His scientific interests lie mostly in Cell biology, Glioma, Stem cell, Neuroglia and Biochemistry. His Cell biology research includes elements of Molecular biology, Embryonic stem cell and Immunology. His Immunology study combines topics in areas such as Progenitor cell, Transforming growth factor and Neuroepithelial cell, Neural stem cell.
His Glioma research incorporates elements of Spheroid, Vascular niche, PI3K/AKT/mTOR pathway and Stem like cell. Hervé Chneiweiss interconnects Medulloblastoma, Cell sorting and Glioblastoma in the investigation of issues within Stem cell. His Neuroglia research includes themes of Astrocyte and Caspase 8.
Cell biology, Cancer research, Glioma, Cancer stem cell and Stem cell are his primary areas of study. His work deals with themes such as Biochemistry and Astrocyte, which intersect with Cell biology. His Cancer research research also works with subjects such as
The Cancer stem cell study combines topics in areas such as EGR1, Homeobox protein NANOG, Pharmacology and Cell growth. Hervé Chneiweiss works in the field of Stem cell, namely Neural stem cell. His research integrates issues of Apoptosis and Kinase in his study of Signal transduction.
Hervé Chneiweiss focuses on Cancer research, Glioma, Cell, Genome editing and Cancer stem cell. His work carried out in the field of Cancer research brings together such families of science as Cell culture, Cell growth, Cancer cell, Phenotype and Pharmacology. His Glioma research is multidisciplinary, incorporating perspectives in Biopsy, Magnetic resonance imaging and Nuclear medicine.
The concepts of his Cancer stem cell study are interwoven with issues in Signal transduction and Gene expression profiling. His study in the fields of MAPK/ERK pathway under the domain of Signal transduction overlaps with other disciplines such as Population and Bisacodyl. His Cell biology research is multidisciplinary, relying on both Telomere Homeostasis and Genetics.
His primary areas of investigation include Cancer research, Glioma, Genome editing, Cancer stem cell and Cell. His study focuses on the intersection of Cancer research and fields such as Cell growth with connections in the field of Progenitor cell, Mitosis, Immunology and Cyclin D1. His study in Glioma is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha, Chemokine and Growth factor.
He combines subjects such as Phenotype and Signal transduction with his study of Cancer stem cell. His Cell study combines topics from a wide range of disciplines, such as Chromatin and Transcription factor. His Stem cell study incorporates themes from Cell cycle checkpoint, Radioresistance, DNA repair and Temozolomide.
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Spinocerebellar ataxia 3 and Machado-Joseph disease: clinical, molecular, and neuropathological features
Alexandra Durr;Giovanni Stevanin;Geraldine Cancel;Charles Duyckaerts.
Annals of Neurology (1996)
PEA-15 Mediates Cytoplasmic Sequestration of ERK MAP Kinase
Etienne Formstecher;Joe W. Ramos;Mireille Fauquet;David A. Calderwood.
Developmental Cell (2001)
Molecular and Clinical Correlations in Spinocerebellar Ataxia 2: A Study of 32 Families
Géraldine Cancel;Alexandra Dürr;Olivier Didierjean;Georges Imbert.
Human Molecular Genetics (1997)
Autosomal dominant cerebellar ataxia type I in Martinique (French West Indies). Clinical and neuropathological analysis of 53 patients from three unrelated SCA2 families
A Dürr;D Smadja;G Cancel;A Lezin.
Intracellular substrates for extracellular signaling. Characterization of a ubiquitous, neuron-enriched phosphoprotein (stathmin).
A. Sobel;M. C. Boutterin;Laura Beretta;H. Chneiweiss.
Journal of Biological Chemistry (1989)
Characterization of PEA-15, a major substrate for protein kinase C in astrocytes.
H Araujo;N Danziger;J Cordier;J Glowinski.
Journal of Biological Chemistry (1993)
Intraneuronal delivery of protein kinase C pseudosubstrate leads to growth cone collapse
L Theodore;D Derossi;G Chassaing;B Llirbat.
The Journal of Neuroscience (1995)
The miR 302-367 cluster drastically affects self-renewal and infiltration properties of glioma-initiating cells through CXCR4 repression and consequent disruption of the SHH-GLI-NANOG network
Mohamed Fareh;Laurent Turchi;Virginie Virolle;D Debruyne.
Cell Death & Differentiation (2012)
Knock-Out of the Neural Death Effector Domain Protein PEA-15 Demonstrates That Its Expression Protects Astrocytes from TNFα-Induced Apoptosis
Daniel Kitsberg;Etienne Formstecher;Mireille Fauquet;Miroslav Kubes;Miroslav Kubes.
The Journal of Neuroscience (1999)
Vasoactive intestinal polypeptide receptors linked to an adenylate cyclase, and their relationship with biogenic amine- and somatostatin-sensitive adenylate cyclases on central neuronal and glial cells in primary cultures.
H. Chneiweiss;J. Glowinski;J. Prémont.
Journal of Neurochemistry (1985)
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