What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Mitochondrion
Michael R. Duchen mainly focuses on Mitochondrion, Cell biology, Biochemistry, Mitochondrial permeability transition pore and Programmed cell death.
His Mitochondrion study combines topics in areas such as Oxidative stress, Cell, Apoptosis, Oxidative phosphorylation and Calcium signaling.
The study of Cell biology is intertwined with the study of Reperfusion injury in a number of ways.
The concepts of his Biochemistry study are interwoven with issues in Biophysics, Depolarization and Neuroprotection.
His studies in Mitochondrial permeability transition pore integrate themes in fields like Anesthesia, MPTP, Viability assay, Mitochondrial apoptosis-induced channel and Diazoxide.
His studies deal with areas such as Cell physiology, Computational biology, Autolysosome and Chaperone-mediated autophagy as well as Programmed cell death.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS. (1106 citations)
- Mitochondria and calcium: from cell signalling to cell death. (885 citations)
What are the main themes of his work throughout his whole career to date?
Michael R. Duchen focuses on Mitochondrion, Cell biology, Biochemistry, Biophysics and Internal medicine.
His Mitochondrion study integrates concerns from other disciplines, such as Oxidative stress, Calcium, Calcium signaling, Membrane potential and Mitochondrial permeability transition pore.
His Cell biology research incorporates themes from Apoptosis, Programmed cell death and Neurodegeneration.
His study looks at the relationship between Biochemistry and fields such as Neurotoxicity, as well as how they intersect with chemical problems.
He works mostly in the field of Biophysics, limiting it down to topics relating to NAD+ kinase and, in certain cases, Fluorescence-lifetime imaging microscopy and Cofactor, as a part of the same area of interest.
His Internal medicine study combines topics from a wide range of disciplines, such as Endocrinology and Pathology.
He most often published in these fields:
- Mitochondrion (50.34%)
- Cell biology (46.62%)
- Biochemistry (23.99%)
What were the highlights of his more recent work (between 2014-2021)?
- Mitochondrion (50.34%)
- Cell biology (46.62%)
- Internal medicine (15.54%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Mitochondrion, Cell biology, Internal medicine, Endocrinology and Neuroscience.
The various areas that Michael R. Duchen examines in his Mitochondrion study include Calcium, Calcium signaling, Bioinformatics and Mitochondrial permeability transition pore.
His Mitochondrial permeability transition pore study is within the categories of Programmed cell death and Biochemistry.
His Biochemistry research is multidisciplinary, incorporating perspectives in Autofluorescence and Fluorescence-lifetime imaging microscopy.
As part of the same scientific family, Michael R. Duchen usually focuses on Cell biology, concentrating on Neurodegeneration and intersecting with Mitophagy, Autophagy and Crosstalk.
His work deals with themes such as Oxidative phosphorylation, Downregulation and upregulation, Sepsis and Pathology, which intersect with Endocrinology.
Between 2014 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Reversal of Mitochondrial Transhydrogenase Causes Oxidative Stress in Heart Failure (166 citations)
- Promoting the clearance of neurotoxic proteins in neurodegenerative disorders of ageing (152 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
His primary areas of study are Mitochondrion, Cell biology, Neuroscience, Biochemistry and Disease.
His Mitochondrion research is multidisciplinary, incorporating elements of Bioinformatics, Neurodegeneration, Pathology, Mitochondrial permeability transition pore and Mitophagy.
His Mitochondrial permeability transition pore research is classified as research in Programmed cell death.
He interconnects BECN1, Autophagosome, Autolysosome, Computational biology and Physiology in the investigation of issues within Programmed cell death.
His Cell biology research integrates issues from mitochondrial fusion, Calcium and Induced pluripotent stem cell.
His research investigates the link between Calcium and topics such as SERCA that cross with problems in Endocrinology and Internal medicine.
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