2008 - William Allan Award, the American Society of Human Genetics
2007 - Fellow of the American Academy of Arts and Sciences
Haig H. Kazazian spends much of his time researching Genetics, Retrotransposon, Human genome, Gene and Genome. His study involves Transposable element, Nucleic acid sequence, DNA, Allele and genomic DNA, a branch of Genetics. The Retrotransposon study combines topics in areas such as RNA, Reverse transcriptase, Long interspersed nuclear element, Open reading frame and Consensus sequence.
Haig H. Kazazian interconnects Insertional mutagenesis, RNA interference, Transgene and Somatic cell in the investigation of issues within Human genome. Haig H. Kazazian has researched Gene in several fields, including Molecular biology and Mutation. His research in Genome intersects with topics in Evolutionary biology, Disease and Untranslated region.
Genetics, Gene, Retrotransposon, Molecular biology and Human genome are his primary areas of study. His study in Genome, Mutation, Allele, Genetic linkage and Globin falls within the category of Genetics. His research on Gene often connects related areas such as DNA.
His Retrotransposon research includes themes of Long interspersed nuclear element, Genome evolution, RNA and Somatic cell. Haig H. Kazazian usually deals with Molecular biology and limits it to topics linked to Thalassemia and Prenatal diagnosis. Many of his studies on Human genome involve topics that are commonly interrelated, such as Mobile genetic elements.
Haig H. Kazazian mainly investigates Genetics, Retrotransposon, Human genome, Genome and Somatic cell. Genome evolution, Gene, Pseudogene, Ribonucleoprotein and Gene duplication are among the areas of Genetics where the researcher is concentrating his efforts. His Retrotransposon research integrates issues from Insertional mutagenesis, Long interspersed nuclear element, RNA, Cell biology and Alu element.
As a member of one scientific family, Haig H. Kazazian mostly works in the field of Human genome, focusing on Transposable element and, on occasion, Mobile genetic elements, Endogenous retrovirus and Disease. Haig H. Kazazian has included themes like Evolutionary biology, Genome instability and DNA, DNA sequencing in his Genome study. His Somatic cell study which covers Cancer that intersects with Cancer research.
His scientific interests lie mostly in Retrotransposon, Genetics, Human genome, Genome and Somatic cell. His Retrotransposon study integrates concerns from other disciplines, such as Insertional mutagenesis, Ribonucleoprotein, Molecular biology, RNA interference and Cell biology. His studies in Alu element, Gene, Long interspersed nuclear element, RNA and Pseudogene are all subfields of Genetics research.
His Human genome study incorporates themes from Genome evolution, Computational biology and Transgene. His Genome research includes elements of Evolutionary biology and Exon shuffling. His Somatic cell study combines topics in areas such as Immunohistochemistry, Progressive disease, Cell culture and Adenocarcinoma.
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Mobile elements: drivers of genome evolution.
Haig H. Kazazian.
Human L1 Retrotransposon Encodes a Conserved Endonuclease Required for Retrotransposition
Qinghua Feng;John V. Moran;Haig H. Kazazian;Jef D. Boeke.
Linkage of β-thalassaemia mutations and β-globin gene polymorphisms with DNA polymorphisms in human β-globin gene cluster
Stuart H. Orkin;Haig H. Kazazian;Stylianos E. Antonarakis;Sabra C. Goff.
High Frequency Retrotransposition in Cultured Mammalian Cells
John V. Moran;Susan E. Holmes;Thierry P. Naas;Ralph J. DeBerardinis.
Inversions disrupting the factor VIII gene are a common cause of severe haemophilia A.
Delia Lakich;Haig H. Kazazian;Stylianos E. Antonarakis;Stylianos E. Antonarakis;Jane Gitschier.
Nature Genetics (1993)
Biology of Mammalian L1 Retrotransposons
Eric M. Ostertag;Haig H. Kazazian.
Annual Review of Genetics (2001)
Hot L1s account for the bulk of retrotransposition in the human population
Brook Brouha;Joshua Schustak;Richard M. Badge;Richard M. Badge;Sheila Lutz-Prigge.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Haemophilia A resulting from de novo insertion of L1 sequences represents a novel mechanism for mutation in man.
Haig H. Kazazian;Corinne Wong;Hagop Youssoufian;Hagop Youssoufian;Alan F. Scott.
Targeted disruption of the mouse factor VIII gene produces a model of haemophilia A
L. Bi;A. M. Lawler;Stylianos Antonarakis;K. A. High.
Nature Genetics (1995)
Human L1 retrotransposition: cis preference versus trans complementation.
Wei Wei;Nicolas Gilbert;Siew Loon Ooi;Joseph F. Lawler.
Molecular and Cellular Biology (2001)
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