What is he best known for?
The fields of study he is best known for:
- Enzyme
- Gene
- Internal medicine
Christer Hogstrand spends much of his time researching Metallothionein, Rainbow trout, Biochemistry, Environmental chemistry and Zinc.
His Metallothionein research is under the purview of Cadmium.
His studies deal with areas such as Ecology, Proteomics, Animal science and Gill as well as Rainbow trout.
Christer Hogstrand has researched Ecology in several fields, including Salmonidae and Environmental planning.
His research integrates issues of Toxicity, Bioassay, Bioavailability and Pollutant in his study of Environmental chemistry.
Christer Hogstrand combines subjects such as Molecular biology, Receptor, Receptor-Like Protein Tyrosine Phosphatases and Enzyme with his study of Zinc.
His most cited work include:
- The biotic ligand model : a historical overview (551 citations)
- Toward a better understanding of the bioavailability, physiology, and toxicity of silver in fish: Implications for water quality criteria (220 citations)
- Binding and detoxification of heavy metals in lower vertebrates with reference to metallothionein. (209 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Zinc, Metallothionein, Biochemistry, Food science and Environmental chemistry.
Christer Hogstrand interconnects Endocrinology, Internal medicine, Calcium, Cytosol and Cell biology in the investigation of issues within Zinc.
His Metallothionein research incorporates elements of Molecular biology, Rainbow trout, Perch and Gill.
He has included themes like Trout, Acclimatization and Animal science in his Rainbow trout study.
His Biochemistry study often links to related topics such as In vivo.
His Environmental chemistry course of study focuses on Ecology and Toxicity.
He most often published in these fields:
- Zinc (24.29%)
- Metallothionein (19.14%)
- Biochemistry (14.57%)
What were the highlights of his more recent work (between 2017-2021)?
- Risk assessment (5.14%)
- Cell biology (7.71%)
- Lipid metabolism (3.71%)
In recent papers he was focusing on the following fields of study:
His main research concerns Risk assessment, Cell biology, Lipid metabolism, Toxicity and Zinc.
His Cell biology study integrates concerns from other disciplines, such as Oxidative stress, Transcription factor, Mutant, Zebrafish and Morphant.
His Lipid metabolism research is included under the broader classification of Biochemistry.
In his research, Young age and Adult age is intimately related to Tolerable daily intake, which falls under the overarching field of Toxicity.
Zinc and Heteromer are two areas of study in which Christer Hogstrand engages in interdisciplinary work.
As a part of the same scientific study, Christer Hogstrand usually deals with the Autophagy, concentrating on Dietary lipid and frequently concerns with Internal medicine.
Between 2017 and 2021, his most popular works were:
- Risk to human health related to the presence of perfluorooctane sulfonic acid and perfluorooctanoic acid in food (121 citations)
- Guidance on harmonised methodologies for human health, animal health and ecological risk assessment of combined exposure to multiple chemicals (111 citations)
- Risk for animal and human health related to the presence of dioxins and dioxin???like PCBs in feed and food (69 citations)
In his most recent research, the most cited papers focused on:
- Enzyme
- Gene
- Internal medicine
His primary scientific interests are in Risk assessment, Human health, Cell biology, Occurrence data and Mink.
Among his Risk assessment studies, there is a synthesis of other scientific areas such as Animal science, Toxicology, Diol, Fatty acid and Benford's law.
His Animal science research includes themes of European population, Toxicokinetics, Adult age and Human study.
His Cell biology research includes elements of Oxidative stress, Lipid metabolism and Oxidative phosphorylation.
His Mink study incorporates themes from Breastfeeding, Mycotoxin, Epidemiology and Animal data.
His research links Toxicity with Moniliformin.
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