His primary areas of study are Cell biology, Actin, Cytoskeleton, Actin-binding protein and Arp2/3 complex. Much of his study explores Cell biology relationship to Platelet. His Actin study combines topics from a wide range of disciplines, such as Binding protein, Biophysics, Myosin and Filamin.
John H. Hartwig has researched Cytoskeleton in several fields, including Ultrastructure and Band 3, Membrane protein. His work carried out in the field of Actin-binding protein brings together such families of science as Molecular biology, Sequence, Function and Microfilament. The concepts of his Arp2/3 complex study are interwoven with issues in MDia1, Wiskott–Aldrich syndrome protein and Actin remodeling.
John H. Hartwig mostly deals with Cell biology, Actin, Platelet, Cytoskeleton and Arp2/3 complex. His research integrates issues of Actin remodeling, Actin-binding protein, Actin cytoskeleton, Actin remodeling of neurons and Filamin in his study of Cell biology. His research in Actin intersects with topics in Biophysics, Myosin, Protein filament and Cytoplasm.
John H. Hartwig works mostly in the field of Platelet, limiting it down to concerns involving Receptor and, occasionally, Molecular biology. His Cytoskeleton research is multidisciplinary, relying on both Platelet activation and Cell membrane. His Arp2/3 complex research integrates issues from Filopodia, Actin nucleation and Microfilament.
The scientist’s investigation covers issues in Cell biology, Actin, Cytoskeleton, Platelet and Filamin. His study of Microtubule is a part of Cell biology. His studies deal with areas such as Cell, Cell adhesion, Wiskott–Aldrich syndrome, COS cells and Myosin as well as Actin.
In general Cytoskeleton study, his work on Spectrin often relates to the realm of Airway smooth muscle, Rheology and Stress, thereby connecting several areas of interest. His work in the fields of Von Willebrand factor overlaps with other areas such as Megakaryocyte. His work on FLNA as part of general Filamin study is frequently linked to Megakaryocytopoiesis, bridging the gap between disciplines.
John H. Hartwig mostly deals with Cell biology, FLNA, Filamin, Actin cytoskeleton and Cytoskeleton. The various areas that John H. Hartwig examines in his Cell biology study include Actin remodeling of neurons, Actin remodeling and Arp2/3 complex. John H. Hartwig interconnects MDia1 and Immunological synapse in the investigation of issues within Arp2/3 complex.
His work deals with themes such as Ena/Vasp homology proteins and Profilin, which intersect with MDia1. His biological study deals with issues like Fluorescence recovery after photobleaching, which deal with fields such as Differential interference contrast microscopy, Cytoplasmic Dyneins and Tubulin. His Actin research incorporates themes from Mutation, Cell signaling, Transcription factor and Cell adhesion.
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Extracellular DNA traps promote thrombosis
Tobias A. Fuchs;Alexander Brill;Daniel Duerschmied;Daphne Schatzberg.
Proceedings of the National Academy of Sciences of the United States of America (2010)
Filamins as integrators of cell mechanics and signalling.
Thomas P. Stossel;John Condeelis;Lynn Cooley;John H. Hartwig.
Nature Reviews Molecular Cell Biology (2001)
MARCKS is an actin filament crosslinking protein regulated by protein kinase C and calcium-calmodulin.
J. H. Hartwig;M. Thelen;M. Thelen;A. Resen;P. A. Janmey.
Thrombin receptor ligation and activated rac uncap actin filament barbed ends through phosphoinositide synthesis in permeabilized human platelets
John H. Hartwig;Gary M. Bokoch;Christopher L. Carpenter;Paul A. Janmey.
Actin-binding protein requirement for cortical stability and efficient locomotion.
CC Cunningham;JB Gorlin;DJ Kwiatkowski;JH Hartwig.
Nonmuscle Actin-Binding Proteins
T. P. Stossel;C. Chaponnier;R. M. Ezzell;J. H. Hartwig.
Annual Review of Cell Biology (1985)
Blood platelets are assembled principally at the ends of proplatelet processes produced by differentiated megakaryocytes.
Joseph E. Italiano;Patrick Lecine;Ramesh A. Shivdasani;John H. Hartwig;John H. Hartwig.
Journal of Cell Biology (1999)
Paneth cells as a site of origin for intestinal inflammation
Timon E. Adolph;Michal F. Tomczak;Lukas Niederreiter;Hyun Jeong Ko;Hyun Jeong Ko.
Hemostatic, inflammatory, and fibroblast responses are blunted in mice lacking gelsolin
Walter Witke;Arlene H Sharpe;John H Hartwig;Toshifumi Azuma.
The small GTPase RalA targets filamin to induce filopodia
Yasutaka Ohta;Nobuchika Suzuki;Shun Nakamura;John H. Hartwig.
Proceedings of the National Academy of Sciences of the United States of America (1999)
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