Staphylococcus aureus, Microbiology, Bacterial adhesin, Fibronectin and Immunology are his primary areas of study. Mathias Herrmann has researched Staphylococcus aureus in several fields, including Extracellular, Internalization and Antibiotics. His study in Microbiology is interdisciplinary in nature, drawing from both Bacteria, Biofilm, Integrin, Gene and Platelet activation.
His Bacterial adhesin research incorporates themes from Protein A and Fibronectin binding. His study explores the link between Fibronectin and topics such as Coagulase that cross with problems in Laminin. His research integrates issues of Antimicrobial, Intracellular parasite and Virulence in his study of Staphylococcal infections.
His scientific interests lie mostly in Microbiology, Staphylococcus aureus, Immunology, Virulence and Staphylococcal infections. His Microbiology research is multidisciplinary, incorporating elements of Biofilm, Methicillin-resistant Staphylococcus aureus and Virology. His Staphylococcus aureus research incorporates elements of Fibronectin, Bacterial adhesin, Antibiotics and Gene.
His Fibronectin study combines topics in areas such as Extracellular and Fibrinogen. The concepts of his Immunology study are interwoven with issues in Receptor, Cell adhesion and Staphylococcus. His Virulence study integrates concerns from other disciplines, such as Phenotype, Genotyping and Mutant.
His primary scientific interests are in Microbiology, Staphylococcus aureus, Virulence, Staphylococcal infections and Methicillin-resistant Staphylococcus aureus. His work deals with themes such as Ribotyping, Genotype and Virology, which intersect with Microbiology. His research on Staphylococcus aureus concerns the broader Bacteria.
Mathias Herrmann interconnects Operon, Mutant, Multilocus sequence typing and Whole genome sequencing in the investigation of issues within Virulence. His studies deal with areas such as Post-hoc analysis, Hospital hygiene, Incision and drainage and Immunology as well as Staphylococcal infections. His research investigates the connection between Methicillin-resistant Staphylococcus aureus and topics such as Emergency medicine that intersect with issues in Screening study, Young adult and Skin infection.
Mathias Herrmann mainly focuses on Microbiology, Staphylococcus aureus, Virulence, Methicillin-resistant Staphylococcus aureus and Intensive care medicine. His Microbiology research includes themes of Clade, Immunology and Virology. The Staphylococcus aureus study combines topics in areas such as Antibiotics, Biochemistry, Peptidoglycan, Transmission and Host.
His Virulence research integrates issues from Lineage, Genotyping, Multilocus sequence typing, Typing and Sequence analysis. His Methicillin-resistant Staphylococcus aureus study incorporates themes from Young adult, Screening study, Intensive care and Risk factor. He works mostly in the field of Intensive care medicine, limiting it down to topics relating to Extracorporeal membrane oxygenation and, in certain cases, Cardiac surgery, Prospective cohort study and Mycobacterium.
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Small colony variants: a pathogenic form of bacteria that facilitates persistent and recurrent infections
Richard A. Proctor;Christof von Eiff;Barbara C. Kahl;Karsten Becker.
Nature Reviews Microbiology (2006)
Fibronectin, Fibrinogen, and Laminin Act as Mediators of Adherence of Clinical Staphylococcal Isolates to Foreign Material
Mathias Herrmann;Pierre E. Vaudaux;Didier Pittet;Raymond Auckenthaler.
The Journal of Infectious Diseases (1988)
Fibronectin-binding protein acts as Staphylococcus aureus invasin via fibronectin bridging to integrin alpha5beta1.
Bhanu Sinha;Patrice P. Francois;Oliver Nusse;Michelangelo Foti.
Cellular Microbiology (1999)
Induction of Staphylococcus epidermidis biofilm formation via proteolytic processing of the accumulation-associated protein by staphylococcal and host proteases
Holger Rohde;Christoph Burdelski;Katrin Bartscht;Muzaffar Hussain.
Molecular Microbiology (2005)
Persistent Infection with Small Colony Variant Strains of Staphylococcus aureus in Patients with Cystic Fibrosis
Barbara Kahl;Mathias Herrmann;Anne Schulze Everding;Hans G. Koch.
The Journal of Infectious Diseases (1998)
A 140-kilodalton extracellular protein is essential for the accumulation of Staphylococcus epidermidis strains on surfaces.
M Hussain;M Herrmann;C von Eiff;F Perdreau-Remington.
Infection and Immunity (1997)
Protein A is the von Willebrand factor binding protein onStaphylococcus aureus
Jörg Hartleib;Nicola Köhler;Richard B. Dickinson;Gursharan S. Chhatwal.
Staphylococcus aureus extracellular adherence protein serves as anti-inflammatory factor by inhibiting the recruitment of host leukocytes
Triantafyllos Chavakis;Muzaffar Hussain;Sandip M Kanse;Georg Peters.
Nature Medicine (2002)
Fibrinogen and fibronectin binding cooperate for valve infection and invasion in Staphylococcus aureus experimental endocarditis.
Yok-Ai Que;Jacques-Antoine Haefliger;Lionel Piroth;Patrice François.
Journal of Experimental Medicine (2005)
Heterologously expressed Staphylococcus aureus fibronectin-binding proteins are sufficient for invasion of host cells.
Bhanu Sinha;Patrice Francois;Yok-Ai Que;Muzaffar Hussain.
Infection and Immunity (2000)
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