1994 - Prize of the Foundation for Polish Science - Nagroda Fundacji na rzecz Nauki Polskiej for his studies into new immunological mechanisms in autoimmune diseases of the nervous system, Multiple Sclerosis in particular
Krzysztof Selmaj brings together Central nervous system and Experimental autoimmune encephalomyelitis to produce work in his papers. He merges many fields, such as Experimental autoimmune encephalomyelitis and Encephalomyelitis, in his writings. His work blends Encephalomyelitis and Multiple sclerosis studies together. He undertakes interdisciplinary study in the fields of Multiple sclerosis and Lesion through his works. He integrates Lesion with Pathology in his research. While working on this project, he studies both Pathology and Internal medicine. As part of his studies on Internal medicine, Krzysztof Selmaj often connects relevant subjects like Microglia. He combines Microglia and Central nervous system in his research. His Immunology study frequently links to related topics such as Spleen.
As part of his inquiry into Fingolimod and Relapsing remitting, Krzysztof Selmaj is doing Multiple sclerosis research. As part of his Peripheral blood mononuclear cell, Cytotoxicity and Cytotoxic T cell and In vitro studies, he is studying In vitro. His work on Spleen expands to the thematically related Immunology. Krzysztof Selmaj frequently studies issues relating to Adverse effect and Internal medicine. His study deals with a combination of Pathology and Gastroenterology. Krzysztof Selmaj conducts interdisciplinary study in the fields of Gastroenterology and Pathology through his works. He undertakes interdisciplinary study in the fields of Tumor necrosis factor alpha and Lymphotoxin through his research. His study brings together the fields of Stimulation and Endocrinology. His Endocrinology research extends to the thematically linked field of Stimulation.
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A Placebo-Controlled Trial of Oral Fingolimod in Relapsing Multiple Sclerosis
Ludwig Kappos;Ernst Wilhelm Radue;Paul O'Connor;Chris Polman.
The New England Journal of Medicine (2010)
Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
Stephen Sawcer;Garrett Hellenthal;Matti Pirinen;Chris C. A. Spencer.
Nature (2011)
Tumor necrosis factor mediates myelin and oligodendrocyte damage in vitro
Krzysztof W. Selmaj;Cedric S. Raine.
Annals of Neurology (1988)
Placebo-Controlled Phase 3 Study of Oral BG-12 for Relapsing Multiple Sclerosis
Ralf Gold;Ludwig Kappos;Douglas L Arnold;Amit Bar-Or.
The New England Journal of Medicine (2012)
Ocrelizumab versus Placebo in Primary Progressive Multiple Sclerosis
Xavier Montalban;Stephen L. Hauser;Ludwig Kappos;Douglas L. Arnold.
The New England Journal of Medicine (2017)
Ocrelizumab versus Interferon Beta-1a in Relapsing Multiple Sclerosis.
Stephen L. Hauser;Amit Bar-Or;Giancarlo Comi;Gavin Giovannoni.
The New England Journal of Medicine (2017)
Alemtuzumab for patients with relapsing multiple sclerosis after disease-modifying therapy: A randomised controlled phase 3 trial
Alasdair J. Coles;Cary L. Twyman;Douglas L. Arnold;Jeffrey A. Cohen.
The Lancet (2012)
Alemtuzumab versus interferon beta 1a as first-line treatment for patients with relapsing-remitting multiple sclerosis: a randomised controlled phase 3 trial.
Jeffrey A. Cohen;Alasdair J. Coles;Douglas L. Arnold;Christian Confavreux.
The Lancet (2012)
Identification of lymphotoxin and tumor necrosis factor in multiple sclerosis lesions.
K Selmaj;C S Raine;B Cannella;C F Brosnan.
Journal of Clinical Investigation (1991)
Alemtuzumab vs. interferon beta-1a in early multiple sclerosis.
Camms Trial Investigators;Alasdair J Coles;D Alastair S Compston;Krzysztof W Selmaj.
The New England Journal of Medicine (2008)
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