What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in Cancer research, Cell biology, Signal transduction, MAPK/ERK pathway and Autocrine signalling.
His study in Cancer research is interdisciplinary in nature, drawing from both Carcinogenesis, Cellular Dedifferentiation and Radioprotective Agent.
His Cell biology research incorporates elements of Cell, Transcription factor and Radiation induced.
His Signal transduction study integrates concerns from other disciplines, such as Cancer cell, Ceramide and Kinase.
His MAPK/ERK pathway study combines topics from a wide range of disciplines, such as Ursodeoxycholic acid, Endocrinology, Deoxycholic acid and Fas ligand.
His research integrates issues of Epidermal growth factor and Growth factor receptor in his study of Autocrine signalling.
His most cited work include:
- MAPK pathways in radiation responses (496 citations)
- Stress and radiation-induced activation of multiple intracellular signaling pathways. (426 citations)
- Radiation-induced cell signaling: inside-out and outside-in (264 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Cancer research, Cell biology, Signal transduction, Protein kinase B and Molecular biology.
His biological study spans a wide range of topics, including Autophagy, Apoptosis, Programmed cell death and Immunology.
His study connects Caspase and Cell biology.
The Signal transduction study combines topics in areas such as Cancer cell, Cell cycle and Ceramide.
His Protein kinase B research includes elements of XIAP, PI3K/AKT/mTOR pathway, Radiosensitivity and Transfection.
He has included themes like DNA glycosylase, DNA damage, DNA repair, AP site and Adenoviridae in his Molecular biology study.
He most often published in these fields:
- Cancer research (44.00%)
- Cell biology (30.00%)
- Signal transduction (24.00%)
What were the highlights of his more recent work (between 2008-2016)?
- Cancer research (44.00%)
- Apoptosis (18.00%)
- Molecular biology (24.00%)
In recent papers he was focusing on the following fields of study:
Adly Yacoub mainly investigates Cancer research, Apoptosis, Molecular biology, Signal transduction and Cell biology.
His Cancer research study integrates concerns from other disciplines, such as Autophagy and Programmed cell death.
His work in the fields of Apoptosis, such as Fas receptor, overlaps with other areas such as Cell killing.
Adly Yacoub works mostly in the field of Molecular biology, limiting it down to concerns involving Adenoviridae and, occasionally, Virus and Gene delivery.
His research investigates the connection with Signal transduction and areas like Ceramide which intersect with concerns in Sphingolipid and Toxicity.
His research on Cell biology frequently links to adjacent areas such as Cell growth.
Between 2008 and 2016, his most popular works were:
- Mutations in the Phosphatidylinositol-3-Kinase Pathway Predict for Antitumor Activity of the Inhibitor PX-866 whereas Oncogenic Ras Is a Dominant Predictor for Resistance (239 citations)
- Mitochondrial localized Stat3 promotes breast cancer growth via phosphorylation of serine 727. (112 citations)
- Mechanism of Autophagy to Apoptosis Switch Triggered in Prostate Cancer Cells by Antitumor Cytokine Melanoma Differentiation-Associated Gene 7/Interleukin-24 (102 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Cancer research, Autophagy, Signal transduction, Kinase and Sorafenib.
His work deals with themes such as Molecular biology and Ceramide, which intersect with Cancer research.
In Autophagy, Adly Yacoub works on issues like Cancer cell, which are connected to Interleukin 10, Melanoma, Immune system and Cytokine.
He has researched Signal transduction in several fields, including CHEK1, Cell cycle checkpoint, DNA damage and Poly Polymerase Inhibitor.
The concepts of his Kinase study are interwoven with issues in Carcinogenesis, Ceramide synthase and Ral Guanine Nucleotide Exchange Factor.
His study in Programmed cell death is interdisciplinary in nature, drawing from both Unfolded protein response and Cell biology.
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