The scientist’s investigation covers issues in Endocrinology, Internal medicine, Cell biology, Heart failure and Mitochondrion. His research in Endocrinology intersects with topics in Cardiac function curve, Cardiomyopathy, Anticipation and Cardiology. Many of his research projects under Internal medicine are closely connected to Cardiac response with Cardiac response, tying the diverse disciplines of science together.
His Cell biology study combines topics from a wide range of disciplines, such as GATA4, Cancer research, Bioinformatics and Calcineurin. His study in Heart failure is interdisciplinary in nature, drawing from both Receptor, Adenylyl cyclase and Beta adrenergic receptor kinase. His Mitochondrion study combines topics in areas such as mitochondrial fusion, MFN2 and Mitophagy.
His primary areas of study are Cell biology, Internal medicine, Endocrinology, Mitochondrion and Heart failure. His biological study spans a wide range of topics, including Autophagy, Apoptosis, Biochemistry and Mitochondrial permeability transition pore. His Internal medicine study frequently draws parallels with other fields, such as Cardiology.
Gerald W. Dorn has included themes like Cardiac function curve, Receptor, Adrenergic receptor and Signal transduction in his Endocrinology study. His Mitochondrion study integrates concerns from other disciplines, such as mitochondrial fusion, MFN2 and Mitophagy. His Cardiomyopathy research includes themes of Mutation, Genetics, Dilated cardiomyopathy and Bioinformatics.
Gerald W. Dorn mostly deals with Cell biology, Mitochondrion, MFN2, Mitophagy and mitochondrial fusion. His Cell biology research incorporates themes from Autophagy, Mutant, PINK1 and Gene knockdown. In the field of Autophagy, his study on Autolysosome and Chaperone-mediated autophagy overlaps with subjects such as Knowledge base and Interpretation.
The various areas that Gerald W. Dorn examines in his Mitochondrion study include Pathogenesis, Apoptosis, Lung, Myocyte and Organelle. His MFN2 research includes elements of Therapeutic approach, Mitofusin-2, Bone resorption and Cardiomyopathy. His work in the fields of mitochondrial fusion, such as MFN1, intersects with other areas such as Dynamism.
Gerald W. Dorn spends much of his time researching Mitochondrion, Cell biology, Mitophagy, mitochondrial fusion and Heart failure. The study of Mitochondrion is intertwined with the study of Programmed cell death in a number of ways. Gerald W. Dorn interconnects Autophagy and Ubiquitin in the investigation of issues within Cell biology.
His Mitophagy research is multidisciplinary, incorporating perspectives in Necrotic cell, Tissue homeostasis, Homeostasis and Mitochondrial fission. Gerald W. Dorn has researched mitochondrial fusion in several fields, including Agonist and Mutant. Heart failure is the topic of his studies on Internal medicine and Cardiology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
PINK1- Phosphorylated Mitofusin 2 is a Parkin Receptor for Culling Damaged Mitochondria
Yun Chen;Gerald W. Dorn.
Science (2013)
Cytoplasmic Signaling Pathways That Regulate Cardiac Hypertrophy
Jeffery D Molkentin;Gerald W Dorn.
Annual Review of Physiology (2001)
Enhanced Gαq signaling: A common pathway mediates cardiac hypertrophy and apoptotic heart failure
John W. Adams;Yoshihito Sakata;Michael G. Davis;Valerie P. Sah.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Early and Delayed Consequences of β2-Adrenergic Receptor Overexpression in Mouse Hearts Critical Role for Expression Level
Stephen B. Liggett;Nicole M. Tepe;John N. Lorenz;Amy M. Canning.
Circulation (2000)
Nix Is Critical to Two Distinct Phases of Mitophagy, Reactive Oxygen Species-mediated Autophagy Induction and Parkin-Ubiquitin-p62-mediated Mitochondrial Priming
Wen Xing Ding;Wen Xing Ding;Hong Min Ni;Hong Min Ni;Min Li;Yong Liao.
Journal of Biological Chemistry (2010)
Mitochondrial Oxidative Stress Mediates Angiotensin II–Induced Cardiac Hypertrophy and Gαq Overexpression–Induced Heart Failure
Dao Fu Dai;Simon C. Johnson;Jason J. Villarin;Michael T. Chin.
Circulation Research (2011)
Human phospholamban null results in lethal dilated cardiomyopathy revealing a critical difference between mouse and human
Kobra Haghighi;Fotis Kolokathis;Luke Pater;Roy A. Lynch.
Journal of Clinical Investigation (2003)
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