2018 - Member of Academia Europaea
Jörg Striessnig mostly deals with Voltage-dependent calcium channel, Biochemistry, Dihydropyridine, Protein subunit and Cav1.3. Jörg Striessnig has included themes like Extracellular, Biophysics, Electrophysiology and Calcium channel in his Voltage-dependent calcium channel study. His Biophysics research incorporates themes from L-type calcium channel and Isradipine.
His Dihydropyridine research integrates issues from Photoaffinity labeling, Peptide sequence, Voltage-gated ion channel and Gene isoform. In his research on the topic of Protein subunit, Missense mutation, Xenopus and Mutant is strongly related with Molecular biology. His Cav1.3 research focuses on subjects like Endocrinology, which are linked to Cardiac pacemaker and Glutamate receptor.
Jörg Striessnig spends much of his time researching Voltage-dependent calcium channel, Calcium channel, Cav1.3, Biophysics and Biochemistry. His Voltage-dependent calcium channel research incorporates themes from Dihydropyridine, Neuroscience and Cell biology. His Calcium channel study combines topics from a wide range of disciplines, such as Receptor, Drug receptor, Protein subunit and Skeletal muscle.
His Protein subunit research incorporates elements of Molecular biology and Gene isoform. In his research, Neurotransmitter is intimately related to Endocrinology, which falls under the overarching field of Cav1.3. In his work, Pharmacology is strongly intertwined with Voltage-gated ion channel, which is a subfield of Biophysics.
Jörg Striessnig focuses on Cav1.3, Voltage-dependent calcium channel, Neuroscience, Biophysics and Internal medicine. His Cav1.3 research is multidisciplinary, relying on both Ribbon synapse, Mutation, Hair cell, Ca2 channels and Retina. Voltage-dependent calcium channel is a subfield of Calcium that Jörg Striessnig investigates.
The concepts of his Neuroscience study are interwoven with issues in L type ca2 channels and Gene. Jörg Striessnig works mostly in the field of Biophysics, limiting it down to topics relating to Ion channel and, in certain cases, Transmembrane domain, as a part of the same area of interest. Jörg Striessnig has included themes like Endocrinology and Cardiology in his Internal medicine study.
Jörg Striessnig spends much of his time researching Pharmacology, Cav1.3, Neuroscience, Voltage-dependent calcium channel and Clinical pharmacology. His work is dedicated to discovering how Pharmacology, Voltage-gated ion channel are connected with Stereochemistry, Neuroprotective Drugs, Activator, Calcium channel agonists and Ca2 channels and other disciplines. His research integrates issues of Mutation, Exome sequencing, Genetics and Bioinformatics in his study of Cav1.3.
His Neuroscience research includes elements of Subventricular zone, Anatomy and Calcium channel. His studies in Calcium channel integrate themes in fields like Gene isoform and Nifedipine. Jörg Striessnig works on Voltage-dependent calcium channel which deals in particular with T-type calcium channel.
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Purification, molecular cloning, and expression of the mammalian sigma1-binding site
Markus Hanner;Fabian F. Moebius;Astrid Flandorfer;Hans-Gunther Knaus.
Proceedings of the National Academy of Sciences of the United States of America (1996)
Congenital Deafness and Sinoatrial Node Dysfunction in Mice Lacking Class D L-Type Ca2+ Channels
Josef Platzer;Jutta Engel;Anneliese Schrott-Fischer;Kurt Stephan.
Neurobiology of migraine.
Daniela Pietrobon;Jörg Striessnig.
Nature Reviews Neuroscience (2003)
α1D (Cav1.3) Subunits Can Form L-type Ca2+ Channels Activating at Negative Voltages
Alexandra Koschak;Daniel Reimer;Irene G. Huber;Manfred Grabner.
Journal of Biological Chemistry (2001)
Functional role of L-type Cav1.3 Ca2+ channels in cardiac pacemaker activity.
Matteo E. Mangoni;Brigitte Couette;Emmanuel Bourinet;Josef Platzer.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Receptor sites for Ca2+ channel antagonists
William A. Catterall;Jörg Striessnig.
Trends in Pharmacological Sciences (1992)
The Concise Guide to PHARMACOLOGY 2017/18: Overview
Stephen P. H. Alexander;Eamonn Kelly;Neil V. Marrion;John A. Peters.
British Journal of Pharmacology (2017)
Functional embryonic cardiomyocytes after disruption of the L-type alpha1C (Cav1.2) calcium channel gene in the mouse.
Claudia Seisenberger;Verena Specht;Andrea Welling;Josef Platzer.
Journal of Biological Chemistry (2000)
Structural basis of drug binding to L Ca2+ channels
Jörg Striessnig;Manfred Grabner;Jörg Mitterdorfer;Steffen Hering.
Trends in Pharmacological Sciences (1998)
Fast exocytosis with few Ca(2+) channels in insulin-secreting mouse pancreatic B cells.
Sebastian Barg;Xiaosong Ma;Lena Eliasson;Juris Galvanovskis.
Biophysical Journal (2001)
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