2018 - Fellow of the American Society of Mechanical Engineers
2005 - Fellow of the American Association for the Advancement of Science (AAAS)
1966 - Fellow of the American Association for the Advancement of Science (AAAS)
James G. Wilson mainly focuses on Genetics, Internal medicine, Genome-wide association study, Single-nucleotide polymorphism and Allele. His study in Genetic association, Exome, Genotype, Locus and Allele frequency is done as part of Genetics. His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology, Endocrinology, Immunology and Type 2 diabetes.
His research investigates the connection between Single-nucleotide polymorphism and topics such as Candidate gene that intersect with problems in SNP genotyping. His studies in Allele integrate themes in fields like Genetic variation, Disease and Genetic admixture. His study looks at the relationship between Genetic variation and topics such as Genomics, which overlap with DNA Mutational Analysis and Mutation.
James G. Wilson mostly deals with Genetics, Genome-wide association study, Internal medicine, Single-nucleotide polymorphism and Genetic association. His work in Allele, Locus, Exome, Gene and Allele frequency is related to Genetics. His Gene study focuses on Genetic variation in particular.
His study explores the link between Genome-wide association study and topics such as Imputation that cross with problems in Genome. His biological study spans a wide range of topics, including Endocrinology, Type 2 diabetes and Cardiology. His studies deal with areas such as Meta-analysis and Polymorphism as well as Single-nucleotide polymorphism.
His main research concerns Internal medicine, Genome-wide association study, Genetics, Disease and Genetic association. The study incorporates disciplines such as Endocrinology, Type 2 diabetes and Cardiology in addition to Internal medicine. His Genome-wide association study research incorporates themes from Minor allele frequency, Locus, Allele and Bioinformatics.
His Locus study combines topics from a wide range of disciplines, such as Meta-analysis and Genetic variation. His studies in Gene, Gene expression, Whole genome sequencing, Exome and Genome are all subfields of Genetics research. His research integrates issues of Penetrance, Blood lipids and Kidney disease in his study of Disease.
The scientist’s investigation covers issues in Internal medicine, Genetics, Genome-wide association study, Genetic association and Genetic variation. His Internal medicine research incorporates elements of Endocrinology and Cardiology. His work on Genetics is being expanded to include thematically relevant topics such as Gout.
To a larger extent, James G. Wilson studies Single-nucleotide polymorphism with the aim of understanding Genome-wide association study. The various areas that James G. Wilson examines in his Genetic association study include Regulation of gene expression, Albuminuria, QRS complex and Medical genetics. His study looks at the relationship between Genetic variation and fields such as Locus, as well as how they intersect with chemical problems.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33)
R C Turner;R R Holman;C A Cull;I M Stratton.
(1998)
Tight blood pressure control and risk of macrovascular and microvascular complications in type 2 diabetes: UKPDS 38. UK Prospective Diabetes Study Group.
Stearne;SL Palmer;Hammersley;SL Franklin.
(1998)
Analysis of protein-coding genetic variation in 60,706 humans
Monkol Lek;Konrad J. Karczewski;Konrad J. Karczewski;Eric V. Minikel;Eric V. Minikel;Kaitlin E. Samocha.
Nature (2016)
Age-Related Clonal Hematopoiesis Associated with Adverse Outcomes
Siddhartha Jaiswal;Pierre Fontanillas;Jason Flannick;Jason Flannick;Alisa Manning.
The New England Journal of Medicine (2014)
Efficacy of atenolol and captopril in reducing risk of macrovascular and microvascular complications in type 2 diabetes: UKPDS 39
M R Stearne;S L Palmer;M S Hammersley;S L Franklin.
(1998)
The genetic architecture of type 2 diabetes
Christian Fuchsberger;Christian Fuchsberger;Jason A. Flannick;Jason A. Flannick;Tanya M. Teslovich;Anubha Mahajan.
Nature (2016)
An epigenetic biomarker of aging for lifespan and healthspan
Morgan E. Levine;Ake T. Lu;Austin Quach;Brian H. Chen.
Aging (Albany NY) (2018)
The interleukin-6 receptor as a target for prevention of coronary heart disease: a mendelian randomisation analysis.
D I Swerdlow;M V Holmes;K B Kuchenbaecker.
The Lancet (2012)
Loss-of-function mutations in APOC3, triglycerides, and coronary disease
Jacy Crosby;Gina M. Peloso;Gina M. Peloso;Paul L. Auer;David R. Crosslin.
The New England Journal of Medicine (2014)
Association between alcohol and cardiovascular disease:Mendelian randomisation analysis based on individual participant data
Michael V Holmes;Michael V Holmes;Caroline E Dale;Luisa Zuccolo;Richard J Silverwood.
BMJ (2014)
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