What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His primary areas of study are Immunology, Interleukin 33, High endothelial venules, Cytokine and Cell biology.
His is doing research in Immune system and Interleukin 3, both of which are found in Immunology.
His Interleukin 33 research is multidisciplinary, incorporating perspectives in Inflammation, Natural killer T cell, Innate immune system, Innate lymphoid cell and In vivo.
His High endothelial venules research focuses on subjects like Lymphocyte homing receptor, which are linked to Lymph, Lymph node stromal cell, Lymphatic Endothelium, In situ hybridization and Suppression subtractive hybridization.
His research in Cytokine is mostly focused on Interleukin-1 Receptor-Like 1 Protein.
His Cell biology research is multidisciplinary, incorporating elements of Structural motif and Peptide sequence.
His most cited work include:
- Transient increase in obese gene expression after food intake or insulin administration. (986 citations)
- The IL-1-Like Cytokine IL-33 Is Constitutively Expressed in the Nucleus of Endothelial Cells and Epithelial Cells In Vivo : A Novel ‘Alarmin’? (824 citations)
- The IL-1-Like Cytokine IL-33 Is Constitutively Expressed in the Nucleus of Endothelial Cells and Epithelial Cells In Vivo : A Novel ‘Alarmin’? (824 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Internal medicine, Endocrinology, Insulin, Immunology and Interleukin 33.
His Insulin research integrates issues from Diabetes mellitus, Hormone, Stimulation and Gene expression.
His is involved in several facets of Immunology study, as is seen by his studies on Inflammation, Immune system, High endothelial venules, T cell and Natural killer T cell.
Jean-Philippe Girard interconnects Innate immune system, Innate lymphoid cell and Cell biology in the investigation of issues within Interleukin 33.
His research in Cell biology intersects with topics in Endothelial stem cell and Molecular biology.
His Endothelium study which covers Pathology that intersects with Lymphocyte.
He most often published in these fields:
- Internal medicine (44.67%)
- Endocrinology (44.67%)
- Insulin (22.13%)
What were the highlights of his more recent work (between 2010-2021)?
- Interleukin 33 (22.95%)
- Immunology (28.28%)
- Cytokine (15.57%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Interleukin 33, Immunology, Cytokine, Immune system and High endothelial venules.
His Interleukin 33 study combines topics from a wide range of disciplines, such as Inflammation, Innate immune system, Innate lymphoid cell and Arthritis.
His studies in Innate lymphoid cell integrate themes in fields like Interleukin 3, Androgen, Allergic inflammation and Cell biology.
His research investigates the connection between Cytokine and topics such as Liver injury that intersect with problems in Knockout mouse and CXCR3.
Jean-Philippe Girard focuses mostly in the field of Immune system, narrowing it down to topics relating to Antigen and, in certain cases, Endothelium and Type 2 immune response.
His research on High endothelial venules frequently links to adjacent areas such as Lymphocyte homing receptor.
Between 2010 and 2021, his most popular works were:
- Interleukin-33 in health and disease (412 citations)
- IL-33: an alarmin cytokine with crucial roles in innate immunity, inflammation and allergy. (375 citations)
- IL-33: an alarmin cytokine with crucial roles in innate immunity, inflammation and allergy. (375 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
His primary areas of investigation include Immunology, Interleukin 33, High endothelial venules, Innate lymphoid cell and Allergic inflammation.
His Immune system and Interleukin investigations are all subjects of Immunology research.
Cytokine covers he research in Interleukin 33.
Jean-Philippe Girard usually deals with Cytokine and limits it to topics linked to Cell biology and Endogeny, Receptor and Elastase.
His work deals with themes such as Mast cell, Chymase, Tryptase and Mode of action, which intersect with Allergic inflammation.
His Inflammation research incorporates themes from Endocrinology, Homeostasis, Signal transduction and Androgen.
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