What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in Cell biology, Glioma, Stem cell, Neuroglia and Biochemistry.
His Cell biology research includes elements of Molecular biology, Embryonic stem cell and Immunology.
His Immunology study combines topics in areas such as Progenitor cell, Transforming growth factor and Neuroepithelial cell, Neural stem cell.
His Glioma research incorporates elements of Spheroid, Vascular niche, PI3K/AKT/mTOR pathway and Stem like cell.
Hervé Chneiweiss interconnects Medulloblastoma, Cell sorting and Glioblastoma in the investigation of issues within Stem cell.
His Neuroglia research includes themes of Astrocyte and Caspase 8.
His most cited work include:
- Spinocerebellar ataxia 3 and Machado-Joseph disease: clinical, molecular, and neuropathological features (356 citations)
- PEA-15 Mediates Cytoplasmic Sequestration of ERK MAP Kinase (271 citations)
- Molecular and Clinical Correlations in Spinocerebellar Ataxia 2: A Study of 32 Families (220 citations)
What are the main themes of his work throughout his whole career to date?
Cell biology, Cancer research, Glioma, Cancer stem cell and Stem cell are his primary areas of study.
His work deals with themes such as Biochemistry and Astrocyte, which intersect with Cell biology.
His Cancer research research also works with subjects such as
- Progenitor cell most often made with reference to Immunology,
- Telomere most often made with reference to Cancer cell.
The Cancer stem cell study combines topics in areas such as EGR1, Homeobox protein NANOG, Pharmacology and Cell growth.
Hervé Chneiweiss works in the field of Stem cell, namely Neural stem cell.
His research integrates issues of Apoptosis and Kinase in his study of Signal transduction.
He most often published in these fields:
- Cell biology (35.29%)
- Cancer research (18.72%)
- Glioma (20.32%)
What were the highlights of his more recent work (between 2016-2021)?
- Cancer research (18.72%)
- Glioma (20.32%)
- Cell (9.09%)
In recent papers he was focusing on the following fields of study:
Hervé Chneiweiss focuses on Cancer research, Glioma, Cell, Genome editing and Cancer stem cell.
His work carried out in the field of Cancer research brings together such families of science as Cell culture, Cell growth, Cancer cell, Phenotype and Pharmacology.
His Glioma research is multidisciplinary, incorporating perspectives in Biopsy, Magnetic resonance imaging and Nuclear medicine.
The concepts of his Cancer stem cell study are interwoven with issues in Signal transduction and Gene expression profiling.
His study in the fields of MAPK/ERK pathway under the domain of Signal transduction overlaps with other disciplines such as Population and Bisacodyl.
His Cell biology research is multidisciplinary, relying on both Telomere Homeostasis and Genetics.
Between 2016 and 2021, his most popular works were:
- Glut3 Addiction Is a Druggable Vulnerability for a Molecularly Defined Subpopulation of Glioblastoma (53 citations)
- The oncolytic virus Delta-24-RGD elicits an antitumor effect in pediatric glioma and DIPG mouse models. (32 citations)
- Radiosensitization Effect of Talazoparib, a Parp Inhibitor, on Glioblastoma Stem Cells Exposed to Low and High Linear Energy Transfer Radiation. (32 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Cancer research, Glioma, Genome editing, Cancer stem cell and Cell.
His study focuses on the intersection of Cancer research and fields such as Cell growth with connections in the field of Progenitor cell, Mitosis, Immunology and Cyclin D1.
His study in Glioma is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha, Chemokine and Growth factor.
He combines subjects such as Phenotype and Signal transduction with his study of Cancer stem cell.
His Cell study combines topics from a wide range of disciplines, such as Chromatin and Transcription factor.
His Stem cell study incorporates themes from Cell cycle checkpoint, Radioresistance, DNA repair and Temozolomide.
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