D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 84 Citations 38,502 292 World Ranking 9813 National Ranking 5188

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Genetics

His primary areas of investigation include Pulmonary hypertension, Internal medicine, Idiopathic pulmonary fibrosis, Respiratory disease and Pathology. James E. Loyd has included themes like BMPR2, Disease, Pathogenesis and Surgery in his Pulmonary hypertension study. His biological study spans a wide range of topics, including Median survival, Evidence-based medicine and Intensive care medicine.

The study incorporates disciplines such as Endocrinology, Promoter polymorphism and Pediatrics in addition to Internal medicine. His studies deal with areas such as Pulmonary fibrosis, Immunology, Cohort and Interstitial lung disease as well as Idiopathic pulmonary fibrosis. His Pathology research incorporates themes from Surfactant protein C and Lung.

His most cited work include:

  • An Official American Thoracic Society/European Respiratory Society Statement: Update of the International Multidisciplinary Classification of the Idiopathic Interstitial Pneumonias (1870 citations)
  • Heterozygous germline mutations in BMPR2 , encoding a TGF-β receptor, cause familial primary pulmonary hypertension (1186 citations)
  • An Imbalance between the Excretion of Thromboxane and Prostacyclin Metabolites in Pulmonary Hypertension (1001 citations)

What are the main themes of his work throughout his whole career to date?

James E. Loyd mostly deals with Pulmonary hypertension, Internal medicine, Pathology, Lung and BMPR2. His Pulmonary hypertension study combines topics from a wide range of disciplines, such as Respiratory disease, Anesthesia, Receptor, Disease and Pulmonary artery. His Internal medicine research is multidisciplinary, incorporating perspectives in Gastroenterology, Endocrinology, Surgery and Cardiology.

James E. Loyd interconnects Idiopathic pulmonary fibrosis and Interstitial lung disease in the investigation of issues within Pathology. The various areas that James E. Loyd examines in his Idiopathic pulmonary fibrosis study include Telomere, Immunology and Intensive care medicine. His research in BMPR2 intersects with topics in Mutation, Penetrance, Pathogenesis and Bioinformatics.

He most often published in these fields:

  • Pulmonary hypertension (33.67%)
  • Internal medicine (32.32%)
  • Pathology (24.58%)

What were the highlights of his more recent work (between 2014-2021)?

  • Internal medicine (32.32%)
  • Pulmonary hypertension (33.67%)
  • Pathology (24.58%)

In recent papers he was focusing on the following fields of study:

Internal medicine, Pulmonary hypertension, Pathology, Idiopathic pulmonary fibrosis and Pulmonary fibrosis are his primary areas of study. His research integrates issues of Gastroenterology, Endocrinology, Pharmacology and Cardiology in his study of Internal medicine. James E. Loyd has researched Pulmonary hypertension in several fields, including BMPR2, Nitric oxide and Pulmonary artery.

The BMPR2 study combines topics in areas such as ACVRL1 and Pathogenesis. His Pathology research is multidisciplinary, relying on both Idiopathic interstitial pneumonia, Lung and Interstitial lung disease. His Idiopathic pulmonary fibrosis study incorporates themes from Genetics, Gene, Randomized controlled trial, Genotyping and Disease.

Between 2014 and 2021, his most popular works were:

  • Pulmonary Arterial Hypertension: A Current Perspective on Established and Emerging Molecular Genetic Defects. (129 citations)
  • Rare Variants in RTEL1 Are Associated with Familial Interstitial Pneumonia (115 citations)
  • Single-cell RNA sequencing reveals profibrotic roles of distinct epithelial and mesenchymal lineages in pulmonary fibrosis (104 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Genetics

James E. Loyd mainly investigates Idiopathic pulmonary fibrosis, Disease, Pathology, Gene and Genetics. His Idiopathic pulmonary fibrosis research is multidisciplinary, incorporating elements of DNA Mutational Analysis, Genetic testing, Mutation and Locus. His Disease research includes themes of Mendelian inheritance, Genomics, Penetrance, Computational biology and DNA sequencing.

The concepts of his Pathology study are interwoven with issues in Internal medicine, Pulmonary hypertension, Lung and Idiopathic interstitial pneumonia. His Internal medicine research includes elements of Endocrinology, Genetically modified mouse and Downregulation and upregulation. His Gene research incorporates elements of Senescence, Host and Immunology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

An Official American Thoracic Society/European Respiratory Society Statement: Update of the International Multidisciplinary Classification of the Idiopathic Interstitial Pneumonias

William D. Travis;Ulrich Costabel;David M. Hansell;Talmadge E. King.
American Journal of Respiratory and Critical Care Medicine (2013)

3005 Citations

An Imbalance between the Excretion of Thromboxane and Prostacyclin Metabolites in Pulmonary Hypertension

Brian W. Christman;Charles D. McPherson;John H. Newman;Gayle A. King.
The New England Journal of Medicine (1992)

1559 Citations

Heterozygous germline mutations in BMPR2 , encoding a TGF-β receptor, cause familial primary pulmonary hypertension

Kirk B. Lane;Rajiv D. Machado;Michael W. Pauciulo;Jennifer R. Thomson.
Nature Genetics (2000)

1552 Citations

Diagnosis and Assessment of Pulmonary Arterial Hypertension

David B. Badesch;Hunter C. Champion;Miguel Angel Gomez Sanchez;Marius M. Hoeper.
Journal of the American College of Cardiology (2009)

1443 Citations

Telomerase Mutations in Families with Idiopathic Pulmonary Fibrosis

Mary Y. Armanios;Julian J.-L. Chen;Joy D. Cogan;Jonathan K. Alder.
The New England Journal of Medicine (2007)

1388 Citations

Continuous intravenous epoprostenol for pulmonary hypertension due to the scleroderma spectrum of disease. A randomized, controlled trial.

David B. Badesch;Victor F. Tapson;Michael D. McGoon;Bruce H. Brundage.
Annals of Internal Medicine (2000)

1387 Citations

Clinical Practice Guidelines for the Management of Patients with Histoplasmosis: 2007 Update by the Infectious Diseases Society of America

L. Joseph Wheat;Alison G. Freifeld;Martin B. Kleiman;John W. Baddley;John W. Baddley.
Clinical Infectious Diseases (2007)

1372 Citations

Screening, Early Detection, and Diagnosis of Pulmonary Arterial Hypertension: ACCP Evidence-Based Clinical Practice Guidelines

Michael McGoon;David Gutterman;Virginia Steen;Robin Barst.
Chest (2004)

1354 Citations

Acute Exacerbations of Idiopathic Pulmonary Fibrosis

Harold R. Collard;Bethany B. Moore;Kevin R. Flaherty;Kevin K. Brown.
American Journal of Respiratory and Critical Care Medicine (2007)

1220 Citations

A common MUC5B promoter polymorphism and pulmonary fibrosis

Max A. Seibold;Anastasia L. Wise;Marcy C. Speer;Mark P. Steele.
The New England Journal of Medicine (2011)

1015 Citations

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