Motoyuki Sugai focuses on Microbiology, Staphylococcus aureus, Molecular biology, Biochemistry and Autolysin. He is involved in the study of Microbiology that focuses on Antibacterial agent in particular. His research investigates the connection between Antibacterial agent and topics such as Antimicrobial peptides that intersect with problems in Defensin.
In general Staphylococcus aureus study, his work on Bullous impetigo, Staphylococcal scalded skin syndrome and Vancomycin often relates to the realm of Desmoglein 3, thereby connecting several areas of interest. His Molecular biology study combines topics in areas such as GTP-binding protein regulators, Janus kinase 2, Toxin and Open reading frame, Peptide sequence. His Autolysin research integrates issues from Neutrophil extracellular traps, Extracellular, N-acetylmuramoyl-L-alanine amidase, Gene product and Leukocidin.
Microbiology, Staphylococcus aureus, Molecular biology, Biochemistry and Gene are his primary areas of study. His Microbiology research is multidisciplinary, relying on both Methicillin-resistant Staphylococcus aureus, Mutant and Bacteria. The Staphylococcus aureus study which covers Immunology that intersects with Defensin.
As a part of the same scientific study, Motoyuki Sugai usually deals with the Molecular biology, concentrating on Cytolethal distending toxin and frequently concerns with Cell cycle, Apoptosis and Aggregatibacter actinomycetemcomitans. His study looks at the intersection of Biochemistry and topics like Lysostaphin with Endopeptidase. Within one scientific family, he focuses on topics pertaining to Antimicrobial under Antibacterial agent, and may sometimes address concerns connected to Pseudomonas aeruginosa.
Motoyuki Sugai spends much of his time researching Microbiology, Staphylococcus aureus, Gene, Atopic dermatitis and Molecular biology. His work often combines Microbiology and Mechanism studies. Motoyuki Sugai interconnects Biofilm, Immunology, Neutrophil extracellular traps and Staphylococcal Food Poisoning in the investigation of issues within Staphylococcus aureus.
In his study, DNA replication, Complementation and Toxin-antitoxin system is inextricably linked to Cell biology, which falls within the broad field of Gene. His study in Atopic dermatitis is interdisciplinary in nature, drawing from both Cell and Lysosome. Motoyuki Sugai has researched Molecular biology in several fields, including Toxin, Filaggrin, Transformation and Biomedical engineering.
His main research concerns Microbiology, Gene, Genetics, Staphylococcus aureus and Immunology. Motoyuki Sugai studies Microbiology, namely Imipenem. In his research, Virology is intimately related to DNA sequencing, which falls under the overarching field of Imipenem.
His Staphylococcus aureus research focuses on subjects like Immune system, which are linked to Cytokine. His studies in Immunology integrate themes in fields like Porphyromonas gingivalis, Fusobacterium nucleatum and Pregnancy, Early pregnancy factor, Gestation. His work deals with themes such as Nucleic acid sequence and Multiple drug resistance, which intersect with Enterobacteriaceae.
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A Novel Mechanism of Rapid Nuclear Neutrophil Extracellular Trap Formation in Response to Staphylococcus aureus
Florian H. Pilsczek;Davide Salina;Karen K. H. Poon;Candace Fahey.
Journal of Immunology (2010)
Antimicrobial Peptides in the Oral Environment: Expression and Function in Health and Disease
Kazuhisa Ouhara;Hitoshi Komatsuzawa;Sakuo Yamada;Hideki Shiba.
Journal of Antimicrobial Chemotherapy (2005)
Induction of Keratinocyte Migration via Transactivation of the Epidermal Growth Factor Receptor by the Antimicrobial Peptide LL-37
Sho Tokumaru;Koji Sayama;Yuji Shirakata;Hitoshi Komatsuzawa.
Journal of Immunology (2005)
A Staphylococcus aureus autolysin that has an N-acetylmuramoyl-L-alanine amidase domain and an endo-beta-N-acetylglucosaminidase domain: cloning, sequence analysis, and characterization.
Tadahiro Oshida;Motoyuki Sugai;Hitoshi Komatsuzawa;Yeong-Man Hong.
Proceedings of the National Academy of Sciences of the United States of America (1995)
Staphylococcus aureus Susceptibility to Innate Antimicrobial Peptides, β-Defensins and CAP18, Expressed by Human Keratinocytes
Kazushige Midorikawa;Kazuhisa Ouhara;Hitoshi Komatsuzawa;Toshihisa Kawai.
Infection and Immunity (2003)
An autolysin ring associated with cell separation of Staphylococcus aureus.
S Yamada;M Sugai;H Komatsuzawa;S Nakashima.
Journal of Bacteriology (1996)
Efficient Elimination of Multidrug-Resistant Staphylococcus aureus by Cloned Lysin Derived from Bacteriophage ϕMR11
Mohammad Rashel;Jumpei Uchiyama;Takako Ujihara;Yoshio Uehara.
The Journal of Infectious Diseases (2007)
The cell cycle-specific growth-inhibitory factor produced by Actinobacillus actinomycetemcomitans is a cytolethal distending toxin.
Motoyuki Sugai;Toru Kawamoto;Sylvie Y. Pérès;Yoko Ueno.
Infection and Immunity (1998)
Identification of the Staphylococcus aureus etd Pathogenicity Island Which Encodes a Novel Exfoliative Toxin, ETD, and EDIN-B
Takayuki Yamaguchi;Koji Nishifuji;Megumi Sasaki;Yasuyuki Fudaba.
Infection and Immunity (2002)
Enteropathogenic and enterohaemorrhagic Escherichia coli deliver a novel effector called Cif, which blocks cell cycle G2/M transition.
Olivier Marchès;Terence Neil Ledger;Michèle Boury;Masaru Ohara.
Molecular Microbiology (2003)
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