What is he best known for?
The fields of study he is best known for:
His primary areas of study are Genetics, Cell biology, Retinitis pigmentosa, Biochemistry and Chaperone.
His Cell biology research is multidisciplinary, relying on both Membrane protein, Regulation of gene expression and Photoreceptor Connecting Cilium.
His Retinitis pigmentosa research incorporates elements of Retinal degeneration, Positional cloning, Neurodegeneration, Rhodopsin and Proteostasis.
His Chaperone study combines topics from a wide range of disciplines, such as DNAJ Protein and Transport protein.
Michael E. Cheetham usually deals with Protein folding and limits it to topics linked to Computational biology and Atpase activity, Escherichia coli Proteins, Chaperone-mediated autophagy and Autolysosome.
His Programmed cell death research integrates issues from Autophagy, MAP1LC3B, Sequestosome 1, Autophagosome and Physiology.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the nomenclature of the human heat shock proteins (814 citations)
- Structure, function and evolution of DnaJ: conservation and adaptation of chaperone function (489 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Cell biology, Genetics, Retinitis pigmentosa, Rhodopsin and Molecular biology.
His Cell biology study combines topics in areas such as Retinal, Biochemistry and Ubiquitin.
Michael E. Cheetham combines subjects such as Retinal degeneration, Visual phototransduction and Positional cloning with his study of Retinitis pigmentosa.
He works mostly in the field of Rhodopsin, limiting it down to concerns involving Proteostasis and, occasionally, Neurodegeneration.
His Molecular biology research incorporates themes from Complementary DNA and Microtubule-associated protein.
His biological study spans a wide range of topics, including Heat shock protein, NEDD8 and Proteasome.
He most often published in these fields:
- Cell biology (40.17%)
- Genetics (23.85%)
- Retinitis pigmentosa (21.34%)
What were the highlights of his more recent work (between 2017-2021)?
- Cell biology (40.17%)
- Retinal (13.39%)
- Gene (9.62%)
In recent papers he was focusing on the following fields of study:
Michael E. Cheetham focuses on Cell biology, Retinal, Gene, Genetics and Retinitis pigmentosa.
Michael E. Cheetham interconnects Photoreceptor cell, Rhodopsin, Biogenesis and Visual phototransduction in the investigation of issues within Cell biology.
His study focuses on the intersection of Retinal and fields such as Organoid with connections in the field of Molecular biology and Alternative splicing.
His study brings together the fields of Disease and Genetics.
In his work, Endoplasmic reticulum, Erg, ER retention and Programmed cell death is strongly intertwined with Outer nuclear layer, which is a subfield of Retinitis pigmentosa.
In his study, Cytosol is inextricably linked to Autophagy, which falls within the broad field of Programmed cell death.
Between 2017 and 2021, his most popular works were:
- Effect of an intravitreal antisense oligonucleotide on vision in Leber congenital amaurosis due to a photoreceptor cilium defect (89 citations)
- Splice-Modulating Oligonucleotide QR-110 Restores CEP290 mRNA and Function in Human c.2991+1655A>G LCA10 Models (56 citations)
- DNAJ Proteins in neurodegeneration: essential and protective factors. (42 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in Retinal, Gene, Organoid, Molecular biology and Missense mutation.
His Retinal study incorporates themes from Messenger RNA, Oligonucleotide and Cell biology.
Michael E. Cheetham has included themes like DNAJ Protein, Photoreceptor outer segment and Neurodegeneration in his Cell biology study.
The Organoid study combines topics in areas such as Mutation, Compound heterozygosity, RNA splicing and splice.
Missense mutation is a subfield of Genetics that Michael E. Cheetham studies.
His research in Programmed cell death intersects with topics in Autophagy, Chaperone-mediated autophagy, Autolysosome and Knowledge base.
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