreatic β Cells for Fas-mediated Destruction in Insulin-dependent Diabetes Mellitus

    Giorgio Stassi;Ruggero De Maria;Giuliana Trucco;William Rudert

  • Leu 23 induction as an early marker of functional CD3/T cell antigen receptor triggering. Requirement for receptor cross-linking, prolonged elevation of intracellular [Ca++] and stimulation of protein kinase C.

    R Testi;J H Phillips;L L Lanier

  • Sphingomyelin breakdown and cell fate

    Roberto Testi

  • Fas/Apo-1 (CD95) receptor lacking the intracytoplasmic signaling domain protects tumor cells from Fas-mediated apoptosis.

    I Cascino;G Papoff;R De Maria;R Testi

  • Caspases Mediate Retinoic Acid–Induced Degradation of the Acute Promyelocytic Leukemia PML/RARα Fusion Protein

    Clara Nervi;Fabiana F. Ferrara;Mirco Fanelli;Maria Rita Rippo

  • Triggering of human monocyte activation through CD69, a member of the natural killer cell gene complex family of signal transducing receptors

    R De Maria;M G Cifone;R Trotta;M R Rippo

  • GD3 ganglioside directly targets mitochondria in a bcl-2-controlled fashion

    Maria Rita Rippo;Florence Malisan;Luigi Ravagnan;Barbara Tomassini

  • Src kinase phosphorylates Caspase‐8 on Tyr380: a novel mechanism of apoptosis suppression

    Silvia Cursi;Alessandra Rufini;Venturina Stagni;Ivano Condò

  • Involvement of p21ras activation in T cell CD69 expression.

    Daniele d'Ambrosio;Doreen A. Cantrell;Luigi Frati;Angela Santoni

  • Acidic sphingomyelinase (ASM) is necessary for fas-induced GD3 ganglioside accumulation and efficient apoptosis of lymphoid cells

    Ruggero De Maria;Maria Rita Rippo;Edward H. Schuchman;Roberto Testi

  • Fas-FasL interactions: a common pathogenetic mechanism in organ-specific autoimmunity.

    Ruggero De Maria;Roberto Testi

  • Stimulated human lamina propria T cells manifest enhanced Fas-mediated apoptosis.

    Monica Boirivant;Roberta Pica;Ruggero DeMaria;Roberto Testi

  • Differential regulation of apoptotic cell death in senescent human cells

    Barbara Hampel;Florence Malisan;Harald Niederegger;Roberto Testi

Frequent Co-Authors

Angela Santoni
Angela Santoni Sapienza University of Rome
Maria Grazia Cifone
Maria Grazia Cifone University of L'Aquila
Ruggero De Maria
Ruggero De Maria Catholic University of the Sacred Heart
Nicola Toschi
Nicola Toschi University of Rome Tor Vergata
Luigi Frati
Luigi Frati Sapienza University of Rome
Francesco Pallone
Francesco Pallone University of Rome Tor Vergata
Lewis L. Lanier
Lewis L. Lanier University of California, San Francisco
Matilde Todaro
Matilde Todaro University of Palermo
Joseph H. Phillips
Joseph H. Phillips MSD (United States)
Giorgio Stassi
Giorgio Stassi University of Palermo

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