What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in Programmed cell death, Cell biology, Biochemistry, GPX4 and Glutathione peroxidase.
Programmed cell death is a primary field of his research addressed under Apoptosis.
Marcus Conrad works mostly in the field of Cell biology, limiting it down to concerns involving Cell culture and, occasionally, Genetic screen and Cell.
His GPX4 study combines topics from a wide range of disciplines, such as Endocrinology, Coenzyme Q10, Internal medicine, Lipid peroxidation and Neurodegeneration.
He has included themes like Sperm, Epididymis, Peroxidase and Chromatin in his Glutathione peroxidase study.
His work deals with themes such as Molecular biology and Mitochondrion, which intersect with Phospholipid-hydroperoxide glutathione peroxidase.
His most cited work include:
- Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. (1421 citations)
- Ferroptosis: A Regulated Cell Death Nexus Linking Metabolism, Redox Biology, and Disease (1069 citations)
- Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice (748 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Cell biology, GPX4, Programmed cell death, Biochemistry and Glutathione.
The Cell biology study combines topics in areas such as Oxidative stress, Thioredoxin and Cell growth.
The study incorporates disciplines such as Phospholipid-hydroperoxide glutathione peroxidase, Selenoprotein, Internal medicine and Endocrinology in addition to GPX4.
His Phospholipid-hydroperoxide glutathione peroxidase research includes themes of Molecular biology and GPX5.
His Programmed cell death research includes elements of Lipid peroxidation, Cancer cell and Neurodegeneration.
His study in Ferroptosis is interdisciplinary in nature, drawing from both Disease, Neuroscience and Bioinformatics.
He most often published in these fields:
- Cell biology (44.52%)
- GPX4 (35.48%)
- Programmed cell death (32.26%)
What were the highlights of his more recent work (between 2018-2021)?
- Cell biology (44.52%)
- Programmed cell death (32.26%)
- Ferroptosis (17.42%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Cell biology, Programmed cell death, Ferroptosis, GPX4 and Lipid peroxidation.
His research in Cell biology tackles topics such as Glutathione which are related to areas like Caenorhabditis elegans.
His Programmed cell death study incorporates themes from Cancer, Cancer research and Neurodegeneration.
His Ferroptosis research is multidisciplinary, relying on both Niche, Ecology, Disease and Bioinformatics.
GPX4 is a subfield of Biochemistry that Marcus Conrad investigates.
His Lipid peroxidation study integrates concerns from other disciplines, such as Suppressor, Ischemia, Lipid metabolism, NAD+ kinase and Metabolism.
Between 2018 and 2021, his most popular works were:
- FSP1 is a glutathione-independent ferroptosis suppressor (288 citations)
- Ferroptosis at the crossroads of cancer-acquired drug resistance and immune evasion. (150 citations)
- Role of GPX4 in ferroptosis and its pharmacological implication (103 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in GPX4, Lipid peroxidation, Programmed cell death, Cell biology and Ferroptosis.
The concepts of his GPX4 study are interwoven with issues in Phospholipid-hydroperoxide glutathione peroxidase, Lipid signaling, Rational design, Downregulation and upregulation and Allosteric regulation.
His biological study spans a wide range of topics, including Suppressor, MDMX, Lipid metabolism and Endogeny.
His research in Programmed cell death intersects with topics in Cancer and Cancer research.
His Cell biology research incorporates themes from Glutathione, Mdm2, Mutant and NAD+ kinase.
His Ferroptosis research is multidisciplinary, incorporating perspectives in Cancer cell, Therapeutic targeting and Neuroscience.
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