D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 41 Citations 9,542 60 World Ranking 16239 National Ranking 6698

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Genetics

His main research concerns Cell biology, Synaptic vesicle, Mutant, RNA polymerase and Exocytosis. His Cell biology research includes themes of Synaptobrevin and Synaptotagmin 1. Michael L. Nonet has included themes like Phenotype, Caenorhabditis elegans and Syntaxin in his Mutant study.

His biological study spans a wide range of topics, including Mutation, Conserved sequence, Neuroscience, Synaptogenesis and Gene targeting. His RNA polymerase research focuses on subjects like Molecular biology, which are linked to Protein subunit, RNA polymerase I and RNA-dependent RNA polymerase. His work in Exocytosis tackles topics such as Synaptotagmin I which are related to areas like Kiss-and-run fusion.

His most cited work include:

  • Loss of the putative RNA-directed RNA polymerase RRF-3 makes C. elegans hypersensitive to RNAi. (512 citations)
  • Synaptic function is impaired but not eliminated in C. elegans mutants lacking synaptotagmin (493 citations)
  • Caenorhabditis elegans rab-3 Mutant Synapses Exhibit Impaired Function and Are Partially Depleted of Vesicles (306 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Cell biology, Caenorhabditis elegans, Mutant, Synaptic vesicle and Genetics. The various areas that he examines in his Cell biology study include Biochemistry, Synaptobrevin, Active zone, Neuromuscular junction and Syntaxin. His Caenorhabditis elegans research incorporates elements of Mutation, Genome, Green fluorescent protein, Synaptogenesis and Synapse.

His Mutant research incorporates themes from Amino acid, Phenotype, Molecular biology, Intracellular and Effector. His Synaptic vesicle research is multidisciplinary, incorporating perspectives in Transport protein, Exocytosis and Axoplasmic transport. In the field of Genetics, his study on Gene, RNA interference and Genome size overlaps with subjects such as RNA polymerase II and Cre recombinase.

He most often published in these fields:

  • Cell biology (72.46%)
  • Caenorhabditis elegans (49.28%)
  • Mutant (31.88%)

What were the highlights of his more recent work (between 2012-2021)?

  • Cell biology (72.46%)
  • Caenorhabditis elegans (49.28%)
  • Axoplasmic transport (5.80%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Cell biology, Caenorhabditis elegans, Axoplasmic transport, Genetics and Microtubule minus-end binding. His study in Cell biology is interdisciplinary in nature, drawing from both Transgene and Compartment. Within one scientific family, Michael L. Nonet focuses on topics pertaining to Focal adhesion under Caenorhabditis elegans, and may sometimes address concerns connected to Mutant.

His Axoplasmic transport study incorporates themes from Synaptic vesicle, Neuron, Endosome and Actin. His Synaptic vesicle research is multidisciplinary, relying on both Mutation, Protein domain and KIF1A. His RNA, Cleavage stimulation factor, DNA and Cleavage factor study, which is part of a larger body of work in Genetics, is frequently linked to Guide RNA, bridging the gap between disciplines.

Between 2012 and 2021, his most popular works were:

  • Landscape of target:guide homology effects on Cas9-mediated cleavage (59 citations)
  • The Caenorhabditis elegans microtubule minus-end binding homolog PTRN-1 stabilizes synapses and neurites (56 citations)
  • The vesicle protein SAM-4 regulates the processivity of synaptic vesicle transport. (28 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Genetics

Michael L. Nonet mainly focuses on Cell biology, Genetics, Microtubule, Cell division and Microtubule-associated protein. His primary area of study in Cell biology is in the field of Motor protein. The Motor protein study combines topics in areas such as Mutation, Protein domain, Axoplasmic transport and Synaptic vesicle transport.

His Protein domain study frequently draws connections to other fields, such as Synaptic vesicle. Michael L. Nonet has researched Microtubule in several fields, including Caenorhabditis elegans and Bioinformatics. His biological study spans a wide range of topics, including Cleavage stimulation factor, Cleavage factor, RNA and DNA.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Synaptic function is impaired but not eliminated in C. elegans mutants lacking synaptotagmin

Michael L. Nonet;Kiely Grundahl;Barbara J. Meyer;James B. Rand.
Cell (1993)

764 Citations

Loss of the putative RNA-directed RNA polymerase RRF-3 makes C. elegans hypersensitive to RNAi.

Femke Simmer;Marcel Tijsterman;Susan Parrish;Susan Parrish;Sandhya P Koushika.
Current Biology (2002)

682 Citations

Eucaryotic RNA polymerase conditional mutant that rapidly ceases mRNA synthesis.

M Nonet;C Scafe;J Sexton;R Young.
Molecular and Cellular Biology (1987)

502 Citations

Prokaryotic and eukaryotic RNA polymerases have homologous core subunits

Doug Sweetser;Michael Nonet;Richard A. Young.
Proceedings of the National Academy of Sciences of the United States of America (1987)

427 Citations

Functional redundancy and structural polymorphism in the large subunit of RNA polymerase II

Michael Nonet;Doug Sweetser;Richard A. Young.
Cell (1987)

390 Citations

Caenorhabditis elegans rab-3 Mutant Synapses Exhibit Impaired Function and Are Partially Depleted of Vesicles

Michael L. Nonet;Jane E. Staunton;Michael P. Kilgard;Tim Fergestad.
The Journal of Neuroscience (1997)

370 Citations

A post-docking role for active zone protein Rim.

Sandhya P. Koushika;Janet E. Richmond;Gayla Hadwiger;Robby M. Weimer.
Nature Neuroscience (2001)

357 Citations

Defective recycling of synaptic vesicles in synaptotagmin mutants of Caenorhabditis elegans

Erik M. Jorgensen;Erik M. Jorgensen;Erika Hartwieg;Kim Schuske;Michael L. Nonet.
Nature (1995)

354 Citations

Intragenic and Extragenic Suppressors of Mutations in the Heptapeptide Repeat Domain of Saccharomyces Cerevisiae RNA Polymerase II

M L Nonet;R A Young.
Genetics (1989)

344 Citations

Visualization of synaptic specializations in live C. elegans with synaptic vesicle protein-GFP fusions.

Michael L. Nonet.
Journal of Neuroscience Methods (1999)

342 Citations

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