D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 74 Citations 17,080 192 World Ranking 3560 National Ranking 26

Research.com Recognitions

Awards & Achievements

2018 - Member of Academia Europaea

Overview

What is he best known for?

The fields of study Jörg Striessnig is best known for:

  • Gene
  • Enzyme
  • Ion channel

His Biochemistry study frequently draws connections between adjacent fields such as Gene isoform. In most of his Receptor studies, his work intersects topics such as Antagonist. As part of his studies on Antagonist, he frequently links adjacent subjects like Receptor. His work on Dihydropyridine expands to the thematically related Calcium. Dihydropyridine is closely attributed to Calcium in his work. In his research, Jörg Striessnig performs multidisciplinary study on Internal medicine and Endocrinology. In his works, he performs multidisciplinary study on Endocrinology and Internal medicine. His multidisciplinary approach integrates Gene and Peptide sequence in his work. His study brings together the fields of Biochemistry and Peptide sequence.

His most cited work include:

  • Congenital Deafness and Sinoatrial Node Dysfunction in Mice Lacking Class D L-Type Ca2+ Channels (798 citations)
  • Neurobiology of migraine (519 citations)
  • α1D (Cav1.3) Subunits Can Form L-type Ca2+ Channels Activating at Negative Voltages (413 citations)

What are the main themes of his work throughout his whole career to date

Within the field of Protein subunit and Mutation Jörg Striessnig studies Gene. Jörg Striessnig has begun a study into Receptor, looking into Ion channel and Antagonist. As part of his studies on Antagonist, Jörg Striessnig often connects relevant subjects like Receptor. His study in T-type calcium channel extends to Calcium with its themes. Much of his study explores Biochemistry relationship to Binding site. His Biochemistry research extends to the thematically linked field of Binding site. His Organic chemistry study frequently involves adjacent topics like Dihydropyridine. His study brings together the fields of Calcium and Dihydropyridine. His research ties Gating and Biophysics together.

Jörg Striessnig most often published in these fields:

  • Calcium (64.71%)
  • Biochemistry (57.14%)
  • Organic chemistry (52.94%)

What were the highlights of his more recent work (between 2017-2022)?

  • Gene (50.00%)
  • Pharmacology (41.67%)
  • Genetics (41.67%)

In recent works Jörg Striessnig was focusing on the following fields of study:

Gene and splice are frequently intertwined in his study. Splice is closely attributed to Gene in his study. While working in this field, Jörg Striessnig studies both Pharmacology and Drug discovery. He performs integrative study on Drug discovery and Pharmacology. As part of his studies on Genetics, Jörg Striessnig often connects relevant areas like HEK 293 cells. His study ties his expertise on Genetics together with the subject of HEK 293 cells. Neuroscience is closely attributed to Brugada syndrome in his study. His research links Neuroscience with Brugada syndrome. His Key (lock) study frequently links to adjacent areas such as Computer security.

Between 2017 and 2022, his most popular works were:

  • THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: Introduction and Other Protein Targets (226 citations)
  • THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: Ion channels (203 citations)
  • THE CONCISE GUIDE TO PHARMACOLOGY 2021/22: Ion channels (130 citations)

In his most recent research, the most cited works focused on:

  • G protein-coupled receptor
  • Receptor
  • Pharmacology

Jörg Striessnig combines Computational biology and Bioinformatics in his studies. He undertakes interdisciplinary study in the fields of Bioinformatics and Computational biology through his works. He integrates many fields in his works, including Pharmacology and Drug discovery. Jörg Striessnig undertakes multidisciplinary investigations into Drug discovery and Pharmacology in his work. He performs multidisciplinary study in Clinical pharmacology and Internal medicine in his work. Jörg Striessnig brings together Internal medicine and Clinical pharmacology to produce work in his papers. His Computer security study frequently involves adjacent topics like Key (lock). Key (lock) is often connected to Computer security in his work. In his research, Jörg Striessnig performs multidisciplinary study on G protein-coupled receptor and Receptor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Purification, molecular cloning, and expression of the mammalian sigma1-binding site

Markus Hanner;Fabian F. Moebius;Astrid Flandorfer;Hans-Gunther Knaus.
Proceedings of the National Academy of Sciences of the United States of America (1996)

1017 Citations

Congenital Deafness and Sinoatrial Node Dysfunction in Mice Lacking Class D L-Type Ca2+ Channels

Josef Platzer;Jutta Engel;Anneliese Schrott-Fischer;Kurt Stephan.
Cell (2000)

923 Citations

Neurobiology of migraine.

Daniela Pietrobon;Jörg Striessnig.
Nature Reviews Neuroscience (2003)

706 Citations

The Concise Guide to PHARMACOLOGY 2017/18: Overview

Stephen P. H. Alexander;Eamonn Kelly;Neil V. Marrion;John A. Peters.
British Journal of Pharmacology (2017)

490 Citations

α1D (Cav1.3) Subunits Can Form L-type Ca2+ Channels Activating at Negative Voltages

Alexandra Koschak;Daniel Reimer;Irene G. Huber;Manfred Grabner.
Journal of Biological Chemistry (2001)

482 Citations

Functional role of L-type Cav1.3 Ca2+ channels in cardiac pacemaker activity.

Matteo E. Mangoni;Brigitte Couette;Emmanuel Bourinet;Josef Platzer.
Proceedings of the National Academy of Sciences of the United States of America (2003)

470 Citations

Receptor sites for Ca2+ channel antagonists

William A. Catterall;Jörg Striessnig.
Trends in Pharmacological Sciences (1992)

469 Citations

THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: Introduction and Other Protein Targets.

Stephen P.H. Alexander;Eamonn Kelly;Alistair Mathie;John A. Peters.
British Journal of Pharmacology (2019)

341 Citations

Functional embryonic cardiomyocytes after disruption of the L-type alpha1C (Cav1.2) calcium channel gene in the mouse.

Claudia Seisenberger;Verena Specht;Andrea Welling;Josef Platzer.
Journal of Biological Chemistry (2000)

326 Citations

Loss of Ca v 1.3 ( CACNA1D ) function in a human channelopathy with bradycardia and congenital deafness

Shahid M Baig;Alexandra Koschak;Andreas Lieb;Mathias Gebhart.
Nature Neuroscience (2011)

300 Citations

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