Member of the Association of American Physicians
His primary areas of investigation include Pulmonary hypertension, Internal medicine, Idiopathic pulmonary fibrosis, Respiratory disease and Pathology. James E. Loyd has included themes like BMPR2, Disease, Pathogenesis and Surgery in his Pulmonary hypertension study. His biological study spans a wide range of topics, including Median survival, Evidence-based medicine and Intensive care medicine.
The study incorporates disciplines such as Endocrinology, Promoter polymorphism and Pediatrics in addition to Internal medicine. His studies deal with areas such as Pulmonary fibrosis, Immunology, Cohort and Interstitial lung disease as well as Idiopathic pulmonary fibrosis. His Pathology research incorporates themes from Surfactant protein C and Lung.
James E. Loyd mostly deals with Pulmonary hypertension, Internal medicine, Pathology, Lung and BMPR2. His Pulmonary hypertension study combines topics from a wide range of disciplines, such as Respiratory disease, Anesthesia, Receptor, Disease and Pulmonary artery. His Internal medicine research is multidisciplinary, incorporating perspectives in Gastroenterology, Endocrinology, Surgery and Cardiology.
James E. Loyd interconnects Idiopathic pulmonary fibrosis and Interstitial lung disease in the investigation of issues within Pathology. The various areas that James E. Loyd examines in his Idiopathic pulmonary fibrosis study include Telomere, Immunology and Intensive care medicine. His research in BMPR2 intersects with topics in Mutation, Penetrance, Pathogenesis and Bioinformatics.
Internal medicine, Pulmonary hypertension, Pathology, Idiopathic pulmonary fibrosis and Pulmonary fibrosis are his primary areas of study. His research integrates issues of Gastroenterology, Endocrinology, Pharmacology and Cardiology in his study of Internal medicine. James E. Loyd has researched Pulmonary hypertension in several fields, including BMPR2, Nitric oxide and Pulmonary artery.
The BMPR2 study combines topics in areas such as ACVRL1 and Pathogenesis. His Pathology research is multidisciplinary, relying on both Idiopathic interstitial pneumonia, Lung and Interstitial lung disease. His Idiopathic pulmonary fibrosis study incorporates themes from Genetics, Gene, Randomized controlled trial, Genotyping and Disease.
James E. Loyd mainly investigates Idiopathic pulmonary fibrosis, Disease, Pathology, Gene and Genetics. His Idiopathic pulmonary fibrosis research is multidisciplinary, incorporating elements of DNA Mutational Analysis, Genetic testing, Mutation and Locus. His Disease research includes themes of Mendelian inheritance, Genomics, Penetrance, Computational biology and DNA sequencing.
The concepts of his Pathology study are interwoven with issues in Internal medicine, Pulmonary hypertension, Lung and Idiopathic interstitial pneumonia. His Internal medicine research includes elements of Endocrinology, Genetically modified mouse and Downregulation and upregulation. His Gene research incorporates elements of Senescence, Host and Immunology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
An Official American Thoracic Society/European Respiratory Society Statement: Update of the International Multidisciplinary Classification of the Idiopathic Interstitial Pneumonias
William D. Travis;Ulrich Costabel;David M. Hansell;Talmadge E. King.
American Journal of Respiratory and Critical Care Medicine (2013)
An Imbalance between the Excretion of Thromboxane and Prostacyclin Metabolites in Pulmonary Hypertension
Brian W. Christman;Charles D. McPherson;John H. Newman;Gayle A. King.
The New England Journal of Medicine (1992)
Heterozygous germline mutations in BMPR2 , encoding a TGF-β receptor, cause familial primary pulmonary hypertension
Kirk B. Lane;Rajiv D. Machado;Michael W. Pauciulo;Jennifer R. Thomson.
Nature Genetics (2000)
Diagnosis and Assessment of Pulmonary Arterial Hypertension
David B. Badesch;Hunter C. Champion;Miguel Angel Gomez Sanchez;Marius M. Hoeper.
Journal of the American College of Cardiology (2009)
Telomerase Mutations in Families with Idiopathic Pulmonary Fibrosis
Mary Y. Armanios;Julian J.-L. Chen;Joy D. Cogan;Jonathan K. Alder.
The New England Journal of Medicine (2007)
Continuous intravenous epoprostenol for pulmonary hypertension due to the scleroderma spectrum of disease. A randomized, controlled trial.
David B. Badesch;Victor F. Tapson;Michael D. McGoon;Bruce H. Brundage.
Annals of Internal Medicine (2000)
Clinical Practice Guidelines for the Management of Patients with Histoplasmosis: 2007 Update by the Infectious Diseases Society of America
L. Joseph Wheat;Alison G. Freifeld;Martin B. Kleiman;John W. Baddley;John W. Baddley.
Clinical Infectious Diseases (2007)
Screening, Early Detection, and Diagnosis of Pulmonary Arterial Hypertension: ACCP Evidence-Based Clinical Practice Guidelines
Michael McGoon;David Gutterman;Virginia Steen;Robin Barst.
Chest (2004)
Acute Exacerbations of Idiopathic Pulmonary Fibrosis
Harold R. Collard;Bethany B. Moore;Kevin R. Flaherty;Kevin K. Brown.
American Journal of Respiratory and Critical Care Medicine (2007)
A common MUC5B promoter polymorphism and pulmonary fibrosis
Max A. Seibold;Anastasia L. Wise;Marcy C. Speer;Mark P. Steele.
The New England Journal of Medicine (2011)
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