What is he best known for?
The fields of study he is best known for:
- Neuron
- Genetics
- Neurotransmitter
Miguel Díaz-Hernández mostly deals with Cell biology, Receptor, Biochemistry, Extracellular and Neurotransmitter.
Miguel Díaz-Hernández combines subjects such as Hippocampal formation and Huntington's disease with his study of Cell biology.
His research integrates issues of Endocrinology and Calcium in his study of Receptor.
His Biochemistry research integrates issues from Neurodegeneration, Amyloid precursor protein secretase, Amyloid precursor protein and In vivo.
The Extracellular study combines topics in areas such as Calcium in biology, Tau protein and Muscarinic acetylcholine receptor, Muscarinic acetylcholine receptor M3.
The various areas that Miguel Díaz-Hernández examines in his Neurotransmitter study include Kainic acid, Purinergic receptor, Immunology, Cytokine and Apoptosis.
His most cited work include:
- Neuronal induction of the immunoproteasome in Huntington's disease (251 citations)
- Extracellular tau is toxic to neuronal cells. (183 citations)
- Altered P2X7-receptor level and function in mouse models of Huntington’s disease and therapeutic efficacy of antagonist administration (177 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Cell biology, Receptor, Biochemistry, Neuroscience and Purinergic receptor.
His Cell biology research incorporates elements of Huntingtin, Huntington's disease, Genetically modified mouse and In vivo.
His In vivo research includes themes of BACE1-AS, P3 peptide, Biochemistry of Alzheimer's disease, Amyloid precursor protein and GSK-3.
His Receptor study incorporates themes from Anticonvulsant, Endocrinology and Neuroprotection.
Miguel Díaz-Hernández has included themes like Biophysics and Calcium in his Biochemistry study.
His work carried out in the field of Purinergic receptor brings together such families of science as Kinase and Purinergic signalling.
He most often published in these fields:
- Cell biology (63.64%)
- Receptor (43.43%)
- Biochemistry (34.34%)
What were the highlights of his more recent work (between 2015-2021)?
- Neuroscience (33.33%)
- Neuroinflammation (17.17%)
- Purinergic signalling (21.21%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Neuroscience, Neuroinflammation, Purinergic signalling, Receptor and Cell biology.
His Neuroscience study combines topics in areas such as Purinergic receptor and P2x7 receptor.
Calcification and Mesenchymal stem cell is closely connected to Endocrinology in his research, which is encompassed under the umbrella topic of Purinergic signalling.
Miguel Díaz-Hernández interconnects Proteostasis and MAPK/ERK pathway in the investigation of issues within Receptor.
His Cell biology study combines topics from a wide range of disciplines, such as Alzheimer's disease, Senile plaques, Neurotoxicity and Microgliosis.
His Hippocampal formation research is multidisciplinary, relying on both Kainic acid and Status epilepticus.
Between 2015 and 2021, his most popular works were:
- Transient P2X7 Receptor Antagonism Produces Lasting Reductions in Spontaneous Seizures and Gliosis in Experimental Temporal Lobe Epilepsy (76 citations)
- Neuronal P2X7 Receptor: Involvement in Neuronal Physiology and Pathology. (62 citations)
- Neuronal P2X7 Receptor: Involvement in Neuronal Physiology and Pathology. (62 citations)
In his most recent research, the most cited papers focused on:
- Genetics
- Neuron
- Neurotransmitter
The scientist’s investigation covers issues in Neuroscience, Receptor, Internal medicine, Endocrinology and Pathology.
His studies deal with areas such as Extracellular, Tau protein, Intracellular and Phosphorylation as well as Neuroscience.
The concepts of his Receptor study are interwoven with issues in Hippocampal formation, Gliosis and Anticonvulsant, Epilepsy.
When carried out as part of a general Internal medicine research project, his work on Purinergic signalling is frequently linked to work in Metabolic bone disease, Hypophosphatasia and ALPL, therefore connecting diverse disciplines of study.
His Endocrinology research incorporates themes from Calcification and Mesenchymal stem cell, Cell biology.
His research in Pathology intersects with topics in Cell survival, P2x7 receptor, Physiology and Neurotransmitter.
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