D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 65 Citations 32,540 134 World Ranking 3984 National Ranking 2008

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Genetics
  • Internal medicine

The scientist’s investigation covers issues in Endocrinology, Internal medicine, Cell biology, Heart failure and Mitochondrion. His research in Endocrinology intersects with topics in Cardiac function curve, Cardiomyopathy, Anticipation and Cardiology. Many of his research projects under Internal medicine are closely connected to Cardiac response with Cardiac response, tying the diverse disciplines of science together.

His Cell biology study combines topics from a wide range of disciplines, such as GATA4, Cancer research, Bioinformatics and Calcineurin. His study in Heart failure is interdisciplinary in nature, drawing from both Receptor, Adenylyl cyclase and Beta adrenergic receptor kinase. His Mitochondrion study combines topics in areas such as mitochondrial fusion, MFN2 and Mitophagy.

His most cited work include:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • PINK1- Phosphorylated Mitofusin 2 is a Parkin Receptor for Culling Damaged Mitochondria (730 citations)
  • Cytoplasmic Signaling Pathways That Regulate Cardiac Hypertrophy (641 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Cell biology, Internal medicine, Endocrinology, Mitochondrion and Heart failure. His biological study spans a wide range of topics, including Autophagy, Apoptosis, Biochemistry and Mitochondrial permeability transition pore. His Internal medicine study frequently draws parallels with other fields, such as Cardiology.

Gerald W. Dorn has included themes like Cardiac function curve, Receptor, Adrenergic receptor and Signal transduction in his Endocrinology study. His Mitochondrion study integrates concerns from other disciplines, such as mitochondrial fusion, MFN2 and Mitophagy. His Cardiomyopathy research includes themes of Mutation, Genetics, Dilated cardiomyopathy and Bioinformatics.

He most often published in these fields:

  • Cell biology (39.29%)
  • Internal medicine (38.39%)
  • Endocrinology (29.02%)

What were the highlights of his more recent work (between 2016-2021)?

  • Cell biology (39.29%)
  • Mitochondrion (22.77%)
  • MFN2 (11.61%)

In recent papers he was focusing on the following fields of study:

Gerald W. Dorn mostly deals with Cell biology, Mitochondrion, MFN2, Mitophagy and mitochondrial fusion. His Cell biology research incorporates themes from Autophagy, Mutant, PINK1 and Gene knockdown. In the field of Autophagy, his study on Autolysosome and Chaperone-mediated autophagy overlaps with subjects such as Knowledge base and Interpretation.

The various areas that Gerald W. Dorn examines in his Mitochondrion study include Pathogenesis, Apoptosis, Lung, Myocyte and Organelle. His MFN2 research includes elements of Therapeutic approach, Mitofusin-2, Bone resorption and Cardiomyopathy. His work in the fields of mitochondrial fusion, such as MFN1, intersects with other areas such as Dynamism.

Between 2016 and 2021, his most popular works were:

  • Abrogating Mitochondrial Dynamics in Mouse Hearts Accelerates Mitochondrial Senescence (111 citations)
  • A Rab5 endosomal pathway mediates Parkin-dependent mitochondrial clearance. (89 citations)
  • MFN2 agonists reverse mitochondrial defects in preclinical models of Charcot-Marie-Tooth disease type 2A. (77 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Genetics
  • Internal medicine

Gerald W. Dorn spends much of his time researching Mitochondrion, Cell biology, Mitophagy, mitochondrial fusion and Heart failure. The study of Mitochondrion is intertwined with the study of Programmed cell death in a number of ways. Gerald W. Dorn interconnects Autophagy and Ubiquitin in the investigation of issues within Cell biology.

His Mitophagy research is multidisciplinary, incorporating perspectives in Necrotic cell, Tissue homeostasis, Homeostasis and Mitochondrial fission. Gerald W. Dorn has researched mitochondrial fusion in several fields, including Agonist and Mutant. Heart failure is the topic of his studies on Internal medicine and Cardiology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

7788 Citations

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

4781 Citations

PINK1- Phosphorylated Mitofusin 2 is a Parkin Receptor for Culling Damaged Mitochondria

Yun Chen;Gerald W. Dorn.
Science (2013)

894 Citations

Cytoplasmic Signaling Pathways That Regulate Cardiac Hypertrophy

Jeffery D Molkentin;Gerald W Dorn.
Annual Review of Physiology (2001)

876 Citations

Enhanced Gαq signaling: A common pathway mediates cardiac hypertrophy and apoptotic heart failure

John W. Adams;Yoshihito Sakata;Michael G. Davis;Valerie P. Sah.
Proceedings of the National Academy of Sciences of the United States of America (1998)

662 Citations

Early and Delayed Consequences of β2-Adrenergic Receptor Overexpression in Mouse Hearts Critical Role for Expression Level

Stephen B. Liggett;Nicole M. Tepe;John N. Lorenz;Amy M. Canning.
Circulation (2000)

556 Citations

Nix Is Critical to Two Distinct Phases of Mitophagy, Reactive Oxygen Species-mediated Autophagy Induction and Parkin-Ubiquitin-p62-mediated Mitochondrial Priming

Wen Xing Ding;Wen Xing Ding;Hong Min Ni;Hong Min Ni;Min Li;Yong Liao.
Journal of Biological Chemistry (2010)

522 Citations

Mitochondrial Oxidative Stress Mediates Angiotensin II–Induced Cardiac Hypertrophy and Gαq Overexpression–Induced Heart Failure

Dao Fu Dai;Simon C. Johnson;Jason J. Villarin;Michael T. Chin.
Circulation Research (2011)

448 Citations

Human phospholamban null results in lethal dilated cardiomyopathy revealing a critical difference between mouse and human

Kobra Haghighi;Fotis Kolokathis;Luke Pater;Roy A. Lynch.
Journal of Clinical Investigation (2003)

443 Citations

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