D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 41 Citations 7,873 92 World Ranking 17509 National Ranking 7172

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Internal medicine

Mary C. Nakamura mainly investigates Cell biology, Receptor, Immune system, Immunology and Syk. Mary C. Nakamura works mostly in the field of Cell biology, limiting it down to concerns involving ZAP70 and, occasionally, B-cell receptor. His research integrates issues of Myeloid, Cell culture, Transferrin and In vitro in his study of Receptor.

His Immune system research is multidisciplinary, incorporating perspectives in Inflammatory bowel disease and Arthritis. Many of his studies involve connections with topics such as Signal transduction and Immunology. The Cancer research study combines topics in areas such as Osteoclast and Bone resorption.

His most cited work include:

  • The immunomodulatory adapter proteins DAP12 and Fc receptor γ-chain (FcRγ) regulate development of functional osteoclasts through the Syk tyrosine kinase (400 citations)
  • Cutting Edge: Inhibition of TLR and FcR Responses in Macrophages by Triggering Receptor Expressed on Myeloid Cells (TREM)-2 and DAP12 (309 citations)
  • A role for TREM2 ligands in the phagocytosis of apoptotic neuronal cells by microglia. (287 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Cell biology, Immunology, Receptor, Osteoclast and Inflammation. His Cell biology research is multidisciplinary, incorporating elements of Immune receptor, Lymphokine-activated killer cell, Natural killer cell, TREM2 and Major histocompatibility complex. His Receptor study integrates concerns from other disciplines, such as Molecular biology, Lysis and MHC class I.

Mary C. Nakamura interconnects Endocrinology, Bone remodeling, Bone resorption and RANKL in the investigation of issues within Osteoclast. Mary C. Nakamura has researched RANKL in several fields, including Cancer research and Signal transduction. His Inflammation research also works with subjects such as

  • Macrophage which is related to area like Gene expression, Pathology and Periodontitis,
  • Bone healing and related Age related and Wnt signaling pathway.

He most often published in these fields:

  • Cell biology (36.36%)
  • Immunology (42.73%)
  • Receptor (35.45%)

What were the highlights of his more recent work (between 2016-2021)?

  • Cancer research (19.09%)
  • Immunology (42.73%)
  • Inflammation (21.82%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Cancer research, Immunology, Inflammation, Lung and Bone healing. His research on Cancer research also deals with topics like

  • Lung transplantation which intersects with area such as Adoptive cell transfer, Reperfusion injury, Receptor and Ligand,
  • Progenitor cell and Angiogenesis most often made with reference to Mesenchymal stem cell. In general Inflammation, his work in Proinflammatory cytokine and Interleukin 17 is often linked to IκB kinase and Zinc finger linking many areas of study.

His Lung research is multidisciplinary, incorporating perspectives in NKG2D, Innate immune system and Cell. In his study, which falls under the umbrella issue of Bioinformatics, Immune system is strongly linked to Inflammatory response. As part of his studies on Cell biology, Mary C. Nakamura often connects relevant subjects like TREM2.

Between 2016 and 2021, his most popular works were:

  • Effects of Aging on Fracture Healing. (43 citations)
  • Non-catalytic ubiquitin binding by A20 prevents psoriatic arthritis–like disease and inflammation (20 citations)
  • NF-κB/MAPK activation underlies ACVR1-mediated inflammation in human heterotopic ossification (16 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Internal medicine

Mary C. Nakamura mainly focuses on Osteoimmunology, Bone healing, Extramural, Neuroscience and Energy metabolism. His work carried out in the field of Bone healing brings together such families of science as Wnt signaling pathway, Nuclear localization sequence, Angiogenesis and Beta. He merges Extramural with Integrated systems in his research.

His Cartilage research incorporates Infiltration, Transcriptome, Pathology, Macrophage and Age related. His work on Inflammation expands to the thematically related Infiltration. Mary C. Nakamura has included themes like Tumor necrosis factor alpha and Psoriatic arthritis, Arthritis in his Inflammation study.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The immunomodulatory adapter proteins DAP12 and Fc receptor γ-chain (FcRγ) regulate development of functional osteoclasts through the Syk tyrosine kinase

Attila Mócsai;Mary Beth Humphrey;Jessica A. G. Van Ziffle;Yongmei Hu.
Proceedings of the National Academy of Sciences of the United States of America (2004)

515 Citations

A role for TREM2 ligands in the phagocytosis of apoptotic neuronal cells by microglia.

Christine L. Hsieh;Maya Koike;Steve C. Spusta;Erene C. Niemi.
Journal of Neurochemistry (2009)

490 Citations

Cutting Edge: Inhibition of TLR and FcR Responses in Macrophages by Triggering Receptor Expressed on Myeloid Cells (TREM)-2 and DAP12

Jessica A. Hamerman;Jessica R. Jarjoura;Mary Beth Humphrey;Mary C. Nakamura.
Journal of Immunology (2006)

433 Citations

Binding and uptake of H-ferritin are mediated by human transferrin receptor-1

Li Li;Celia J. Fang;James C. Ryan;Eréne C. Niemi.
Proceedings of the National Academy of Sciences of the United States of America (2010)

401 Citations

Glucocorticoid excess in mice results in early activation of osteoclastogenesis and adipogenesis and prolonged suppression of osteogenesis: a longitudinal study of gene expression in bone tissue from glucocorticoid-treated mice.

Wei Yao;Zhiqiang Cheng;Cheryl Busse;Aaron Pham.
Arthritis & Rheumatism (2008)

313 Citations

Triggering Receptor Expressed on Myeloid Cells 2 (TREM2) Deficiency Attenuates Phagocytic Activities of Microglia and Exacerbates Ischemic Damage in Experimental Stroke

Masahito Kawabori;Rachid Kacimi;Tiina Kauppinen;Cyrus Calosing.
The Journal of Neuroscience (2015)

311 Citations

Mouse Ly-49A Interrupts Early Signaling Events in Natural Killer Cell Cytotoxicity and Functionally Associates with the SHP-1 Tyrosine Phosphatase

Mary C. Nakamura;Eréne C. Niemi;Mark J. Fisher;Leonard D. Shultz.
Journal of Experimental Medicine (1997)

311 Citations

Role of ITAM-containing adapter proteins and their receptors in the immune system and bone.

Mary Beth Humphrey;Lewis L. Lanier;Mary C. Nakamura;Mary C. Nakamura.
Immunological Reviews (2005)

276 Citations

TIM-2 is expressed on B cells and in liver and kidney and is a receptor for H-ferritin endocytosis

Thomas T. Chen;Li Li;Dong-Hui Chung;Christopher D.C. Allen.
Journal of Experimental Medicine (2005)

271 Citations

DAP12-mediated signal transduction in natural killer cells. A dominant role for the Syk protein-tyrosine kinase.

Daniel W. McVicar;Lynn S. Taylor;Pierre Gosselin;Jami Willette-Brown.
Journal of Biological Chemistry (1998)

250 Citations

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