His main research concerns Internal medicine, Endocrinology, Cell biology, Pathology and Immunology. While the research belongs to areas of Internal medicine, Masataka Sata spends his time largely on the problem of Cardiology, intersecting his research to questions surrounding Diastole. As a member of one scientific family, he mostly works in the field of Endocrinology, focusing on Signal transduction and, on occasion, Protein kinase A and Downregulation and upregulation.
He has included themes like Endothelial stem cell, Fas ligand, Cellular differentiation, Apoptosis and Neointima in his Cell biology study. His Endothelial stem cell study integrates concerns from other disciplines, such as Extracellular matrix and Homing. His work carried out in the field of Pathology brings together such families of science as Progenitor cell and Myocyte.
Internal medicine, Cardiology, Endocrinology, Pathology and Inflammation are his primary areas of study. His work on Internal medicine is being expanded to include thematically relevant topics such as Diabetes mellitus. His Cardiology research includes elements of Surgery and Diastole.
The concepts of his Endocrinology study are interwoven with issues in Receptor, Apolipoprotein E and Angiogenesis. His Pathology research integrates issues from Neointimal hyperplasia and Neointima. His work in Bone marrow tackles topics such as Progenitor cell which are related to areas like Endothelial stem cell and Immunology.
His primary areas of investigation include Internal medicine, Cardiology, Inflammation, Heart failure and In patient. Masataka Sata interconnects Diabetes mellitus, Endocrinology and Type 2 diabetes in the investigation of issues within Internal medicine. His Cardiology study deals with Clinical endpoint intersecting with Hazard ratio.
The Inflammation study combines topics in areas such as Adipose tissue, Receptor and Pathogenesis, Pathology. The concepts of his Heart failure study are interwoven with issues in Randomized controlled trial and Stress Echocardiography. As part of one scientific family, he deals mainly with the area of Endothelial dysfunction, narrowing it down to issues related to the Apolipoprotein E, and often Deficient mouse.
The scientist’s investigation covers issues in Internal medicine, Cardiology, Inflammation, Endothelial dysfunction and Vasodilation. The study incorporates disciplines such as Endocrinology, Type 2 diabetes and Type 2 Diabetes Mellitus in addition to Internal medicine. His work on Canagliflozin as part of his general Endocrinology study is frequently connected to Nutrient, thereby bridging the divide between different branches of science.
His work is dedicated to discovering how Type 2 Diabetes Mellitus, Adipose tissue are connected with Adipokine, Pathogenesis, Pathology and Vasa vasorum and other disciplines. His work on Ejection fraction and Heart failure as part of general Cardiology study is frequently linked to In patient, bridging the gap between disciplines. His Inflammation research integrates issues from Apolipoprotein B, Receptor, Thermogenesis, Macrophage and Pharmacology.
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Hematopoietic stem cells differentiate into vascular cells that participate in the pathogenesis of atherosclerosis
Masataka Sata;Akio Saiura;Atsushi Kunisato;Akihiro Tojo.
Nature Medicine (2002)
Role of Adiponectin in Preventing Vascular Stenosis THE MISSING LINK OF ADIPO-VASCULAR AXIS
Morihiro Matsuda;Iichiro Shimomura;Masataka Sata;Yukio Arita.
Journal of Biological Chemistry (2002)
Periostin is essential for cardiac healing after acute myocardial infarction
Masashi Shimazaki;Kazuto Nakamura;Isao Kii;Takeshi Kashima.
Journal of Experimental Medicine (2008)
Notch1 but Not Notch2 Is Essential for Generating Hematopoietic Stem Cells from Endothelial Cells
Keiki Kumano;Shigeru Chiba;Atsushi Kunisato;Masataka Sata.
Immunity (2003)
Circulating smooth muscle progenitor cells contribute to atherosclerosis
Akio Saiura;Masataka Sata;Yasunobu Hirata;Ryozo Nagai.
Nature Medicine (2001)
Krüppel-like zinc-finger transcription factor KLF5/BTEB2 is a target for angiotensin II signaling and an essential regulator of cardiovascular remodeling.
Takayuki Shindo;Ichiro Manabe;Yasushi Fukushima;Kazuyuki Tobe.
Nature Medicine (2002)
Diverse contribution of bone marrow cells to neointimal hyperplasia after mechanical vascular injuries.
Kimie Tanaka;Masataka Sata;Yasunobu Hirata;Ryozo Nagai.
Circulation Research (2003)
A mouse model of vascular injury that induces rapid onset of medial cell apoptosis followed by reproducible neointimal hyperplasia.
Masataka Sata;Yoshie Maejima;Fumiko Adachi;Keiko Fukino.
Journal of Molecular and Cellular Cardiology (2000)
Coronary Atherosclerosis Is Associated With Macrophage Polarization in Epicardial Adipose Tissue
Yoichiro Hirata;Minoru Tabata;Hirotsugu Kurobe;Tatsuo Motoki.
Journal of the American College of Cardiology (2011)
Oxidized LDL Activates Fas-mediated Endothelial Cell Apoptosis
Masataka Sata;Kenneth Walsh.
Journal of Clinical Investigation (1998)
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