1998 - Fellow of the American Statistical Association (ASA)
Neuroscience, Dopamine, Genetics, Genomic imprinting and Neurochemical are his primary areas of study. His study in Neuroscience is interdisciplinary in nature, drawing from both Prepulse inhibition and Genotype. His Dopamine study is concerned with Internal medicine in general.
Lawrence Stephen Wilkinson combines subjects such as Angelman syndrome, Brain function, Function, Brain development and Ploidy with his study of Genomic imprinting. Lawrence Stephen Wilkinson has researched Neurochemical in several fields, including Striatum, Prefrontal cortex and Classical conditioning. His Gene research includes themes of Cognition and Turner syndrome.
His primary areas of study are Neuroscience, Genomic imprinting, Genetics, Internal medicine and Endocrinology. His is doing research in Dopamine, Neurochemical, Prefrontal cortex, Brain function and Stimulus, both of which are found in Neuroscience. His research in Neurochemical intersects with topics in Basal ganglia, Amphetamine and Reinforcement.
His Genomic imprinting study combines topics in areas such as Developmental psychology, Cognition, Epigenetics and Imprinting. The Gene, Allele and Phenotype research he does as part of his general Genetics study is frequently linked to other disciplines of science, such as GRB10 and Mechanism, therefore creating a link between diverse domains of science. His Endocrinology research is multidisciplinary, incorporating elements of Agonist, Monoamine neurotransmitter, Serotonin and Antagonist.
The scientist’s investigation covers issues in Neuroscience, Genetics, Psychiatry, Genomic imprinting and Schizophrenia. His Associative learning study, which is part of a larger body of work in Neuroscience, is frequently linked to Single process, bridging the gap between disciplines. His study in the field of Copy-number variation is also linked to topics like GRB10.
His Psychiatry research incorporates elements of Neurogenesis, Genetic variation, Pharmacology toxicology and Voltage-dependent calcium channel. His Genomic imprinting research integrates issues from Nucleus accumbens, Immediate early gene, Neurodevelopmental disorder, Internal medicine and Epigenetics. His Schizophrenia research includes elements of Synaptic plasticity, Fragile X syndrome, Hippocampal formation, Hippocampus and FMR1.
Lawrence Stephen Wilkinson mainly investigates Psychiatry, Genetics, Impulsivity, Genomic imprinting and Schizophrenia. His study on Psychiatry also encompasses disciplines like
His Copy-number variation research is multidisciplinary, incorporating perspectives in Associative learning, Neuroscience, Human genetics and Epigenetics of schizophrenia. His Impulsivity research is multidisciplinary, relying on both Endocrinology, Orbitofrontal cortex, Agonist, Nucleus accumbens and Immediate early gene. The various areas that Lawrence Stephen Wilkinson examines in his Genomic imprinting study include Neurodevelopmental disorder, Internal medicine, Imprinting and Germline.
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Isolation rearing of rats produces a deficit in prepulse inhibition of acoustic startle similar to that in schizophrenia.
Mark A. Geyer;Lawrence Stephen Wilkinson;Trevor Humby;Trevor W. Robbins.
Biological Psychiatry (1993)
6-Hydroxydopamine lesions of the prefrontal cortex in monkeys enhance performance on an analog of the Wisconsin Card Sort Test: possible interactions with subcortical dopamine.
A. C. Roberts;M. A. De Salvia;Lawrence Stephen Wilkinson;P. Collins.
The Journal of Neuroscience (1994)
Genomic imprinting effects on brain development and function.
Lawrence Stephen Wilkinson;William Davies;Anthony Roger Isles.
Nature Reviews Neuroscience (2007)
Pathological changes in dopaminergic nerve cells of the substantia nigra and olfactory bulb in mice transgenic for truncated human alpha-synuclein(1-120): implications for Lewy body disorders.
George K. Tofaris;Pablo Gracia Reitbock;Trevor Humby;Sarah L. Lambourne.
The Journal of Neuroscience (2006)
Behavioural, histochemical and biochemical consequences of selective immunolesions in discrete regions of the basal forebrain cholinergic system
E. M. Torres;T. A. Perry;A. Blokland;Lawrence Stephen Wilkinson.
Neuroscience (1994)
Isolation rearing in rats: Pre- and postsynaptic changes in striatal dopaminergic systems
F. S. Hall;Lawrence Stephen Wilkinson;Trevor Humby;W. Inglis.
Pharmacology, Biochemistry and Behavior (1998)
Social Isolation in the Rat Produces Developmentally Specific Deficits in Prepulse Inhibition of the Acoustic Startle Response Without Disrupting Latent Inhibition
Lawrence Stephen Wilkinson;Simon S. Killcross;Trevor Humby;Frank S. Hall.
Neuropsychopharmacology (1994)
Dissociable effects on spatial maze and passive avoidance acquisition and retention following AMPA- and ibotenic acid-induced excitotoxic lesions of the basal forebrain in rats: differential dependence on cholinergic neuronal loss.
K. J. Page;B. J. Everitt;T. W. Robbins;H. M. Marston.
Neuroscience (1991)
Repeated maternal separation of preweanling rats attenuates behavioral responses to primary and conditioned incentives in adulthood
Keith Matthews;Lawrence Stephen Wilkinson;Trevor W. Robbins.
Physiology & Behavior (1996)
Maternal deprivation of neonatal rats produces enduring changes in dopamine function
F. S. Hall;F. S. Hall;Lawrence Stephen Wilkinson;Trevor Humby;T. W. Robbins.
Synapse (1999)
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