What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in Cell biology, Molecular biology, Biochemistry, Gene expression and Transfection.
Michael R. H. White interconnects NFKB1, Transcription factor, Apoptosis, Programmed cell death and Transcription in the investigation of issues within Cell biology.
In his research, Transcription Factor RelA is intimately related to Cell nucleus, which falls under the overarching field of Transcription factor.
His Molecular biology research incorporates themes from Reporter gene, Messenger RNA, Bovine serum albumin, Nuclear protein and Oligonucleotide.
Gene expression is often connected to Aspergillus nidulans in his work.
His Transfection research includes elements of Regulation of gene expression, Insert and Gene silencing.
His most cited work include:
- TRY - a global database of plant traits (1611 citations)
- Oscillations in NF-κB Signaling Control the Dynamics of Gene Expression (993 citations)
- Pulsatile Stimulation Determines Timing and Specificity of NF-κB-Dependent Transcription (438 citations)
What are the main themes of his work throughout his whole career to date?
Michael R. H. White mainly investigates Cell biology, Molecular biology, Gene expression, Internal medicine and Endocrinology.
As a part of the same scientific family, Michael R. H. White mostly works in the field of Cell biology, focusing on Cell and, on occasion, Signalling.
Michael R. H. White has included themes like Luciferase, Cell culture, Transfection, Mitosis and Reporter gene in his Molecular biology study.
His study ties his expertise on Regulation of gene expression together with the subject of Gene expression.
His Endocrinology study focuses on Prolactin in particular.
His Signal transduction course of study focuses on Tumor necrosis factor alpha and Cytokine and NFKB1.
He most often published in these fields:
- Cell biology (29.39%)
- Molecular biology (17.18%)
- Gene expression (11.83%)
What were the highlights of his more recent work (between 2015-2021)?
- Cell biology (29.39%)
- Transcription (7.63%)
- Tumor necrosis factor alpha (4.58%)
In recent papers he was focusing on the following fields of study:
His main research concerns Cell biology, Transcription, Tumor necrosis factor alpha, Signal transduction and Regulation of gene expression.
His biological study spans a wide range of topics, including NFKB1, Transcription factor, Apoptosis and Cell growth.
His work carried out in the field of Transcription brings together such families of science as Luciferase and Endocrinology.
His work in Tumor necrosis factor alpha tackles topics such as Cytokine which are related to areas like Cancer research.
His studies deal with areas such as Cell, Single-cell analysis and Secretion as well as Signal transduction.
His study focuses on the intersection of Regulation of gene expression and fields such as Molecular biology with connections in the field of Cell culture, DNA damage and Real-time polymerase chain reaction.
Between 2015 and 2021, his most popular works were:
- Inflammasome-dependent IL-1β release depends upon membrane permeabilisation. (121 citations)
- The intervertebral disc contains intrinsic circadian clocks that are regulated by age and cytokines and linked to degeneration (55 citations)
- Signal transduction controls heterogeneous NF-κB dynamics and target gene expression through cytokine-specific refractory states (51 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Cell biology, Regulation of gene expression, Signal transduction, Genetics and NFKB1.
His Cell biology research is multidisciplinary, incorporating elements of Cell growth, Bioinformatics, Period Circadian Proteins, CLOCK Proteins and PER2.
His work deals with themes such as Molecular biology, Cell, Transcription and Kinase, which intersect with Regulation of gene expression.
His Signal transduction research focuses on Tumor necrosis factor alpha and how it relates to Cytokine.
His study explores the link between NFKB1 and topics such as NF-κB that cross with problems in Tumor Necrosis Factor alpha-Induced Protein 3 and Gene silencing.
His Gene expression study introduces a deeper knowledge of Gene.
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