2019 - Member of the National Academy of Sciences
2010 - Fellow of the American Academy of Arts and Sciences
2002 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary areas of investigation include Genetics, Leukemia, Mutation, Myeloid and Myeloid leukemia. Timothy J. Ley works mostly in the field of Genetics, limiting it down to concerns involving Computational biology and, occasionally, Medical genetics. His Leukemia research is multidisciplinary, incorporating perspectives in Cancer research, Stem cell, Promyelocytic leukemia protein and Somatic evolution in cancer.
His Cancer research study combines topics from a wide range of disciplines, such as Acute promyelocytic leukemia, Cancer, Lymphokine-activated killer cell and Neuroblastoma RAS viral oncogene homolog. His research in Myeloid intersects with topics in Haematopoiesis, Methyltransferase, Clone and DNA methylation. His Myeloid leukemia study combines topics in areas such as Enasidenib, Clonal architecture, NPM1 and Oncology.
The scientist’s investigation covers issues in Molecular biology, Gene, Leukemia, Genetics and Cancer research. Timothy J. Ley combines subjects such as Gene expression, DNA methylation, Cytotoxic T cell, Granzyme B and Globin with his study of Molecular biology. His study in Leukemia is interdisciplinary in nature, drawing from both Myeloid, Haematopoiesis, Myeloid leukemia and Acute promyelocytic leukemia.
His study looks at the intersection of Myeloid and topics like Gene expression profiling with Transcriptome. His research investigates the link between Haematopoiesis and topics such as Bone marrow that cross with problems in Oncology. Genome, Mutation, Whole genome sequencing, Human genome and Genomics are among the areas of Genetics where the researcher is concentrating his efforts.
Timothy J. Ley mainly focuses on Cancer research, Leukemia, Internal medicine, Oncology and Myeloid leukemia. The study incorporates disciplines such as DNA methylation, RNA, microRNA, Metastasis and Promyelocytic leukemia protein in addition to Cancer research. His Leukemia research incorporates themes from Haematopoiesis, Karyotype, Transplantation, Myeloid and Polymerase chain reaction.
The various areas that Timothy J. Ley examines in his Oncology study include Cancer, Somatic evolution in cancer, Azacitidine, Decitabine and Myelodysplastic syndromes. His work carried out in the field of Myeloid leukemia brings together such families of science as Clonal architecture, CD34, Primary tumor, Peripheral blood and DNA sequencing. His Exome sequencing research entails a greater understanding of Genetics.
Leukemia, Genetics, Myeloid, Myeloid leukemia and Mutation are his primary areas of study. His Leukemia research incorporates elements of Progenitor cell, Haematopoiesis and Disease. As a part of the same scientific family, he mostly works in the field of Haematopoiesis, focusing on Gene and, on occasion, Adenocarcinoma.
His Myeloid research is multidisciplinary, incorporating elements of Exome sequencing, Chemotherapy, Induction chemotherapy, Oncology and Bone marrow. His Myeloid leukemia research is multidisciplinary, relying on both Breast cancer and Clonal architecture, Somatic cell. The study incorporates disciplines such as Molecular biology, Cancer research and DNA methylation, DNA methyltransferase in addition to Mutation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The cancer genome atlas pan-cancer analysis project
John N Weinstein;John N Weinstein;Eric A. Collisson;Gordon B Mills;Kenna R Mills Shaw;Kenna R Mills Shaw.
Nature Genetics (2013)
Genomic and epigenomic landscapes of adult de novo acute myeloid leukemia
Timothy J. Ley;Christopher Miller;Li Ding;Benjamin J. Raphael.
The New England Journal of Medicine (2013)
Mutational landscape and significance across 12 major cancer types
Cyriac Kandoth;Michael D. McLellan;Fabio Vandin;Kai Ye.
Recurring mutations found by sequencing an acute myeloid leukemia genome.
Elaine R. Mardis;Li Ding;David J. Dooling;David E. Larson.
The New England Journal of Medicine (2009)
Clonal evolution in relapsed acute myeloid leukaemia revealed by whole-genome sequencing
Li Ding;Timothy J. Ley;David E. Larson;Christopher A. Miller.
DNMT3A Mutations in Acute Myeloid Leukemia
Timothy J. Ley;Li Ding;Matthew J. Walter;Michael D. McLellan.
The New England Journal of Medicine (2010)
International network of cancer genome projects
Thomas J. Hudson;Thomas J. Hudson;Warwick Anderson;Axel Aretz;Anna D. Barker.
The genetic basis of early T-cell precursor acute lymphoblastic leukaemia
Jinghui Zhang;Li Ding;Linda Holmfeldt;Gang Wu.
DNA sequencing of a cytogenetically normal acute myeloid leukaemia genome
Timothy J. Ley;Elaine R. Mardis;Li Ding;Bob Fulton.
The Origin and Evolution of Mutations in Acute Myeloid Leukemia
John S. Welch;Timothy J. Ley;Daniel C. Link;Christopher A. Miller.
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