What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Mitochondrion
David A. Hood focuses on Mitochondrion, Skeletal muscle, Mitochondrial biogenesis, Cell biology and Biochemistry.
His Mitochondrion study combines topics in areas such as Denervation, Endocrinology, Internal medicine, Muscle contraction and Apoptosis.
His studies in Skeletal muscle integrate themes in fields like Autophagy, Mitophagy, Reactive oxygen species and Cytochrome c oxidase.
His Mitochondrial biogenesis research includes themes of AMP-activated protein kinase, Peroxisome, Mitochondrial Turnover and Mitochondrial DNA.
His studies deal with areas such as Receptor, Transcription factor and Organelle biogenesis as well as Cell biology.
As a part of the same scientific study, David A. Hood usually deals with the Physiology, concentrating on Sequestosome 1 and frequently concerns with MAP1LC3B, Autolysosome, Chaperone-mediated autophagy and BECN1.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Invited Review: contractile activity-induced mitochondrial biogenesis in skeletal muscle. (592 citations)
What are the main themes of his work throughout his whole career to date?
David A. Hood mostly deals with Skeletal muscle, Cell biology, Mitochondrion, Mitochondrial biogenesis and Internal medicine.
His research integrates issues of Endurance training, Muscle contraction, Myocyte and Mitochondrial Turnover, Mitophagy in his study of Skeletal muscle.
David A. Hood interconnects Autophagy, Apoptosis and Mitochondrial protein import in the investigation of issues within Cell biology.
His work in Mitochondrion addresses issues such as Molecular biology, which are connected to fields such as Messenger RNA.
His Mitochondrial biogenesis research is multidisciplinary, incorporating perspectives in Gene expression, Transcription factor, DNAJA3, Transcription and Organelle biogenesis.
His research in Internal medicine focuses on subjects like Endocrinology, which are connected to Cytochrome c oxidase.
He most often published in these fields:
- Skeletal muscle (59.44%)
- Cell biology (50.00%)
- Mitochondrion (50.35%)
What were the highlights of his more recent work (between 2012-2021)?
- Skeletal muscle (59.44%)
- Cell biology (50.00%)
- Mitochondrion (50.35%)
In recent papers he was focusing on the following fields of study:
David A. Hood mainly investigates Skeletal muscle, Cell biology, Mitochondrion, Mitochondrial biogenesis and Mitophagy.
His Skeletal muscle study combines topics from a wide range of disciplines, such as Autophagy, Endurance training and Mitochondrial Turnover.
His Cell biology study incorporates themes from Messenger RNA and Muscle contraction.
His Mitochondrion study deals with the bigger picture of Biochemistry.
His Mitochondrial biogenesis research is multidisciplinary, relying on both AMP-activated protein kinase, Transcription factor, Myogenesis, Unfolded protein response and Neuroscience.
His study in Mitophagy is interdisciplinary in nature, drawing from both Oxidative stress, Skeletal muscle mitochondria, Atrophy and Organelle biogenesis.
Between 2012 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Role of PGC-1α during acute exercise-induced autophagy and mitophagy in skeletal muscle. (131 citations)
- Sirtuin 1-mediated effects of exercise and resveratrol on mitochondrial biogenesis (122 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Mitochondrion
His scientific interests lie mostly in Cell biology, Mitochondrion, Skeletal muscle, Mitochondrial biogenesis and Mitophagy.
The study incorporates disciplines such as Oxidative stress, Endurance training and Mitochondrial DNA in addition to Cell biology.
His biological study spans a wide range of topics, including Endocrinology, Internal medicine, Denervation and Endoplasmic reticulum.
His Skeletal muscle research incorporates elements of Reactive oxygen species, Mitochondrial Turnover, Function and Muscle contraction.
His Mitochondrial biogenesis research is multidisciplinary, incorporating elements of Ca2+/calmodulin-dependent protein kinase, AMP-activated protein kinase, AMPK, Myogenesis and Transcription factor.
His Physiology research integrates issues from BECN1, Autolysosome, Chaperone-mediated autophagy, Computational biology and Programmed cell death.
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