2007 - Fellow of the American Association for the Advancement of Science (AAAS)
The scientist’s investigation covers issues in Cancer research, Lung cancer, Surgery, Tumor suppressor gene and Apoptosis. His Cancer research research is multidisciplinary, incorporating elements of Cell culture, Cell growth, Viral vector, Genetic enhancement and In vivo. His Lung cancer study which covers Mutation that intersects with Exon.
The concepts of his Surgery study are interwoven with issues in Respiratory disease and Cancer. His Apoptosis research includes themes of Molecular biology, Downregulation and upregulation and Cell biology. His study in Internal medicine is interdisciplinary in nature, drawing from both Gastroenterology and Oncology.
Jack A. Roth mainly investigates Cancer research, Lung cancer, Internal medicine, Cancer and Surgery. Jack A. Roth has researched Cancer research in several fields, including Cell culture, Apoptosis, Genetic enhancement, Gene and Tumor suppressor gene. His research in Cell culture intersects with topics in Molecular biology and Cell.
His Lung cancer study incorporates themes from Immunology, Chemotherapy and Carcinogenesis. His biological study spans a wide range of topics, including Gastroenterology and Oncology. His Surgery research includes elements of Respiratory disease, Lung and Esophageal cancer.
His primary scientific interests are in Lung cancer, Internal medicine, Cancer research, Oncology and Cancer. His study focuses on the intersection of Lung cancer and fields such as Surgery with connections in the field of Esophagectomy. His study in Chemotherapy, Confidence interval, Carcinoma and Neoadjuvant therapy is done as part of Internal medicine.
His Cancer research research integrates issues from Cell, Cancer cell, Immune system, Immunotherapy and non-small cell lung cancer. He combines subjects such as PD-L1, Targeted therapy, Hazard ratio, Radiation therapy and Cohort with his study of Oncology. His Cancer study combines topics from a wide range of disciplines, such as Mesothelioma and Gene, Genetic enhancement.
Lung cancer, Internal medicine, Oncology, Cancer research and Cancer are his primary areas of study. His Lung cancer study combines topics in areas such as Radiation therapy, Surgery, Immunology, Carcinoma and Adenocarcinoma. The Surgery study combines topics in areas such as Adverse effect and Hazard ratio.
His work carried out in the field of Oncology brings together such families of science as Survival rate, Immune checkpoint inhibitors, Confidence interval, Targeted therapy and Lung. His Cancer research research is multidisciplinary, relying on both Tumor suppressor gene, Treatment of lung cancer, PI3K/AKT/mTOR pathway and Immune system. His Cancer research incorporates elements of Mesothelioma and Gene, Genetic enhancement.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Clinical and Biological Features Associated With Epidermal Growth Factor Receptor Gene Mutations in Lung Cancers
Hisayuki Shigematsu;Hisayuki Shigematsu;Li Lin;Takao Takahashi;Masaharu Nomura.
Journal of the National Cancer Institute (2005)
Somatic mutations affect key pathways in lung adenocarcinoma
Li Ding;Gad Getz;David A. Wheeler;Elaine R. Mardis.
Nature (2008)
Chemotherapy followed by surgery compared with surgery alone for localized esophageal cancer
David P. Kelsen;Robert Ginsberg;Thomas F. Pajak;Daniel G. Sheahan.
The New England Journal of Medicine (1998)
BRAF and RAS mutations in human lung cancer and melanoma
Marcia S. Brose;Patricia Volpe;Michael Feldman;Madhu Kumar.
Cancer Research (2002)
A Randomized Trial Comparing Perioperative Chemotherapy and Surgery With Surgery Alone in Resectable Stage IIIA Non-Small-Cell Lung Cancer
Jack A Roth;Frank V Fossella;Ritsuko Komaki;M. Bernadette Ryan.
Journal of the National Cancer Institute (1994)
Characterizing the cancer genome in lung adenocarcinoma
Barbara A. Weir;Barbara A. Weir;Michele S. Woo;Gad Getz;Sven Perner;Sven Perner.
Nature (2007)
Stereotactic ablative radiotherapy versus lobectomy for operable stage I non-small-cell lung cancer: a pooled analysis of two randomised trials
Joe Y Chang;Suresh Senan;Marinus A. Paul;Reza John Mehran.
Lancet Oncology (2015)
Wild-type human p53 and a temperature-sensitive mutant induce Fas/APO-1 expression.
L B Owen-Schaub;W Zhang;J C Cusack;L S Angelo.
Molecular and Cellular Biology (1995)
Retrovirus-mediated wild-type p53 gene transfer to tumors of patients with lung cancer.
J. A. Roth;D. Nguyen;D. D. Lawrence;B. L. Kemp.
Nature Medicine (1996)
Gene Therapy for Cancer: What Have We Done and Where Are We Going?
Jack A. Roth;Richard J. Cristiano.
Journal of the National Cancer Institute (1997)
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