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Michael R. Bösl

Michael R. Bösl

Overview

Michael R. Bösl is affiliated with the University of Würzburg in Germany. Their research primarily focuses on the field of Biochemistry, Genetics and Molecular Biology, with notable contributions in subfields such as Molecular Biology and Cancer Research.

The main topics investigated by Michael R. Bösl include:

  • Glycosylation and Glycoproteins Research
  • Protein Structure and Dynamics
  • Ubiquitin and proteasome pathways
  • Epigenetics and DNA Methylation
  • Fibroblast Growth Factor Research
  • Cancer, Hypoxia, and Metabolism

The scientist has published in various venues, including:

  • Thrombosis Research
  • International Journal of Molecular Sciences

Recent papers authored by Michael R. Bösl are:

  • Mutations of the dimerization site of glycoprotein (GP) VI result in abolished expression, 2023, Thrombosis Research
  • Loss of SPRED3 Causes Primary Hypothyroidism and Alters Thyroidal Expression of Autophagy Regulators LC3, p62, and ATG5 in Mice, 2025, International Journal of Molecular Sciences

Michael R. Bösl has collaborated frequently with several co-authors, among them:

  • Stefano Navarro
  • Timo Vögtle
  • Nina Groß
  • Julia Preu
  • Maximilian Englert

Best Publications

  • Loss of the ClC-7 Chloride Channel Leads to Osteopetrosis in Mice and Man

    Uwe Kornak;Dagmar Kasper;Michael R Bösl;Edelgard Kaiser

  • Arc/Arg3.1 Is Essential for the Consolidation of Synaptic Plasticity and Memories

    Niels Plath;Ora Ohana;Ora Ohana;Björn Dammermann;Mick L. Errington

  • Disruption of ClC-3, a Chloride Channel Expressed on Synaptic Vesicles, Leads to a Loss of the Hippocampus

    Sandra M. Stobrawa;Tilman Breiderhoff;Shigeo Takamori;Dominique Engel

  • Kindlin-2 controls bidirectional signaling of integrins

    Eloi Montanez;Siegfried Ussar;Martina Schifferer;Michael Bösl

  • Impaired renal Na+ retention in the sgk1-knockout mouse

    Peer Wulff;Volker Vallon;Dan Yang Huang;Harald Völkl

  • Early embryonic lethality caused by targeted disruption of the mouse selenocysteine tRNA gene (Trsp)

    Michael R. Bösl;Kazuaki Takaku;Masanobu Oshima;Susumu Nishimura

  • Glycinergic neurons expressing enhanced green fluorescent protein in bacterial artificial chromosome transgenic mice

    Hanns Ulrich Zeilhofer;Barbara Studler;Dimitrula Arabadzisz;Claude Schweizer

  • Male germ cells and photoreceptors, both dependent on close cell-cell interactions, degenerate upon ClC-2 Cl(-) channel disruption.

    Michael R. Bösl;Valentin Stein;Christian Hübner;Anselm A. Zdebik

  • Lifeact mice for studying F-actin dynamics

    Julia Riedl;Kevin C Flynn;Aurelia Raducanu;Florian Gärtner

  • Aire-dependent production of XCL1 mediates medullary accumulation of thymic dendritic cells and contributes to regulatory T cell development

    Yu Lei;Yu Lei;Adiratna Mat Ripen;Naozumi Ishimaru;Izumi Ohigashi

  • SOX9 is a major negative regulator of cartilage vascularization, bone marrow formation and endochondral ossification

    Takako Hattori;Catharina Müller;Sonja Gebhard;Eva Bauer

  • Spontaneous relapsing-remitting EAE in the SJL/J mouse: MOG-reactive transgenic T cells recruit endogenous MOG-specific B cells.

    Bernadette Pöllinger;Gurumoorthy Krishnamoorthy;Kerstin Berer;Hans Lassmann

  • Orai1 (CRACM1) is the platelet SOC channel and essential for pathological thrombus formation

    Attila Braun;Bernhard Nieswandt;Dávid Varga-Szabó

  • Gene Targeting Reveals a Widespread Role for the High-Mobility-Group Transcription Factor Sox11 in Tissue Remodeling

    Elisabeth Sock;Stefanie D. Rettig;Janna Enderich;Michael R. Bösl

  • Sox10 is required for Schwann cell identity and progression beyond the immature Schwann cell stage

    Markus Finzsch;Silke Schreiner;Tatjana Kichko;Peter Reeh

  • Connective Tissue Growth Factor Causes Glaucoma by Modifying the Actin Cytoskeleton of the Trabecular Meshwork

    Benjamin Junglas;Sabrina Kuespert;Amin A. Seleem;Tobias Struller

  • Requirements for FGF3 and FGF10 during inner ear formation

    Yolanda Alvarez;Maria Teresa Alonso;Maria Teresa Alonso;Victor Vendrell;Laura Cecilia Zelarayan

  • A hypomorphic mouse model of dystrophic epidermolysis bullosa reveals mechanisms of disease and response to fibroblast therapy

    Anja Fritsch;Stefan Loeckermann;Johannes S. Kern;Attila Braun

  • Sox12 deletion in the mouse reveals nonreciprocal redundancy with the related Sox4 and Sox11 transcription factors

    Melanie Hoser;Michaela R. Potzner;Julia M. C. Koch;Michael R. Bösl

  • Impairment of L-type Ca2+ channel-dependent forms of hippocampal synaptic plasticity in mice deficient in the extracellular matrix glycoprotein tenascin-C.

    Matthias R. Evers;Benedikt Salmen;Olena Bukalo;Astrid Rollenhagen

Frequent Co-Authors

Ernst R. Tamm
Ernst R. Tamm University of Regensburg
Hans-Martin Jäck
Hans-Martin Jäck University of Erlangen-Nuremberg
Reinhard Fässler
Reinhard Fässler Max Planck Society
Klaus von der Mark
Klaus von der Mark University of Erlangen-Nuremberg
Hanns Ulrich Zeilhofer
Hanns Ulrich Zeilhofer University of Zurich
Jürgen Wienands
Jürgen Wienands University of Göttingen
Ingrid Hausser
Ingrid Hausser University Hospital Heidelberg
David P. Wolfer
David P. Wolfer University of Zurich
Josef Pfeilschifter
Josef Pfeilschifter Goethe University Frankfurt
Leena Bruckner-Tuderman
Leena Bruckner-Tuderman University of Freiburg

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