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José L. Venero

José L. Venero

D-Index & Metrics

Neuroscience

D-Index
58
Citations
10994
World Ranking
4196
National Ranking
58

Overview

José L. Venero is affiliated with the University of Seville in Spain. Their research focuses primarily on immunology, microbiology, and neuroscience, with significant contributions to related fields such as medicine, neurology, molecular biology, cellular and molecular neuroscience, and pharmacology.

The scientist's work covers key topics including:

  • Galectins and Cancer Biology
  • Neuroinflammation and Neurodegeneration Mechanisms
  • Inflammation biomarkers and pathways
  • Macrophage Migration Inhibitory Factor
  • Parkinson's Disease Mechanisms and Treatments
  • Signaling Pathways in Disease
  • Immune cells in cancer

José L. Venero has published extensively in several venues, with frequent publications appearing in:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • IBRO Neuroscience Reports
  • Acta Neuropathologica
  • International Journal of Molecular Sciences
  • Cell Death and Disease

Their recent papers include:

  • "Microglia: Agents of the CNS Pro-Inflammatory Response" (2020), published in Cells
  • "APOE in the bullseye of neurodegenerative diseases: impact of the APOE genotype in Alzheimer's disease pathology and brain diseases" (2022), published in Molecular Neurodegeneration
  • "Galectin-3, a rising star in modulating microglia activation under conditions of neurodegeneration" (2022), published in Cell Death and Disease
  • "Hyperinflammation and Fibrosis in Severe COVID-19 Patients: Galectin-3, a Target Molecule to Consider" (2020), published in Frontiers in Immunology
  • "ARG1-expressing microglia show a distinct molecular signature and modulate postnatal development and function of the mouse brain" (2023), published in Nature Neuroscience

Frequently collaborating researchers include:

  • Rocío Ruiz
  • Juan García-Revilla
  • Tomas Deierborg
  • Antonio Boza-Serrano
  • Ana M. Espinosa-Oliva

Best Publications

  • Caspase signalling controls microglia activation and neurotoxicity

    Miguel A. Burguillos;Tomas Deierborg;Edel Kavanagh;Annette Persson

  • Microglial subtypes: diversity within the microglial community.

    Vassilis Stratoulias;Jose Luis Venero;Jose Luis Venero;Marie-Ève Tremblay;Bertrand Joseph

  • The Single Intranigral Injection of LPS as a New Model for Studying the Selective Effects of Inflammatory Reactions on Dopaminergic System

    A.J. Herrera;A. Castaño;J.L. Venero;J. Cano

  • Microglia-Secreted Galectin-3 Acts as a Toll-like Receptor 4 Ligand and Contributes to Microglial Activation

    Miguel Angel Burguillos;Miguel Angel Burguillos;Martina Svensson;Tim Schulte;Antonio Boza-Serrano

  • Galectin-3, a novel endogenous TREM2 ligand, detrimentally regulates inflammatory response in Alzheimer’s disease

    Antonio Boza-Serrano;Rocío Ruiz;Raquel Sánchez-Varo;Juan García-Revilla

  • Aquaporins in the central nervous system.

    José L. Venero;Marı́a L. Vizuete;Alberto Machado;Josefina Cano

  • Microglia: Agents of the CNS Pro-Inflammatory Response

    José A. Rodríguez-Gómez;Edel Kavanagh;Pinelopi Engskog-Vlachos;Mikael K. R. Engskog

  • Differential upregulation of aquaporin-4 mRNA expression in reactive astrocytes after brain injury: potential role in brain edema.

    M L Vizuete;J L Venero;C Vargas;A A Ilundáin

  • APOE in the bullseye of neurodegenerative diseases: impact of the APOE genotype in Alzheimer’s disease pathology and brain diseases

    Unknown

  • Minocycline reduces the lipopolysaccharide-induced inflammatory reaction, peroxynitrite-mediated nitration of proteins, disruption of the blood-brain barrier, and damage in the nigral dopaminergic system.

    Mayka Tomás-Camardiel;Inmaculada Rite;Antonio J Herrera;Rocio M de Pablos

  • Stress Increases Vulnerability to Inflammation in the Rat Prefrontal Cortex

    R. M. de Pablos;R. F. Villarán;S. Argüelles;A. J. Herrera

  • Ulcerative colitis exacerbates lipopolysaccharide-induced damage to the nigral dopaminergic system: potential risk factor in Parkinson`s disease.

    Ruth F. Villarán;Ana M. Espinosa-Oliva;Manuel Sarmiento;Rocío M. De Pablos

  • Chronic stress enhances microglia activation and exacerbates death of nigral dopaminergic neurons under conditions of inflammation

    Rocío M de Pablos;Antonio J Herrera;Ana M Espinosa-Oliva;Manuel Sarmiento;Manuel Sarmiento

  • Chronic stress as a risk factor for Alzheimer’s disease

    Alberto Machado;Antonio J Herrera;Rocío M de Pablos;Ana María Espinosa-Oliva

  • Detailed localization of aquaporin-4 messenger RNA in the CNS: preferential expression in periventricular organs.

    J.L. Venero;M.L. Vizuete;A.A. Ilundáin;A. Machado

  • Stress is critical for LPS-induced activation of microglia and damage in the rat hippocampus

    A.M. Espinosa-Oliva;R.M. de Pablos;R.F. Villarán;S. Argüelles

  • Blood–brain barrier disruption induces in vivo degeneration of nigral dopaminergic neurons

    Inmaculada Rite;Alberto Machado;Josefina Cano;José L. Venero

  • Galectin-3, a rising star in modulating microglia activation under conditions of neurodegeneration

    Unknown

  • Blood-brain barrier disruption highly induces aquaporin-4 mRNA and protein in perivascular and parenchymal astrocytes: protective effect by estradiol treatment in ovariectomized animals.

    M. Tomás-Camardiel;J.L. Venero;A.J. Herrera;R.M. De Pablos

  • Galectin-3 released in response to traumatic brain injury acts as an alarmin orchestrating brain immune response and promoting neurodegeneration

    Ping Kei Yip;Alejandro Carrillo-Jimenez;Paul King;Anna Vilalta

  • Inflammatory process as a determinant factor for the degeneration of substantia nigra dopaminergic neurons.

    A. J. Herrera;M. Tomás-Camardiel;J. L. Venero;J. Cano

  • Expression of neurotrophin and trk receptor genes in adult rats with fimbria transections: Effect of intraventricular nerve growth factor and brain-derived neurotrophic factor administration

    J.L. Venero;B. Knüsel;K.D. Beck;F. Hefti

Frequent Co-Authors

Josefina Cano
Josefina Cano University of Seville
Ping K. Yip
Ping K. Yip Queen Mary University of London
Peter St George-Hyslop
Peter St George-Hyslop Columbia University
Antonia Gutierrez
Antonia Gutierrez University of Malaga
Michael T. Heneka
Michael T. Heneka University Hospital Bonn
Marie-Ève Tremblay
Marie-Ève Tremblay University of Victoria
Nicola R. Sibson
Nicola R. Sibson University of Oxford
Mikko Airavaara
Mikko Airavaara University of Helsinki
John V. Priestley
John V. Priestley Queen Mary University of London
Esther Berrocoso
Esther Berrocoso University of Cádiz

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